What is the difference between Charcot-Bouchard (small aneurysms) and Saccular (arterial) aneurysms?

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Last updated: December 16, 2025View editorial policy

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Main Differences Between Charcot-Bouchard and Saccular Aneurysms

Charcot-Bouchard aneurysms are small microaneurysms (typically <1mm) that form in deep penetrating arteries like lenticulostriate vessels due to chronic hypertension, while saccular aneurysms are larger berry-like outpouchings that occur at major arterial bifurcations of the Circle of Willis.

Size and Morphology

Charcot-Bouchard aneurysms:

  • Microscopic in size, typically measuring 0.3-1.0 mm in diameter
  • Multiple small dilations along penetrating vessels
  • Not visible on standard angiography due to their minute size

Saccular aneurysms:

  • Range from small (<5mm) to giant (≥25mm) 1
  • Defined morphologically as having a distinct neck and dome structure 1
  • Classified as saccular when axial and lateral dimensions are nearly equal 1
  • Readily visible on angiography and imaging studies 1

Location

Charcot-Bouchard aneurysms:

  • Located on small penetrating arteries including lenticulostriate arteries, pontine perforators, and posterior inferior cerebellar artery branches 2
  • Deep brain structures: basal ganglia, thalamus, pons, cerebellum
  • These are NOT typical sites for saccular aneurysms 2

Saccular aneurysms:

  • Occur at major arterial bifurcations of the Circle of Willis 2, 3
  • Most common locations include the anterior communicating artery (34.8% of ruptured cases), posterior communicating artery, middle cerebral artery bifurcation, and internal carotid artery 2, 4
  • Proximal locations on or near the Circle of Willis represent the typical pattern 2

Pathophysiology

Charcot-Bouchard aneurysms:

  • Result from chronic hypertensive damage to small vessel walls
  • Associated with lipohyalinosis and fibrinoid necrosis
  • Represent end-organ damage from longstanding hypertension

Saccular aneurysms:

  • Develop from combination of congenital/inherited arterial wall defects and acquired degenerative vascular disease 3
  • Form at hemodynamic stress points where arterial branching occurs 2, 3
  • Associated with flow-related wall stress at bifurcations 5

Clinical Presentation

Charcot-Bouchard aneurysms:

  • Cause deep intracerebral hemorrhage (hypertensive hemorrhage)
  • Bleeding occurs in basal ganglia, thalamus, pons, or cerebellum
  • Associated with chronic, poorly controlled hypertension

Saccular aneurysms:

  • Typically present with subarachnoid hemorrhage when ruptured 1, 3
  • May present with mass effect symptoms, cranial nerve compression, or remain asymptomatic until rupture 1
  • Rupture risk correlates with size, with aneurysms <5mm having very low rupture rates while those >10mm have significant rupture risk 3

Treatment Implications

Charcot-Bouchard aneurysms:

  • Not amenable to surgical or endovascular treatment due to microscopic size
  • Management focuses on blood pressure control and prevention

Saccular aneurysms:

  • Treatable with surgical clipping or endovascular coiling 1
  • Treatment thresholds typically at ≥5.5 cm for men or ≥5.0 cm for women in aortic locations 1
  • For intracranial saccular aneurysms, treatment decisions based on size, location, morphology, and rupture risk 1

References

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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