Is Wolff-Parkinson-White (WPW) syndrome hereditary?

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Is Wolff-Parkinson-White Syndrome Hereditary?

WPW syndrome can be hereditary in rare cases, but the vast majority of cases (>95%) occur sporadically without a clear genetic inheritance pattern. 1, 2

Genetic Forms of WPW

PRKAG2-Related Familial WPW

  • A specific hereditary form exists caused by mutations in the PRKAG2 gene, which encodes the gamma2 regulatory subunit of AMP-activated protein kinase. 3
  • This familial form follows an autosomal dominant inheritance pattern, meaning affected individuals have a 50% chance of passing the condition to each child. 3
  • The PRKAG2-related WPW is associated with cardiac hypertrophy and glycogen storage cardiomyopathy, distinguishing it from typical isolated WPW. 4, 3
  • This genetic form accounts for less than 1% of all WPW cases (only 0.6% in one large genetic study). 2

Other Genetic Associations

  • Recent genetic studies have identified rare de novo variants in genes associated with arrhythmia and cardiomyopathy (ANK2, NEBL, PITX2, PRDM16) in some WPW patients. 2
  • There is an increased burden of rare deleterious variants in genes linked to atrial fibrillation in WPW patients compared to controls, suggesting some genetic susceptibility. 2
  • Familial occurrence has been documented in only seven instances in the literature prior to 1982, with one additional family showing autosomal dominant inheritance affecting multiple siblings. 5

Clinical Implications for Family Screening

When to Consider Genetic Testing

  • Obtain ECGs in siblings of young athletes with WPW or bifascicular block patterns to screen for familial forms. 4
  • Family history assessment should focus on pre-excitation in first-degree relatives, sudden cardiac death in young family members, and cardiomyopathy. 6
  • Echocardiography is essential to identify PRKAG2-related familial WPW, which presents with cardiac hypertrophy and glycogen storage features. 4, 6

Risk Stratification in Familial Cases

  • Familial WPW is associated with a high incidence of sudden death, making risk stratification particularly important in these rare cases. 6
  • Electrophysiological study for risk stratification should be reserved for selected patients with a family history of sudden cardiac death. 4

Sporadic WPW (The Majority)

  • Most WPW cases (>95%) occur sporadically due to congenital presence of accessory pathways without identifiable genetic mutations. 1, 7
  • The anatomical substrate is a direct muscular connection between atria and ventricles that forms during cardiac development, typically without hereditary basis. 4
  • Prevalence in the general population is 0.1-0.3%, with most cases representing isolated congenital anomalies rather than inherited conditions. 4, 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Familial Wolff-Parkinson-White syndrome.

Journal of electrocardiology, 1982

Guideline

Monitoring and Management of Wolff-Parkinson-White Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Current management of the Wolff-Parkinson-White syndrome.

Journal of cardiac surgery, 1993

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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