Treatment of Supraventricular Tachycardia (SVT)
For acute SVT management, begin with vagal maneuvers (specifically the modified Valsalva maneuver in supine position with legs raised), followed immediately by adenosine 6 mg IV rapid push if vagal maneuvers fail, and proceed to synchronized cardioversion if the patient is hemodynamically unstable or medications fail. 1, 2
Acute Management Algorithm
Step 1: Vagal Maneuvers (First-Line)
- Perform the modified Valsalva maneuver as the initial intervention: patient bears down against a closed glottis for 10-30 seconds (equivalent to 30-40 mmHg pressure) while supine, then immediately lies flat with legs raised 1, 2
- If Valsalva fails, attempt carotid sinus massage after confirming absence of carotid bruits by auscultation—apply steady pressure over the right or left carotid sinus for 5-10 seconds 1
- Alternative vagal maneuver: apply an ice-cold, wet towel to the face to trigger the diving reflex 1, 3
- Success rate of vagal maneuvers is approximately 27.7% when switching between techniques 1
- Critical caveat: Never apply pressure to the eyeball—this practice is dangerous and abandoned 1
Step 2: Adenosine (First-Line Medication)
- Administer adenosine 6 mg rapid IV bolus through a large peripheral vein, followed immediately by saline flush 1, 2, 4
- If ineffective after 1-2 minutes, give 12 mg rapid IV bolus (may repeat once) 1, 4
- Effectiveness: 90-95% conversion rate for AVNRT and AVRT 1, 2, 3
- Essential preparation: Have resuscitative equipment immediately available, as adenosine can cause transient asystole, high-grade AV block, or precipitate atrial fibrillation 4
- Contraindications: Second- or third-degree AV block (without pacemaker), sick sinus syndrome, bronchospastic lung disease (asthma), and known hypersensitivity 4
- Warning: Fatal cardiac arrest, sustained ventricular tachycardia, and myocardial infarction have occurred—avoid in patients with acute myocardial ischemia or unstable angina 4
Step 3: Alternative Medications (If Adenosine Fails or Contraindicated)
- For hemodynamically stable patients, use IV calcium channel blockers or beta-blockers 1, 2:
- Critical warning: Do NOT give verapamil or diltiazem if ventricular tachycardia or pre-excited atrial fibrillation is suspected—this can cause hemodynamic collapse or ventricular fibrillation 1, 3
- Avoid calcium channel blockers in patients with suspected systolic heart failure 1
Step 4: Synchronized Cardioversion
- For hemodynamically unstable patients: Perform immediate synchronized cardioversion when vagal maneuvers and adenosine fail or are not feasible 1
- For hemodynamically stable patients: Use synchronized cardioversion when pharmacological therapy fails or is contraindicated 1
- Initial energy: 50-100 joules for SVT 3
- Safety note: Synchronized cardioversion is safe and effective in all trimesters of pregnancy if necessary 1, 5
Long-Term Management
Pharmacological Prevention
- Beta-blockers are the first-line option for long-term prevention of recurrent SVT 2, 6
- Calcium channel blockers (diltiazem or verapamil) are reasonable alternatives to beta-blockers 2
- Important caveat: When discontinuing beta-blockers in patients with coronary artery disease, taper gradually over 1-2 weeks to avoid severe exacerbation of angina, myocardial infarction, or ventricular arrhythmias 6
Catheter Ablation (Curative Option)
- Catheter ablation is the definitive curative treatment for patients with 2, 7:
- Frequent symptomatic episodes
- Poor tolerance or ineffectiveness of medications
- Patient preference for non-pharmacological approach
- Ablation is curative in the majority of patients and should be discussed with all patients after initial SVT episode 7
Special Populations
Pregnancy
- Vagal maneuvers remain first-line and are safe during pregnancy 1, 5
- Adenosine is safe due to its extremely short half-life (unlikely to reach fetal circulation) 1, 5
- Avoid atenolol and verapamil for long-term management due to teratogenic effects 5
- Synchronized cardioversion can be performed safely at all stages of pregnancy if necessary 1
Pre-Excitation Syndromes (e.g., Wolff-Parkinson-White)
- Avoid all AV nodal blocking agents (adenosine, beta-blockers, calcium channel blockers, digoxin) in pre-excited atrial fibrillation—these can accelerate ventricular rate and cause hemodynamic collapse 1, 3
- Use IV procainamide or ibutilide for hemodynamically stable pre-excited AF 2
- Use immediate cardioversion for hemodynamically unstable patients 8
Patients with Low Baseline Heart Rate
- Vagal maneuvers remain safe as first-line treatment 3
- Reduce adenosine dosing and monitor carefully for prolonged bradycardia 3
- Consider synchronized cardioversion earlier in the treatment algorithm, as these patients may not tolerate rate-controlling medications 3
- Avoid high doses of AV nodal blocking agents (beta-blockers, calcium channel blockers) as they may exacerbate underlying bradycardia 3
Critical Diagnostic Considerations
- Always obtain a 12-lead ECG during tachycardia before treatment to differentiate SVT mechanisms 2
- Distinguish SVT with aberrancy from ventricular tachycardia before initiating treatment—giving calcium channel blockers or beta-blockers for VT can be fatal 1, 2
- Rule out organic heart disease (especially mitral stenosis from rheumatic heart disease) in patients presenting with SVT 5
Common Pitfalls to Avoid
- Never abruptly discontinue beta-blocker therapy in patients with coronary artery disease—this can precipitate severe angina, MI, or ventricular arrhythmias 6
- Do not use calcium channel blockers or beta-blockers if pre-excited AF or VT is suspected 1, 3
- Prepare for atrial fibrillation when giving adenosine—it may precipitate AF requiring cardioversion 3, 4
- Automatic tachycardias (ectopic atrial tachycardia, multifocal atrial tachycardia) will not respond to cardioversion and require different treatment approaches 3
- Beta-blockers may mask hypoglycemia in diabetic patients (tachycardia will be blunted, though dizziness and sweating persist) 6