What are the key components of an electrocardiogram (EKG) reading?

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Last updated: December 20, 2025View editorial policy

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Key Components of an EKG Reading

A systematic EKG interpretation must evaluate five critical components in sequence: rate and rhythm, intervals (PR, QRS, QT), axis, waveform morphology (P waves, QRS complexes, ST segments, T waves), and any abnormalities in repolarization patterns.

Essential Waveforms and Intervals

P Wave and PR Interval

  • The P wave represents atrial depolarization and should be assessed for presence, morphology, and relationship to QRS complexes to determine rhythm origin 1.
  • The PR interval measures atrioventricular conduction time and helps identify conduction abnormalities 1.

QRS Complex

  • The QRS complex represents ventricular depolarization and must be evaluated for duration, amplitude, and morphology 1.
  • QRS duration and morphology changes directly affect ST-T wave interpretation through secondary repolarization abnormalities 1.
  • In bundle branch blocks, ST-segment and T-wave vectors are directed opposite to the mean QRS vector (left bundle branch block) or opposite to the slow terminal QRS component (right bundle branch block) 1.

ST Segment

  • The ST segment reflects the plateau phase of ventricular repolarization and is measured at the J point (junction of QRS and ST segment) 1.
  • ST-segment displacement must be referenced against the TP or PR segment as the baseline 1.
  • Normal ST elevation varies by age, sex, and race: in lead V2, the upper 98th percentile is approximately 0.3 mV in white men <40 years, 0.25 mV in white men ≥40 years, and 0.15 mV in white women 1.
  • Distinguish primary from secondary repolarization abnormalities: primary abnormalities indicate actual changes in ventricular myocyte repolarization (ischemia, electrolyte abnormalities, drugs), while secondary changes result from altered depolarization sequence without cellular repolarization changes 1.

T Wave

  • The T wave represents ventricular repolarization (phase 3 of the action potential) and should be assessed for amplitude, morphology, and axis 1.
  • When QRS axis is normal, an abnormal T-wave axis direction indicates primary repolarization abnormalities 1.
  • T-wave changes may be primary (from ischemia, myocarditis, drugs, electrolyte abnormalities, hyperventilation, position changes, catecholamines) or secondary (from conduction abnormalities) 1.

U Wave

  • The U wave is a low-amplitude deflection after the T wave, most evident in leads V2 and V3 with normal amplitude approximately 0.33 mV or 11% of T-wave amplitude 1, 2.
  • U waves are heart-rate dependent: rarely present at rates >95 bpm, present in 90% of cases at rates <65 bpm 1, 2.
  • Report U waves when inverted, merged with T wave, or amplitude exceeds T wave 1, 2.
  • Inverted U waves in V2-V5 are abnormal and may indicate acute ischemia or hypertension 1, 2.
  • Increased U-wave amplitude or U-T fusion occurs with hypokalemia, quinidine-like drugs, increased sympathetic tone, or long QT syndrome 1, 2.

QT Interval

  • The QT interval extends from QRS onset to T-wave end, representing total ventricular depolarization and repolarization time 1.
  • Measure QT in the lead showing the longest interval, usually V2 or V3; if this differs by >40 ms from other leads, the measurement may be erroneous 1.
  • When T and U waves are superimposed, measure QT in leads without U waves (often aVR, aVL) or extend the T-wave downslope tangent to the TP segment 1.
  • Visually validate all computer-generated QT measurements due to their critical importance for identifying arrhythmia risk 1.
  • QT prolongation requires clinical evaluation for causes including drugs, hypokalemia, hypocalcemia, and congenital long QT syndrome 1.

Critical Measurement Techniques

Lead Selection

  • QRS onset occurs up to 20 ms earlier in V2 and V3 than in limb leads, affecting interval measurements 1.
  • Modern digital ECG machines record all leads simultaneously, allowing temporal alignment and superimposition for more accurate measurement of QRS onset and T-wave end 1.

Common Pitfalls

  • Automated QT measurements may be longer than single-lead measurements because simultaneous recording captures the earliest QRS onset and latest T-wave end across all leads 1.
  • Low-frequency filtering removes baseline drift but prevents determination of absolute DC voltage levels, so ST elevation may reflect PR/TP depression, true ST elevation, or both 1.
  • Secondary repolarization changes from transient conduction abnormalities (ectopic beats, transient bundle branch blocks) usually revert promptly, but changes from prolonged ventricular pacing take hours to days to resolve 1.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

U Wave Characteristics and Clinical Significance

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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