What is the recommended use and management of Prograf (tacrolimus) in patients who have undergone organ transplantation?

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Last updated: December 3, 2025View editorial policy

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Prograf (Tacrolimus) in Organ Transplantation

Recommended Use and Indications

Prograf (tacrolimus) is FDA-approved for prophylaxis of organ rejection in adult patients receiving allogeneic kidney, liver, or heart transplants, and in pediatric patients receiving liver transplants, always used in combination with other immunosuppressants. 1


Dosing Recommendations by Organ Type

Kidney Transplantation

  • With azathioprine: Start 0.2 mg/kg/day divided every 12 hours, targeting trough levels of 7-20 ng/mL for months 1-3, then 5-15 ng/mL for months 4-12 1
  • With MMF/IL-2 receptor antagonist: Start 0.1 mg/kg/day divided every 12 hours, targeting 4-11 ng/mL for months 1-12 1
  • Long-term maintenance: Most patients can be maintained on 4-6 ng/mL after the first year 2, 3

Liver Transplantation

  • Adults with corticosteroids: Start 0.1-0.15 mg/kg/day divided every 12 hours, targeting 5-20 ng/mL for months 1-12 1
  • Pediatric patients: Start 0.15-0.2 mg/kg/day divided every 12 hours, targeting 5-20 ng/mL for months 1-12 1
  • Renal-sparing regimens: Target lower levels of 6-10 ng/mL during the first month, then 4-8 ng/mL thereafter when combined with short-course corticosteroids 2
  • With basiliximab and/or MMF/azathioprine: Target even lower levels of 4-7 ng/mL during the first month, followed by 3-5 ng/mL 2

Heart Transplantation

  • With azathioprine or MMF: Start 0.075 mg/kg/day divided every 12 hours, targeting 10-20 ng/mL for months 1-3, then 5-15 ng/mL for month 4 and beyond 1

Critical Administration Guidelines

Administer tacrolimus capsules consistently either with or without food—never alternate—as food decreases absorption by 37% with a 77% decrease in maximum plasma concentration. 2

  • Avoid grapefruit and grapefruit juice, which can significantly alter drug levels 1
  • Do not interchange tacrolimus formulations (immediate-release vs. extended-release) without careful monitoring, as they are not bioequivalent and medication errors can lead to serious patient harm 1, 4
  • Instruct patients to recognize the appearance of their specific tacrolimus capsules to prevent accidental substitution 1

Therapeutic Drug Monitoring Protocol

Immediate Post-Operative Period

  • Monitor trough levels daily or every other day until target levels are reached 2, 5
  • Continue monitoring every 2-3 days until hospital discharge 3, 5

First 3 Months

  • Weeks 2-4: Monitor 2-3 times per week 2
  • Months 2-3: Monitor weekly 2

Months 4-12

  • Months 4-6: Monitor every 2 weeks 2
  • Months 7-12: Monitor monthly 2

Beyond First Year

  • Monitor every 2-3 months once stable 2
  • Increase monitoring frequency when adding or withdrawing CYP3A4-affecting medications, switching formulations, during allograft dysfunction, or when hospitalized 3, 5

Essential Laboratory Monitoring

Beyond trough levels, rigorous monitoring of multiple parameters is essential given tacrolimus's narrow therapeutic window and potential for serious toxicity. 2

  • Renal function: Monitor serum creatinine regularly to detect nephrotoxicity 6, 2, 1
  • Electrolytes: Monitor potassium (risk of hyperkalemia) and magnesium (risk of hypomagnesemia) closely 6, 2, 1
  • Glucose: Monitor blood glucose levels regularly due to risk of new-onset diabetes after transplant 1
  • Complete blood count: Monitor for bone marrow suppression, leukopenia, and anemia 6, 2, 1
  • Liver function tests: Monitor hepatic function regularly 2, 1
  • Blood pressure: Measure frequently, as hypertension is common and may require antihypertensive therapy 6, 1
  • Lipid profile: Monitor for dyslipidemia 6

Special Populations and Dose Adjustments

Patients at Risk of Renal Dysfunction

  • Use basiliximab induction with MMF or azathioprine to allow a 5-day delay in introducing tacrolimus 2, 3
  • Target lower trough concentrations (4-7 ng/mL initially, then 3-5 ng/mL) when combined with other immunosuppressants 2, 3

