What degrades calcitonin gene-related peptide (CGRP)?

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Last updated: November 24, 2025View editorial policy

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CGRP Degradation

Neutral endopeptidase (NEP) is the primary enzyme responsible for degrading CGRP in human tissues, while angiotensin-converting enzyme (ACE) appears to have an inhibitory rather than degradative role in CGRP metabolism. 1

Primary Degradation Pathway

  • NEP serves as the most important enzyme for CGRP degradation in human skin and peripheral tissues. 1
  • When NEP is blocked with phosphoramidon, CGRP levels increase significantly and can be directly measured in tissue samples, demonstrating NEP's critical role in CGRP catabolism. 1
  • Inhibition of NEP with phosphoramidon increases both the intensity and size of CGRP-mediated neurogenic flare responses, providing functional evidence of reduced CGRP degradation. 1

Role of ACE in CGRP Metabolism

  • ACE does not contribute to CGRP degradation; instead, it appears to have an inhibitory role in CGRP metabolism. 2
  • When ACE is blocked with captopril alongside NEP inhibition, CGRP levels actually decrease compared to NEP inhibition alone, suggesting ACE blockade augments CGRP catabolism rather than preventing it. 2
  • Combined inhibition of NEP and ACE does not produce additive effects on CGRP accumulation, further supporting that these enzymes do not work synergistically in CGRP degradation. 2

Clinical Implications

  • Brush border peptidases in the intestinal epithelium also degrade peptides including CGRP-related molecules, representing an additional site of peptide metabolism. 3
  • The specificity of NEP for CGRP degradation has therapeutic relevance for conditions involving neurogenic inflammation, including migraine and complex regional pain syndrome. 1
  • Monoclonal antibodies targeting CGRP can effectively block both exogenous and endogenous CGRP activity, demonstrating CGRP's neurotransmitter role and providing an alternative therapeutic approach to enzyme inhibition. 4

Functional Consequences of Reduced Degradation

  • NEP inhibition increases CGRP-mediated vasodilation and flare responses, confirming that endogenous NEP activity normally limits CGRP bioavailability. 5
  • Blocking CGRP degradation with phosphoramidon paradoxically suppresses sweating while increasing the area of axon reflex flare, likely due to elevated CGRP and substance P levels. 5

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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