Does pre-eclampsia cause an increase in white blood cell (WBC) count?

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Does Pre-eclampsia Cause White Blood Cell Count to Increase?

Yes, pre-eclampsia is associated with elevated white blood cell (WBC) counts as part of a systemic inflammatory response that characterizes this disorder.

Inflammatory Nature of Pre-eclampsia

Pre-eclampsia fundamentally represents an excessive maternal inflammatory response to pregnancy, involving activation of multiple inflammatory pathways 1, 2. The condition is characterized by:

  • Leukocyte activation as a core feature of the systemic inflammatory response 1
  • Intravascular inflammation involving white blood cells, endothelial cells, and the clotting cascade 2
  • Neutrophil and lymphocyte elevation that significantly exceeds levels seen in normal pregnancy 3

The inflammatory response in pre-eclampsia is not a separate phenomenon but rather represents decompensation of the universal maternal inflammatory adaptation to pregnancy 2.

Evidence for Elevated WBC in Pre-eclampsia

Recent research directly demonstrates increased white blood cell parameters in pre-eclamptic patients:

  • WBC count is significantly higher in women with pre-eclampsia compared to normal pregnant controls 3
  • Neutrophil counts are significantly elevated in the pre-eclampsia group 3
  • Lymphocyte counts are also significantly increased 3

A 2024 study found that the systemic immune-inflammation index (which incorporates neutrophil, lymphocyte, and platelet counts) had 77.5% sensitivity and 67.5% specificity for diagnosing pre-eclampsia at a cut-off value of 758.39 × 10⁹/L 3.

Pathophysiological Mechanism

The WBC elevation occurs through a specific cascade:

  • Placental oxidative stress from inadequate blood supply triggers release of inflammatory factors into maternal circulation 1
  • Syncytiotrophoblast debris and antiangiogenic factors (such as sFlt-1) activate the maternal inflammatory system 1, 4
  • Endothelial dysfunction results from this inflammatory activation, creating a feed-forward cycle 1, 2
  • Intravascular inflammation involves activation of leukocytes, complement, and coagulation systems 2

Clinical Monitoring Implications

While WBC elevation is characteristic of pre-eclampsia, platelet count monitoring is more clinically critical for management decisions:

  • Platelet counts are significantly lower in pre-eclampsia compared to controls 3
  • Progressive thrombocytopenia is an indication for delivery at any gestational age 5, 6
  • Twice-weekly monitoring of hemoglobin, platelet count, liver enzymes, and renal function is recommended 5, 6

The NHLBI Working Group notes that while activation of the coagulation cascade can be detected in most pre-eclamptic women, consumption of procoagulants sufficient to be detected by standard testing occurs in only about 10% of cases 5.

Important Caveats

  • WBC elevation alone is not diagnostic of pre-eclampsia, as normal pregnancy itself involves increased inflammatory markers 2
  • The difference between normal pregnancy and pre-eclampsia is quantitative rather than qualitative—pre-eclampsia represents the extreme end of normal maternal inflammatory adaptation 2
  • WBC count should not guide delivery decisions; instead, focus on blood pressure control, platelet count, liver/renal function, and fetal status 5, 6

The systemic immune-inflammation index may serve as a helpful adjunctive marker because it simultaneously evaluates neutrophils and lymphocytes (indicating inflammation) along with platelets (indicating coagulopathy) 3.

References

Research

Inflammation and pre-eclampsia.

Seminars in fetal & neonatal medicine, 2006

Research

Preeclampsia: an excessive maternal inflammatory response to pregnancy.

American journal of obstetrics and gynecology, 1999

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Management and Treatment of Preeclampsia

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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