What medications can cause hyperaldosteronism?

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Medications That Cause Hyperaldosteronism

The medications that most commonly cause true hyperaldosteronism (elevated aldosterone levels) are systemic corticosteroids, particularly fludrocortisone, which directly increases aldosterone-like activity through mineralocorticoid receptor activation. 1, 2

However, it's critical to distinguish between true hyperaldosteronism (elevated aldosterone) and pseudohyperaldosteronism (suppressed aldosterone with mineralocorticoid-like effects), as the latter is far more common with medication use.

Medications Causing True Hyperaldosteronism

Systemic Corticosteroids

  • Fludrocortisone is the primary medication that causes true mineralocorticoid excess, acting on distal renal tubules to enhance sodium reabsorption and increase potassium and hydrogen ion excretion 2
  • Other systemic corticosteroids (dexamethasone, methylprednisolone, prednisone, prednisolone) can elevate blood pressure through mineralocorticoid effects, though less potently than fludrocortisone 1

Medications Causing Pseudohyperaldosteronism (Suppressed Aldosterone)

These agents mimic aldosterone's effects while actually suppressing both renin and aldosterone levels—this is the more common medication-related scenario:

Licorice and Glycyrrhizin-Containing Products

  • Glycyrrhizic acid (licorice) inhibits 11-beta-hydroxysteroid dehydrogenase type-2, allowing cortisol to activate mineralocorticoid receptors, causing hypertension, hypokalemia, and metabolic alkalosis with suppressed renin and aldosterone 3, 4, 5
  • Found in herbal supplements, confectionary products, and some traditional medicines 4, 5
  • Can cause life-threatening refractory hypokalemia and hypertensive crisis 5
  • Drug interactions: Cilostazol combined with glycyrrhizin can precipitate severe pseudohyperaldosteronism even in patients who previously tolerated glycyrrhizin alone 6

Other Medications That Raise Blood Pressure (But Don't Cause Hyperaldosteronism)

The following medications elevate blood pressure through non-aldosterone mechanisms and should not be confused with causes of hyperaldosteronism:

  • Oral contraceptives 1
  • NSAIDs (antagonize antihypertensive effects but don't cause hyperaldosteronism) 1
  • CNS stimulants (amphetamines, methylphenidate) 1
  • Decongestants (pseudoephedrine, phenylephrine) 1
  • Immunosuppressants (cyclosporine) 1
  • Antidepressants (MAOIs, SNRIs, TCAs) 1
  • Recreational drugs (cocaine, methamphetamine, anabolic steroids) 1
  • Herbal supplements (ephedra/ma huang, St. John's wort with MAOIs) 1

Clinical Pitfalls to Avoid

  • Don't confuse medication-induced hypertension with hyperaldosteronism—most blood pressure-elevating medications do NOT cause aldosterone excess 1
  • Always obtain a comprehensive medication history including over-the-counter products, herbal supplements, dietary habits (especially licorice-containing foods), and recreational drugs when evaluating hypertension with hypokalemia 1, 4
  • Pseudohyperaldosteronism from licorice presents identically to primary hyperaldosteronism (hypertension, hypokalemia, metabolic alkalosis) but has suppressed renin and aldosterone levels, not elevated aldosterone 3, 4, 5
  • Licorice toxicity can be refractory to potassium replacement and requires discontinuation of the offending agent for resolution 5

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Syndromes that Mimic an Excess of Mineralocorticoids.

High blood pressure & cardiovascular prevention : the official journal of the Italian Society of Hypertension, 2016

Research

Pseudo Hyperaldosteronism Secondary to Herbal Medicine Use.

Journal of community hospital internal medicine perspectives, 2022

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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