What are the symptoms of growth hormone tumors?

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Symptoms of Growth Hormone Tumors

Growth hormone-secreting tumors present with dramatically different symptoms depending on whether they occur before or after epiphyseal closure: children develop accelerated growth velocity and tall stature (gigantism), while adults develop acromegalic features with tissue overgrowth. 1

Primary Clinical Manifestations in Children and Adolescents

The most prominent clinical feature of GH excess in children before epiphyseal closure is increased growth velocity. 1 Serial height measurements and photographs are particularly useful for timing disease onset. 1

Growth-Related Symptoms

  • Height >2 standard deviation scores (SDS) above age-adjusted and sex-adjusted normal values or 2 SDS above mid-parental target height 1
  • Persistently elevated growth velocity (>2 SDS) 1
  • Delayed bone age despite excessive height 1
  • Pubertal delay or arrested puberty (due to gonadotrophin inhibition from prolactin co-secretion or mass effects) 1

Acromegalic Features in Children

  • Acral enlargement (disproportionately enlarged hands and feet) 1
  • Coarsened facial features 1
  • Prognathism (jaw protrusion) 1
  • Dental malocclusion and teeth separation 1
  • Frontal bossing 1

Mass Effect Symptoms

  • Headache 1
  • Visual field defects (from optic chiasm compression) 1, 2
  • Cranial nerve palsies, particularly oculomotor nerve dysfunction with large tumors 2

Metabolic and Systemic Symptoms

  • Joint pain and arthritis 1, 3
  • Excessive sweating (hyperhidrosis) 1, 4
  • Insulin resistance and occasionally secondary diabetes mellitus 1
  • Hypertension 1
  • Sleep disturbance and sleep apnea 1
  • Increased appetite and BMI (particularly in X-linked acrogigantism) 1

Adult Acromegalic Manifestations

Adults with GH-secreting tumors develop the classic features of acromegaly, as epiphyseal closure prevents further linear growth. 3

Characteristic Physical Changes

  • Enlarged hands, feet, and facial features 3, 5
  • Prognathism and dental malocclusion 3
  • Coarsened facial features 3
  • Soft tissue swelling 3

Common Systemic Symptoms

  • Headaches 4, 5
  • Arthralgias and joint pain 3, 5
  • Fatigue 5
  • Hyperhidrosis (excessive sweating) 4, 5
  • Decreased libido (particularly with dual-staining tumors) 4

Associated Comorbidities

  • Glucose intolerance and diabetes mellitus 1, 3, 5
  • Hypertension 1, 3, 5
  • Carpal tunnel syndrome 1
  • Left ventricular hypertrophy and diastolic dysfunction 1
  • Sleep apnea 1
  • Kyphosis 1

Syndrome-Specific Presentations

X-Linked Acrogigantism

  • Tall stature onset before age 5 years (usually before 2 years) in all patients 1
  • Disproportionately enlarged hands, feet, and teeth separation 1
  • Acanthosis nigricans 1
  • Markedly increased BMI and appetite 1

McCune-Albright Syndrome

  • Early-onset GH excess (from 3 years onwards) 1
  • Café-au-lait pigmentation 1
  • Fibrous dysplasia 1
  • Precocious puberty or other hormone excess conditions 1

Carney Complex

  • Typical skin pigmentation 1
  • Myxomas 1
  • Testicular and adrenal disease 1

Hormonal Co-Secretion Symptoms

Hyperprolactinemia occurs in 65% of pediatric acromegaly cases and 34-36% of gigantism cases, causing additional symptoms. 1

  • Galactorrhea 1
  • Amenorrhea in females 1
  • Hypogonadism and loss of libido in males 1
  • Further pubertal delay (exacerbating gigantism) 1

Rare Ectopic Presentations

When GH excess results from ectopic GHRH-secreting tumors (carcinoid, pancreatic neuroendocrine tumors), additional symptoms may include: 6

  • Respiratory wheezing or dyspnea 6
  • Facial flushing 6
  • Peptic ulcers 6
  • Renal stones 6

Critical Diagnostic Indicators

Testing for GH excess should be offered to children and young people with excess height (>2 SDS) or consistently elevated height velocity and acromegalic features, with or without delayed/arrested puberty or family history of pituitary adenoma. 1

The diagnosis is supported by elevated serum IGF-1 levels (age-adjusted, sex-adjusted, and Tanner stage-matched) and failure of GH suppression below 1 μg/L after oral glucose load. 1

Almost 50% of pediatric gigantism cases have an identifiable genetic basis, making genetic assessment particularly important. 1 Evaluation for syndromic causes (MEN1, Carney complex, McCune-Albright syndrome) should be performed in all children with GH excess. 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Hypothalamic Tumors: Clinical Presentation and Diagnosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Acromegaly pathogenesis and treatment.

The Journal of clinical investigation, 2009

Research

[Current diagnosis and treatment of acromegaly].

Revista medica del Instituto Mexicano del Seguro Social, 2015

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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