What causes Strongyloides (threadworm) infection?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: December 23, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Causes of Strongyloides (Threadworm) Infection

Transmission Mechanism

Strongyloides stercoralis infection occurs when infective filariform larvae penetrate intact skin after contact with soil contaminated by human feces containing the parasite. 1, 2, 3

Primary Routes of Infection

  • Percutaneous transmission is the predominant route, occurring when bare skin contacts contaminated soil containing infective filariform larvae 2, 4
  • After skin penetration, larvae migrate through the venous system to the lungs, ascend the bronchi to the trachea, are coughed up and swallowed, then establish residence in the small intestine 2
  • Autoinfection represents a unique feature of this parasite, where filariform larvae can develop in the gut lumen and re-penetrate the intestinal mucosa or perianal skin without leaving the host, allowing infection to persist for decades 3, 4, 5

Geographic and Environmental Factors

  • The parasite is widespread throughout tropical and subtropical regions with poor sanitation, though small endemic foci exist in temperate regions including the rural southeastern United States 6, 2, 4
  • Globally, 300-600 million people are infected through contact with fecally contaminated soil 3
  • In the United States, strongyloidiasis is most commonly reported among refugees and immigrants from endemic areas 4

Less Common Transmission Routes

  • Intimate interhuman contact can rarely transmit infection 5
  • Solid organ transplantation from an infected donor represents another uncommon but documented route 5

Critical Autoinfection Cycle

The autoinfective component of the life cycle distinguishes Strongyloides from other helminths, as invasive filariform larvae can develop anew in the gut lumen, perpetuating cycles of penetration, tissue migration, and reproduction entirely within the host 3, 5. This mechanism explains why chronic infection can persist asymptomatically for decades after leaving endemic areas, and why immunosuppression can trigger catastrophic hyperinfection syndrome when large numbers of larvae pierce the bowel wall and disseminate throughout organs 3, 5, 7.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Cutaneous Strongyloides stercoralis infection: an unusual presentation.

Journal of the American Academy of Dermatology, 2003

Research

Human strongyloidiasis: complexities and pathways forward.

Clinical microbiology reviews, 2023

Guideline

Epidemiology and Clinical Manifestations of Strongyloidiasis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Strongyloides stercoralis: A Neglected but Fatal Parasite.

Tropical medicine and infectious disease, 2022

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.