What is the dosage and treatment protocol for penicillamine (D-penicillamine) in medical practice?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: December 4, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

D-Penicillamine Dosing and Treatment Protocol

For Wilson disease, start D-penicillamine at 250-500 mg/day and titrate by 250 mg increments every 4-7 days to a maximum of 1,000-1,500 mg/day in 2-4 divided doses, with maintenance typically at 750-1,000 mg/day in 2 divided doses. 1

Dosing Regimen

Initial Dosing

  • Adults: Begin with 250-500 mg/day, increase by 250 mg increments every 4-7 days until reaching 1,000-1,500 mg/day 1
  • Children: 20 mg/kg/day rounded to nearest 250 mg, given in 2-3 divided doses 1
  • Administration timing: 1 hour before or 2 hours after meals (food reduces absorption by ~50%) 1
    • Closer proximity to meals is acceptable if it ensures compliance 1

Maintenance Dosing

  • Target dose: 750-1,000 mg/day in 2 divided doses 1
  • Maximum: 1,000-1,500 mg/day in divided doses 1
  • Critical warning: Doses ≥1,500 mg/day given at once may cause rapid, irreversible neurological deterioration 1

Mandatory Supplementation

  • Pyridoxine (Vitamin B6): 25-50 mg daily throughout treatment 1
    • Required because penicillamine interferes with pyridoxine action 1

Monitoring Treatment Adequacy

Primary Monitoring Parameters

  • 24-hour urinary copper excretion: Should be 200-500 μg/day (3-8 μmol/day) on treatment 1
  • Non-ceruloplasmin-bound copper: Should normalize with effective treatment 1
  • Compliance check: After 2 days off penicillamine, urinary copper should be ≤1.6 μmol/24h; values >1.6 μmol/24h suggest non-adherence 1

Expected Clinical Response Timeline

  • Hepatic disease: Recovery of synthetic function and improvement in jaundice/ascites typically occurs within 2-6 months, with continued improvement through first year 1
  • Neurologic disease: Improvement is slower, may take up to 3 years 1
  • Serum ceruloplasmin: Tends to decrease after treatment initiation; may remain low or increase as hepatic function recovers 1

Critical Adverse Effects Requiring Immediate Discontinuation

Early Reactions (First 1-3 Weeks)

  • Fever and cutaneous eruptions 1
  • Lymphadenopathy 1
  • Neutropenia or thrombocytopenia 1
  • Proteinuria 1
  • Action: Discontinue immediately; do NOT attempt prednisone co-treatment 1

Late Reactions

  • Nephrotoxicity: Proteinuria or cellular elements in urine—discontinue immediately 1
  • Severe bone marrow toxicity: Severe thrombocytopenia or total aplasia 1
  • Lupus-like syndrome: Hematuria, proteinuria, positive ANA 1
  • Goodpasture syndrome (with higher doses) 1

Very Late Reactions

  • Myasthenia gravis 1
  • Polymyositis 1
  • Severe allergic response upon restarting after discontinuation 1
  • Hepatotoxicity and hepatic siderosis 1

Dermatologic Toxicities

  • Elastosis perforans serpiginosa 1
  • Pemphigus or pemphigoid lesions 1
  • Cutis laxa (with long-term high-dose use) 2
  • Lichen planus, aphthous stomatitis 1

Common Pitfalls and Caveats

Neurologic Worsening

  • 10-50% of patients experience worsening neurologic symptoms during initial treatment 1
  • In recent series, 13.8% had neurologic deterioration with penicillamine (highest among all Wilson disease treatments) 1
  • Mitigation: Start with incremental doses (125-250 mg/day) and increase slowly 1

Overall Discontinuation Rate

  • 20-30% of patients require discontinuation due to severe side effects 1
  • Long-term studies show only 30-40% still taking drug at 2 years 3

Non-Compliance Consequences

  • Failure to comply leads to significant liver disease progression and liver failure within 1-12 months 1
  • May result in death or necessitate liver transplantation 1

Pharmacokinetic Considerations

  • Bioavailability: 40-70% with wide interindividual variation 1, 4
  • Peak plasma concentration: 1-3 hours post-dose 1, 4
  • Protein binding: 80% bound to plasma proteins 1
  • Elimination half-life: 1.7-7 hours, but metabolites can persist for months after discontinuation 1
  • Excretion: >80% renal 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Clinical pharmacokinetics of D-penicillamine.

Clinical pharmacokinetics, 1987

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.