What is ascitis?

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Definition of Ascites

Ascites is the pathological accumulation of fluid within the peritoneal cavity, most commonly caused by portal hypertension in cirrhosis (75-85% of cases). 1, 2

Core Definition and Pathophysiology

  • Ascites represents fluid accumulation specifically in the peritoneal cavity, not in subcutaneous tissues or other compartments. 3

  • The condition develops when portal hypertension increases hydrostatic pressure at the sinusoidal level, combined with splanchnic arterial vasodilation and activation of sodium-retaining mechanisms (renin-angiotensin-aldosterone system, sympathetic nervous system, and arginine vasopressin). 1, 4

  • Portal hypertension triggers intestinal microvasculature to generate angiogenic factors, leading to portosystemic collateral development and further exacerbation of the hemodynamic abnormalities. 1

Clinical Grading System

Ascites severity is classified into three grades based on clinical presentation: 1, 5

  • Grade 1 (Mild): Detectable only by ultrasound examination
  • Grade 2 (Moderate): Causes moderate symmetrical abdominal distension with positive shifting dullness
  • Grade 3 (Large/Tense): Causes marked abdominal distension, typically exceeding 5 liters of fluid

Physical Examination Findings

  • Shifting dullness becomes detectable when approximately 1,500 mL of free fluid accumulates in the abdomen, with 83% sensitivity and 56% specificity for detecting ascites. 1, 6, 5

  • Physical examination is unreliable in obese patients or when smaller fluid volumes are present, requiring ultrasound confirmation which can detect as little as 100 mL. 1, 5

Clinical Classifications

Uncomplicated Ascites

Ascites that is not infected and not associated with hepatorenal syndrome (HRS). 1

Refractory Ascites

Ascites that cannot be mobilized or recurs early after therapeutic paracentesis despite maximal medical treatment, including two subgroups: 1

  • Diuretic-resistant ascites: Refractory to dietary sodium restriction and intensive diuretic treatment
  • Diuretic-intractable ascites: Refractory due to diuretic-induced complications preventing effective dosing

Etiologic Classification by SAAG

The serum-ascites albumin gradient (SAAG) is the most accurate single test for classifying ascites etiology with 97% accuracy: 6, 7

SAAG ≥11 g/L indicates portal hypertension: 1, 6

  • Cirrhosis (most common)
  • Cardiac failure
  • Portal vein thrombosis
  • Hypothyroidism (limited data)

SAAG <11 g/L indicates non-portal hypertensive causes: 1, 6

  • Peritoneal carcinomatosis
  • Peritoneal tuberculosis
  • Pancreatitis
  • Bowel perforation
  • Nephrotic syndrome

Critical Diagnostic Pitfalls

  • Do not confuse ascites with subcutaneous edema of the abdominal wall—these represent distinct fluid compartments requiring different diagnostic and therapeutic approaches. 3

  • Do not assume all abdominal distension is ascites—careful physical examination with shifting dullness testing and ultrasound confirmation are essential before proceeding to paracentesis. 6, 5

  • Do not delay diagnostic paracentesis in hospitalized patients or those with new-onset ascites—each hour of delay increases in-hospital mortality by 3.3% when spontaneous bacterial peritonitis is present. 1, 5

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Abdominal Subcutaneous Edema

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Pathophysiology and treatment of ascites and the hepatorenal syndrome.

Bailliere's clinical gastroenterology, 1992

Guideline

Ascites Diagnosis and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Evaluation and Management of Abdominal Ascites

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Ascites in Children.

Indian journal of pediatrics, 2016

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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