What is the pathophysiology of croup?

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Pathophysiology of Croup

Croup results from viral infection causing inflammation, edema, and hypersecretion in the larynx, trachea, and bronchi, leading to upper airway obstruction that manifests as the characteristic barking cough, inspiratory stridor, and respiratory distress. 1

Viral Etiology and Mechanism

  • Parainfluenza viruses (types 1 and 2) are the primary causative agents, accounting for the majority of croup cases and up to 11% of all hospitalizations in children under 5 years of age 1
  • Other viral pathogens include respiratory syncytial virus (RSV), rhinoviruses, human coronaviruses, adenoviruses, influenza viruses, and human metapneumovirus 1
  • Influenza-associated croup tends to follow a more severe clinical course than parainfluenza-induced croup and carries higher risk of bacterial tracheitis as a complication 1

Anatomic and Physiologic Changes

The viral infection triggers a cascade of pathophysiologic changes in the upper airway:

  • Epithelial destruction or minimal epithelial changes occur, but all viral infections uniformly cause vasodilation and hypersecretion in the affected airways 1
  • The inflammation and edema primarily affect the subglottic region, larynx, trachea, and bronchi, creating varying degrees of upper airway obstruction 2, 3
  • The narrowing of the airway lumen produces the characteristic inspiratory stridor as turbulent airflow occurs through the constricted passage 4, 5

Clinical Manifestations of Airway Obstruction

The pathophysiologic changes translate into specific clinical features:

  • Inspiratory stridor results from turbulent airflow through the narrowed upper airway during inspiration 4, 2
  • Barking cough is produced by air passing through the inflamed, edematous larynx 5, 2
  • Hoarseness occurs due to laryngeal involvement and vocal cord edema 4
  • Respiratory distress develops when obstruction becomes severe enough to impair adequate ventilation 2

Age-Related Susceptibility

  • Croup predominantly affects children 6 months to 6 years of age, with peak incidence in those 6 months to 3 years 6, 4, 2
  • The anatomic characteristics of the pediatric airway—including smaller diameter, more compliant cartilage, and relatively larger tongue—make young children particularly vulnerable to symptomatic obstruction from even modest amounts of edema 2

Temporal Pattern

  • Croup typically presents in late fall or early winter with seasonal variation corresponding to parainfluenza virus circulation 6
  • The illness is usually preceded by upper respiratory infection symptoms, followed by the acute onset of barking cough and stridor 6, 5
  • Most children experience symptom resolution within 2 days, though the clinical course can vary 5

References

Guideline

Primary Cause of Croup

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Croup: Diagnosis and Management.

American family physician, 2018

Research

Recurrent Croup.

Pediatric clinics of North America, 2022

Research

Viral croup: a current perspective.

Journal of pediatric health care : official publication of National Association of Pediatric Nurse Associates & Practitioners, 2004

Research

Croup: an overview.

American family physician, 2011

Research

Croup.

The Journal of family practice, 1993

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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