What does a high CFF (Clot Formation Time) MA (Maximum Amplitude) on a TEG (Thromboelastography) test indicate?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: December 7, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

High CFF MA on TEG Indicates Hypercoagulability from Elevated Fibrinogen

A high Maximum Amplitude (MA) on Citrated Functional Fibrinogen (CFF) TEG indicates a hypercoagulable state driven by elevated fibrinogen levels and increased clot strength, which increases thrombotic risk and may warrant anticoagulation therapy. 1

Understanding CFF MA

Maximum Amplitude (MA) represents the maximum strength of the clot and primarily reflects fibrinogen contribution when measured on the CFF assay. 1 The CFF test uses a platelet inhibitor (GPIIb/IIIa antagonist) to isolate fibrinogen's contribution to clot strength, distinguishing it from platelet-mediated clot firmness. 2

  • Normal MA values typically range from 50-70 mm, though specific reference ranges vary by device and clinical context. 3
  • Increased MA indicates hypercoagulability, which correlates with enhanced thrombotic risk. 1
  • CFF MA values are consistently higher than ROTEM FIBTEM MCF values for the same fibrinogen concentration—for example, at Clauss fibrinogen levels of 1.9-2.1 g/L, median CFF MA is 16.3 mm versus FIBTEM MCF of 12.0 mm. 4

Clinical Significance and Thrombotic Risk

High CFF MA reflects elevated fibrinogen levels and predicts increased risk for thrombotic events. 1, 5

  • Elevated MA has been associated with higher risk for ischemic events in cardiovascular disease patients. 5
  • In stroke patients, higher MA tertiles independently predict unfavorable one-year functional outcomes (OR = 1.192, p = 0.022). 5
  • Pregnancy naturally increases MA values due to physiologic hypercoagulability, with progressive increases throughout gestation. 2

Management Approach

For severe hypercoagulability with high thrombotic risk, consider therapeutic anticoagulation with low molecular weight heparin (LMWH), particularly in pregnant patients where LMWH is preferred over unfractionated heparin. 1

  • In pregnancy, 40 mg enoxaparin demonstrates greater anticoagulant effect than 7500 IU unfractionated heparin, with 73% of patients becoming hypocoagulable versus only 47% with UFH. 1
  • The anticoagulant effect on TEG is defined as a difference >25% between plain and heparinase R time samples. 2
  • Women with thrombophilia receiving prophylactic LMWH show significant increases in MCF (the ROTEM equivalent of MA) while clotting times remain unchanged. 2

Special Clinical Contexts

Pregnancy and Preeclampsia

  • Mild preeclampsia patients are more hypercoagulable than healthy pregnant women, but as severity worsens, coagulability paradoxically decreases, particularly when platelet counts fall below 100,000/mm³. 2
  • Women with VTE risk factors post-delivery maintain similar postpartum hypercoagulability patterns regardless of delivery mode. 2
  • Higher fibrinogen targets (>2.0 g/L) may be needed in obstetric hemorrhage management. 3

Obesity

  • Increasing BMI shows a trend toward greater hypercoagulability, though the hypercoagulable effect of term pregnancy appears greater in magnitude than obesity alone. 2

Critical Pitfalls to Avoid

Anemia can paradoxically show hypercoagulable TEG results (including elevated MA) due to decreased blood viscosity, potentially masking true coagulopathy or exaggerating hypercoagulability. 1, 3

  • TEG shows high coefficients of variance (7.1-39.9% for TEG parameters), requiring interpretation in clinical context. 1
  • Results are not interchangeable between TEG and ROTEM devices—CFF MA values differ significantly from FIBTEM MCF values even at identical fibrinogen concentrations. 1, 4
  • TEG is performed at 37°C and cannot assess hypothermia effects on coagulation. 1
  • Standard TEG is insensitive to antiplatelet agents and cannot reliably detect platelet dysfunction. 1, 3

Monitoring and Follow-up

  • Repeat TEG 15-30 minutes after any anticoagulation intervention to assess response. 1
  • Consider correlation with Clauss fibrinogen levels, though be aware that individual Clauss values correspond with different CFF MA values. 4
  • Amplitude at 10 minutes (A10) can predict final MA, allowing faster clinical decision-making without waiting for complete test results. 6

References

Guideline

Interpreting Thromboelastography (TEG) Parameters and Clinical Applications

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Correcting Low Maximum Amplitude on Thromboelastography

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.