What are the differential diagnoses for an initial palmoplantar skin rash potentially caused by Augmentin (amoxicillin-clavulanate), including conditions other than drug eruption?

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Differential Diagnosis of Palmoplantar Rash in Augmentin Use

The palmoplantar rash from Augmentin is most likely a drug eruption (hypersensitivity reaction), but the differential diagnosis must include infectious causes (hand-foot-mouth disease, secondary syphilis, Rocky Mountain spotted fever), inflammatory conditions (palmoplantar pustulosis, dyshidrotic eczema, psoriasis), and other drug-induced patterns (fixed drug eruption, erythema multiforme). 1

Primary Consideration: Drug-Induced Eruption

The FDA label for amoxicillin-clavulanate explicitly lists skin rashes and urticaria as occurring in 3% of patients, with serious hypersensitivity reactions including erythema multiforme and Stevens-Johnson syndrome reported in post-marketing surveillance 1. The timing (typically within days to 2 weeks of starting therapy) and distribution pattern are critical diagnostic clues 2.

Specific Drug Eruption Patterns to Consider:

  • Maculopapular/exanthematous eruption: The most common drug reaction pattern, presenting as symmetric erythematous macules and papules that may involve palms and soles 2

  • Fixed drug eruption: Presents as well-demarcated erythematous plaques that recur in the same location with re-exposure; palmoplantar involvement is possible 2

  • Erythema multiforme: Target lesions with acral distribution including palms and soles; can be triggered by beta-lactam antibiotics 1, 2

  • Urticarial reaction: Pruritic wheals that may involve any body surface including palmoplantar areas 1, 2

Critical Differential Diagnoses Beyond Drug Eruption

Infectious Etiologies:

  • Hand-foot-mouth disease (HFMD): Caused by enteroviruses (CV-A6, CV-A16, enterovirus 71); presents with vesicles on palms, soles, and oral mucosa—the vesicular morphology distinguishes it from drug eruptions 3

  • Secondary syphilis: Can present with palmoplantar macules or papules; requires serologic testing (RPR/VDRL with confirmatory treponemal testing) 3

  • Rocky Mountain spotted fever: Blanching pink macules evolving to petechiae involving palms and soles, accompanied by fever and tick exposure history 3

Inflammatory Dermatoses:

  • Palmoplantar pustulosis (PPP): Chronic inflammatory condition with sterile pustules on palms and soles; distinguished by pustular rather than maculopapular morphology and chronic recurrent course 4

  • Dyshidrotic eczema: Intensely pruritic vesicles on lateral fingers, palms, and soles; vesicles are deeper-seated than HFMD 4

  • Psoriasis (palmoplantar variant): Well-demarcated erythematous plaques with silvery scale; chronic course distinguishes from acute drug eruption 5, 4

  • Hyperkeratotic hand eczema: Thickened, fissured skin on palms; chronic presentation unlike acute drug reaction 5

Other Drug-Induced Patterns (if patient on multiple medications):

  • Chemotherapy-induced palmar-plantar erythrodysesthesia (PPES): Caused by agents like capecitabine, 5-fluorouracil, doxorubicin; progresses from dysesthesia and tingling to burning pain, swelling, erythema, and potentially blisters 5, 6, 7

  • Hand-foot skin reaction (HFSR): Associated with BRAF/MEK inhibitors and tyrosine kinase inhibitors; presents with painful hyperkeratosis at pressure-bearing areas 5

  • Lichenoid drug eruption: Lichen planus-like lesions with possible palmoplantar hyperkeratosis; reported with various medications including imatinib 8

Systemic Conditions:

  • Kawasaki disease (primarily pediatric): Erythema and edema of palms/soles with subsequent periungual desquamation at 2-3 weeks, but lacks vesicles; accompanied by fever, conjunctivitis, and other systemic features 3

  • Drug hypersensitivity syndrome (DRESS): Severe reaction with facial edema, diffuse rash (may include palmoplantar involvement), fever, lymphadenopathy, eosinophilia, and organ involvement; typically occurs 2-8 weeks after drug initiation 9

Diagnostic Approach

Immediately assess for severe cutaneous adverse reactions (SCARs): Look for mucosal involvement, facial edema, confluent erythema, skin pain, blistering, or systemic symptoms (fever, lymphadenopathy, organ dysfunction) that would indicate Stevens-Johnson syndrome, toxic epidermal necrolysis, or DRESS 1, 9. If present, discontinue Augmentin immediately and consider hospitalization 1.

Key Historical Elements:

  • Timing: Drug eruptions typically appear 7-14 days after first exposure or within 1-3 days on re-exposure 2
  • Morphology: Vesicles suggest HFMD or dyshidrotic eczema; pustules suggest PPP; target lesions suggest erythema multiforme; maculopapular pattern suggests exanthematous drug eruption 3, 2, 4
  • Associated symptoms: Burning/tingling suggests PPES (if on chemotherapy); pruritus is common in drug eruptions; pain suggests HFSR or severe drug reaction 5
  • Concurrent medications: Review for chemotherapy agents, TKIs, or other drugs associated with palmoplantar reactions 5, 7
  • Infectious exposures: Sick contacts (HFMD), tick exposure (RMSF), sexual history (syphilis) 3

Physical Examination Priorities:

  • Distribution: Symmetric involvement favors drug eruption; unilateral or asymmetric suggests other causes 2
  • Mucosal involvement: Oral lesions with palmoplantar rash suggest HFMD, erythema multiforme, or SJS/TEN 3, 1, 2
  • Periorbital sparing: Typical of EGFR inhibitor reactions, not expected with Augmentin 5
  • Systemic signs: Fever, lymphadenopathy, or hepatosplenomegaly suggest DRESS, Kawasaki disease, or infectious etiology 3, 9

Common Pitfalls

  • Mononucleosis misdiagnosis: Patients with infectious mononucleosis who receive amoxicillin develop an erythematous rash in a high percentage of cases; this is not a true allergy but rather a drug-virus interaction 1. Consider monospot or EBV serology if clinical suspicion exists.

  • Delayed recognition of DRESS: Unlike typical drug eruptions, DRESS can present 2-8 weeks after drug initiation with systemic involvement; check CBC with differential, liver enzymes, and renal function if systemic symptoms present 9

  • Confusing PPES with drug allergy: If patient is on chemotherapy (particularly capecitabine, 5-FU, or doxorubicin), the palmoplantar eruption may be PPES rather than Augmentin-related; PPES has characteristic progression from dysesthesia to painful erythema 5, 6, 7

References

Research

Drug eruptions: approaching the diagnosis of drug-induced skin diseases.

Journal of drugs in dermatology : JDD, 2003

Guideline

Hand-Foot-Mouth Disease Diagnosis and Differential Diagnosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Palmar Erythema Causes and Associations

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Differential diagnosis of severe cutaneous drug eruptions.

American journal of clinical dermatology, 2003

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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