Does lisinopril (angiotensin-converting enzyme inhibitor) increase cortisol levels?

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Does Lisinopril Increase Cortisol?

No, lisinopril does not increase cortisol levels. There is no evidence in the medical literature that lisinopril, an ACE inhibitor, affects cortisol production or secretion.

Mechanism and Pharmacology

  • Lisinopril inhibits angiotensin-converting enzyme (ACE), which blocks the conversion of angiotensin I to angiotensin II, thereby reducing peripheral vascular resistance and blood pressure 1
  • The drug is not metabolized and is excreted unchanged in the urine, with no hepatic metabolism required for its pharmacologic effect 2, 3
  • Lisinopril's mechanism of action involves the renin-angiotensin-aldosterone system (RAAS), reducing plasma angiotensin II and aldosterone while increasing plasma renin activity 1
  • There is no interaction between ACE inhibitors and the hypothalamic-pituitary-adrenal (HPA) axis or cortisol synthesis pathways 4, 5

Clinical Evidence

  • Multiple comprehensive reviews of lisinopril's pharmacology and clinical effects make no mention of cortisol elevation or HPA axis effects 4, 2, 3, 1, 5
  • In diabetic patients treated with lisinopril, the drug does not adversely affect glycemic control or metabolic parameters, which would be expected if cortisol were elevated 4
  • Large-scale trials including ALLHAT, GISSI-3, and EUCLID that evaluated lisinopril in thousands of patients did not report cortisol-related adverse effects 6, 4, 5

Important Distinction from Other Medications

  • Etomidate, an anesthetic induction agent, is known to suppress the HPA axis and reduce cortisol response, but this is unrelated to ACE inhibitors 6
  • The Surviving Sepsis Campaign guidelines specifically warn about etomidate's adrenal suppression effects when used before steroid therapy, but make no such warnings about ACE inhibitors 6

Clinical Implications

  • If a patient on lisinopril presents with elevated cortisol, investigate other causes of hypercortisolism using standard diagnostic approaches including 24-hour urinary free cortisol, late-night salivary cortisol, and dexamethasone suppression testing 7, 8
  • Do not attribute elevated cortisol to lisinopril therapy, as this would delay appropriate workup for Cushing's syndrome or other causes of hypercortisolism 6, 7
  • Lisinopril can be safely continued during evaluation for hypercortisolism, as it does not interfere with cortisol measurement or HPA axis testing 8

References

Research

The clinical pharmacology of lisinopril.

Journal of cardiovascular pharmacology, 1987

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Diagnostic Approach to Elevated Morning Cortisol with Normal DHEA

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Cortisol Level Interpretation Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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