What is the half-life of fosphenytoin (antiepileptic medication)?

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Fosphenytoin Half-Life

The conversion half-life of fosphenytoin to phenytoin is approximately 15 minutes, after which fosphenytoin is completely metabolized to its active form, phenytoin. 1

Conversion Pharmacokinetics

  • Fosphenytoin itself has a conversion half-life of 8-15 minutes when administered intravenously, during which it is rapidly converted to phenytoin by blood and tissue phosphatases 2, 3, 4, 5

  • The FDA label specifically states the conversion half-life is approximately 15 minutes 1

  • Following IV administration, maximum plasma fosphenytoin concentrations are achieved at the end of infusion, with complete conversion to phenytoin occurring rapidly thereafter 1

Route-Specific Considerations

Intravenous Administration

  • Therapeutic free phenytoin concentrations are achieved within minutes after completion of the infusion 1, 2
  • The rapid 15-minute conversion allows fosphenytoin infused at 100-150 mg PE/min to produce phenytoin levels comparable to phenytoin sodium infused at 50 mg/min 1

Intramuscular Administration

  • Following IM administration, the absorption rate becomes the rate-limiting step with a half-life range of 22-41 minutes 3
  • Peak fosphenytoin concentrations occur at approximately 30 minutes post-dose 1
  • Peak phenytoin concentrations occur at 2-4 hours after IM administration 2
  • Therapeutic phenytoin levels are generally achieved within 30 minutes to 1 hour after IM injection 1, 2

Clinical Implications

  • Fosphenytoin is entirely eliminated through metabolism to phenytoin—it is not excreted unchanged in urine 1

  • The bioavailability of derived phenytoin is approximately 100% following either IV or IM administration 1, 2, 3

  • Once converted to phenytoin, the subsequent elimination follows phenytoin pharmacokinetics (half-life 11-55 hours in children, 63-88 hours in neonates) 6

References

Research

Fosphenytoin: a novel phenytoin prodrug.

Pharmacotherapy, 1996

Research

Fosphenytoin (Cerebyx).

Clinical neuropharmacology, 1997

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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