African-American Patients

  • May require dosage adjustments according to FDA labeling 1

Hepatic and Renal Impairment

  • Dosage adjustments are necessary 1

Critical Drug Interactions

Tacrolimus is metabolized through the hepatic CYP3A4 system, making it highly susceptible to drug interactions that can dramatically alter levels. 6, 3

  • CYP3A4 inhibitors (e.g., azole antifungals, macrolide antibiotics, calcium channel blockers) will increase tacrolimus levels 6
  • CYP3A4 inducers (e.g., rifampin, phenytoin, carbamazepine) will decrease tacrolimus levels 6
  • Cannabidiol can interact with tacrolimus 1
  • Mycophenolic acid products: Switching from cyclosporine to tacrolimus can increase MPA exposure; monitor for MPA-related adverse reactions 1
  • Tacrolimus itself inhibits CYP3A4 and may reduce clearance of digoxin, colchicine, and HMG-CoA reductase inhibitors 6

Infection Prophylaxis Requirements

Prophylaxis against Pneumocystis jiroveci must be implemented with tacrolimus use. 6, 2

  • Consider antifungal prophylaxis in patients receiving steroids for neurotoxicity treatment 2
  • Long-term antiviral therapy is recommended to prevent CMV and EBV infections 6
  • Use molecular diagnostic tests (PP65 for CMV, PCR) for early viral replication detection with prompt preemptive therapy 6

Major Toxicities and Black Box Warnings

The FDA has issued a black box warning for increased susceptibility to serious infections and possible development of lymphoma and other malignancies that may lead to hospitalization or death. 1

Common Serious Adverse Effects (>15% incidence):

  • Nephrotoxicity (acute and/or chronic)—reduce dose and use caution with other nephrotoxic drugs 1
  • Neurotoxicity including tremor, headache, paresthesia, and risk of Posterior Reversible Encephalopathy Syndrome (PRES)—monitor for neurologic abnormalities and reduce or discontinue if severe 1
  • New-onset diabetes after transplant—monitor blood glucose closely 1
  • Hyperkalemia—monitor serum potassium and consider carefully before using with other agents associated with hyperkalemia 1
  • Hypertension—may require antihypertensive therapy 1
  • Hypomagnesemia 1

Other Significant Toxicities:

  • Myocardial hypertrophy—consider dose reduction or discontinuation 1
  • Pure red cell aplasia—consider discontinuation 1
  • Thrombotic microangiopathy (including hemolytic uremic syndrome and thrombotic thrombocytopenic purpura), especially with infections and certain concomitant medications 1
  • Anaphylactic reactions with IV formulation—observe patients for signs and symptoms 1

Important Contraindications and Precautions

  • Contraindicated in patients with hypersensitivity to tacrolimus or HCO-60 (polyoxyl 60 hydrogenated castor oil) 1
  • Not recommended for use with sirolimus in liver and heart transplant due to increased risk of serious adverse reactions 1
  • Avoid live vaccines during tacrolimus therapy 1
  • Not interchangeable with extended-release tacrolimus products without appropriate dose adjustments and monitoring 1

Long-Term Management Considerations

  • Monitor for donor-specific antibodies combined with liver enzymes and non-invasive tests to identify patients at risk of subclinical rejection 2, 5
  • Many long-term survivors can maintain normal function with tacrolimus levels substantially lower than initial targets, but weigh benefits against risk of subclinical rejection 2
  • Conversion to CNI-free everolimus-based regimens can be achieved in more than 50% of patients beyond the first year 2

Common Pitfalls to Avoid

  • Never switch between tacrolimus formulations (brand to generic, immediate-release to extended-release) without increasing monitoring frequency, as bioavailability differences can precipitate rejection 5, 4
  • Trough levels may not always correlate well with overall drug exposure (AUC), particularly with extended-release formulations 4
  • Do not assume therapeutic drug monitoring strategies for one formulation apply equally to another 4
  • Recognize that tacrolimus has higher rates of diabetes mellitus and neurotoxicity compared to cyclosporine, while cyclosporine has higher rates of hypertension, hyperlipidemia, hirsutism, and gingival hyperplasia 6, 7, 8, 9

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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