What is the management of hyponatremia?

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Management of Hyponatremia

Initial Assessment and Classification

Hyponatremia is defined as serum sodium <135 mmol/L and should be further investigated when levels are <131 mmol/L. 1

Your initial workup must include:

  • Serum and urine osmolality 1
  • Urine sodium concentration 1
  • Uric acid level 1
  • Assessment of extracellular fluid volume status (hypovolemic, euvolemic, or hypervolemic) 1, 2

Volume status assessment is critical but challenging—physical examination alone has poor accuracy (sensitivity 41.1%, specificity 80%). 1 Look specifically for:

  • Hypovolemic signs: orthostatic hypotension, dry mucous membranes, decreased skin turgor, flat neck veins 1
  • Hypervolemic signs: peripheral edema, ascites, jugular venous distention 1
  • Euvolemic: absence of both hypovolemic and hypervolemic signs 1

A urine sodium <30 mmol/L predicts response to saline with 71-100% positive predictive value in hypovolemic states. 1 Serum uric acid <4 mg/dL suggests SIADH with 73-100% positive predictive value. 1


Treatment Based on Symptom Severity

Severe Symptomatic Hyponatremia (Seizures, Coma, Altered Mental Status)

For severe symptoms, immediately administer 3% hypertonic saline with a target correction of 6 mmol/L over 6 hours or until severe symptoms resolve. 1, 3

  • Give 100 mL boluses of 3% saline over 10 minutes, repeatable up to three times at 10-minute intervals until symptoms improve 1
  • Total correction must not exceed 8 mmol/L in 24 hours to prevent osmotic demyelination syndrome 1, 2, 3
  • Consider ICU admission for close monitoring 1
  • Monitor serum sodium every 2 hours during initial correction 1

Calculate sodium deficit: Desired increase in Na (mEq/L) × (0.5 × ideal body weight in kg) 1

Mild to Moderate Symptomatic or Asymptomatic Hyponatremia

Treatment depends on volume status (see below). The correction rate should not exceed 8 mmol/L in 24 hours. 1, 2


Treatment Based on Volume Status

Hypovolemic Hyponatremia

Discontinue diuretics and administer isotonic saline (0.9% NaCl) for volume repletion. 2, 4

  • Initial infusion rate: 15-20 mL/kg/h, then 4-14 mL/kg/h based on response 1
  • Urine sodium <30 mmol/L confirms appropriate diagnosis 1
  • Once euvolemic, reassess and adjust management 1

Euvolemic Hyponatremia (SIADH)

Fluid restriction to 1 L/day is the cornerstone of treatment for SIADH. 1, 2, 5

  • If no response to fluid restriction, add oral sodium chloride 100 mEq three times daily 1
  • For resistant cases, consider pharmacological options: 1
    • Urea (40 g in 100-150 mL normal saline every 8 hours) 1
    • Loop diuretics 1
    • Demeclocycline 1
    • Lithium 1
    • Vaptans (tolvaptan 15 mg once daily, titrate to 30-60 mg) 2, 6

Critical distinction: In neurosurgical patients, differentiate SIADH from cerebral salt wasting (CSW), as CSW requires volume and sodium replacement, NOT fluid restriction. 1, 2 CSW is characterized by true hypovolemia with CVP <6 cm H₂O, high urine sodium >20 mmol/L despite volume depletion, and evidence of extracellular volume depletion. 1

Hypervolemic Hyponatremia (Heart Failure, Cirrhosis)

Implement fluid restriction to 1-1.5 L/day for serum sodium <125 mmol/L. 1, 2, 4

  • Discontinue diuretics temporarily if sodium <125 mmol/L 2
  • In cirrhotic patients, consider albumin infusion alongside fluid restriction 2
  • Avoid hypertonic saline unless life-threatening symptoms are present, as it worsens ascites and edema 1, 2
  • For persistent severe hyponatremia despite fluid restriction, consider vaptans (tolvaptan) with extreme caution 2, 6

Important: In cirrhosis, it is sodium restriction (not fluid restriction) that results in weight loss, as fluid passively follows sodium. 2


Critical Correction Rate Guidelines

The maximum correction rate is 8 mmol/L in 24 hours for all patients. 1, 2, 3

High-risk patients require even slower correction (4-6 mmol/L per day): 1, 2

  • Advanced liver disease
  • Alcoholism
  • Malnutrition
  • Prior encephalopathy
  • Severe hyponatremia (<120 mmol/L)
  • Hypokalemia, hypophosphatemia

If overcorrection occurs (>8 mmol/L in 24 hours):

  • Immediately discontinue current fluids and switch to D5W (5% dextrose in water) 2
  • Consider administering desmopressin to slow or reverse the rapid rise 2
  • Target relowering to bring total 24-hour correction to ≤8 mmol/L 2

Special Considerations for Neurosurgical Patients

Cerebral Salt Wasting (CSW)

CSW should be treated with replacement of sodium and intravenous fluids, NOT fluid restriction. 1, 2

  • For severe symptoms: 3% hypertonic saline plus fludrocortisone (0.1-0.2 mg daily) in ICU 1
  • Hydrocortisone may be used to prevent natriuresis in subarachnoid hemorrhage patients 1
  • Never use fluid restriction in CSW—this worsens outcomes 1, 2

Subarachnoid Hemorrhage

  • Fludrocortisone may be considered in patients at risk of vasospasm 1
  • Do not treat with fluid restriction in patients at risk of vasospasm 1
  • Hydrocortisone may prevent natriuresis 1

Pharmacological Interventions

Vaptans (Vasopressin Receptor Antagonists)

Tolvaptan is FDA-approved for euvolemic and hypervolemic hyponatremia. 6

  • Starting dose: 15 mg once daily, titrate to 30-60 mg based on response 6
  • Avoid fluid restriction during first 24 hours to prevent overly rapid correction 6
  • Monitor sodium every 8 hours initially 6
  • Contraindicated with strong CYP3A inhibitors (ketoconazole increases tolvaptan AUC 5.4-fold) 6
  • Use with extreme caution in cirrhosis due to 10% risk of gastrointestinal bleeding (vs. 2% placebo) 6

Monitoring and Prevention of Complications

Monitoring Schedule

  • Severe symptoms: Check serum sodium every 2 hours during initial correction 1
  • Mild symptoms: Check every 4 hours after symptom resolution 1
  • Continue daily monitoring until stable 1

Osmotic Demyelination Syndrome

Watch for signs typically occurring 2-7 days after rapid correction: 1, 2

  • Dysarthria
  • Dysphagia
  • Oculomotor dysfunction
  • Quadriparesis

Prevention is key—never exceed 8 mmol/L correction in 24 hours. 1, 2, 3


Common Pitfalls to Avoid

  • Overly rapid correction (>8 mmol/L in 24 hours) leading to osmotic demyelination syndrome 1, 2
  • Using fluid restriction in cerebral salt wasting—this worsens outcomes 1, 2
  • Inadequate monitoring during active correction 1
  • Failing to recognize and treat the underlying cause 1
  • Using hypertonic saline in hypervolemic hyponatremia without life-threatening symptoms 1, 2
  • Ignoring mild hyponatremia (130-135 mmol/L) as clinically insignificant—it increases fall risk (21% vs. 5%) and mortality (60-fold increase) 1, 3
  • Using normal saline in SIADH or hypervolemic states—this can worsen hyponatremia 1

References

Guideline

Management of Hyponatremia

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Management of Sodium Imbalance

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Management of hyponatremia.

American family physician, 2004

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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