How do you classify supraventricular tachycardia (SVT)?

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Classification of Supraventricular Tachycardia

Supraventricular tachycardia is classified into three primary anatomic categories based on the tissue involved in the tachycardia circuit: atrial tachycardias (originating from atrial tissue), atrioventricular junctional tachycardias (involving the AV node), and atrioventricular tachycardias (requiring an accessory pathway). 1

Primary Classification Framework

The most clinically useful classification system divides SVT by anatomic origin and mechanism 1:

1. Atrial Tachycardias

These originate from atrial tissue above the AV node and include 1:

  • Sinus tachycardias:

    • Physiological sinus tachycardia (appropriate response to exercise, stress, or other stimuli) 1
    • Inappropriate sinus tachycardia (sinus rate >100 bpm at rest with mean 24-hour rate >90 bpm without physiological cause) 1, 2
    • Sinus node reentrant tachycardia (microreentry within sinus node complex with P-wave morphology indistinguishable from sinus rhythm) 1
  • Focal atrial tachycardia: Arises from a localized atrial site with regular, organized atrial activity and discrete P waves with isoelectric segments between them 1, 2

  • Multifocal atrial tachycardia (MAT): Irregular SVT with ≥3 distinct P-wave morphologies at different rates 1

  • Macroreentrant atrial tachycardias (atrial flutter):

    • Cavotricuspid isthmus-dependent (typical atrial flutter, counter-clockwise or clockwise) 1
    • Non-cavotricuspid isthmus-dependent (atypical flutter including mitral isthmus-dependent and other left or right atrial circuits) 1

2. Atrioventricular Junctional Tachycardias

These involve the AV node as the critical component 1:

  • Atrioventricular nodal reentrant tachycardia (AVNRT): The most common form of paroxysmal SVT, caused by reentry within the AV node using dual pathways (fast and slow conduction pathways) 2, 3

    • Typical (slow-fast) AVNRT: Atrial activation nearly simultaneous with QRS, producing pseudo S waves in inferior leads and pseudo R' in V1 1
    • Atypical (fast-slow or slow-slow) AVNRT: P wave closer to subsequent QRS, producing long RP interval 1
  • Non-paroxysmal junctional tachycardia: Automatic focus in AV junction 1

  • Focal junctional tachycardia: Ectopic focus within junctional tissue 1

3. Atrioventricular Tachycardias (Accessory Pathway-Mediated)

These require an accessory pathway connecting atrium and ventricle 1:

  • Wolff-Parkinson-White (WPW) syndrome: Documented SVT or symptoms consistent with SVT in a patient with ventricular pre-excitation during sinus rhythm (incidence 0.1-0.3% in general population) 2

  • Atrioventricular reentrant tachycardia (AVRT):

    • Orthodromic AVRT: Anterograde conduction down AV node, retrograde up accessory pathway (narrow QRS with P wave in early ST segment, >70 ms after QRS) 1
    • Antidromic AVRT: Anterograde conduction down accessory pathway, retrograde up AV node (wide QRS) 1
    • Permanent form of junctional reciprocating tachycardia (PJRT): Orthodromic AVRT via slowly conducting accessory pathway with decremental properties, producing long RP interval 1
  • Concealed accessory pathways: Capable only of retrograde conduction 1

Practical ECG-Based Classification Algorithm

When evaluating SVT on ECG, use this systematic approach 1:

Step 1: Assess QRS Width

  • Narrow QRS (<120 ms): Almost always SVT 4
  • Wide QRS (>120 ms): May be SVT with bundle branch block, SVT with accessory pathway conduction, or ventricular tachycardia—when uncertain, treat as VT 1, 4

Step 2: Assess Rhythm Regularity

  • Irregular ventricular rate: Suggests atrial fibrillation, MAT, or atrial flutter with variable AV conduction 1
  • Regular ventricular rate: Proceed to Step 3 1

Step 3: Compare Atrial and Ventricular Rates

  • Atrial rate exceeds ventricular rate: Atrial flutter or atrial tachycardia (with 2:1 or variable block) 1
  • 1:1 AV relationship: Proceed to Step 4 1

Step 4: Identify P Wave Location (RP Interval)

  • No visible P waves (hidden in QRS): Most likely AVNRT 4
  • Pseudo R' in V1 or pseudo S in inferior leads: Pathognomonic for typical AVNRT 1, 4
  • Short RP (P wave closer to prior QRS, RP <70 ms): Typical AVNRT or orthodromic AVRT 1
  • Long RP (P wave closer to subsequent QRS): Atypical AVNRT, PJRT, or atrial tachycardia 1
  • P wave in ST segment separated from QRS by >70 ms: Orthodromic AVRT most likely 1, 4

Critical Distinguishing Features for Wide QRS Tachycardia

When QRS is >120 ms, differentiate VT from SVT using 1:

  • AV dissociation with ventricular rate faster than atrial rate: Proves VT (visible in only 30% of cases) 1, 4
  • Fusion complexes: Pathognomonic for VT 1, 4
  • Capture beats: Diagnostic of VT 4
  • Concordance of precordial QRS complexes (all positive or all negative): Suggests VT or pre-excitation 1
  • QRS identical to sinus rhythm: Consistent with SVT with pre-existing bundle branch block 1

Common Pitfalls to Avoid

  • Never assume hemodynamic stability indicates SVT—VT can be well-tolerated, especially in younger patients without structural heart disease 4
  • Atrial fibrillation with rapid ventricular response may appear regular at very fast rates and be misdiagnosed as regular SVT 1, 4
  • Pre-excitation on baseline ECG mandates electrophysiology referral due to sudden death risk, particularly if atrial fibrillation develops 4
  • Automatic ECG interpretations are unreliable—always interpret manually 4
  • In patients with prior myocardial infarction, wide-complex tachycardia is VT until proven otherwise 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Supraventricular Tachycardia Types and Characteristics

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Clinical Diagnosis of Supraventricular Tachycardia (SVT) and Ventricular Tachycardia (VT)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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