Is there an increased risk of cardiovascular disease with ankylosing spondylitis?

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Last updated: December 26, 2025View editorial policy

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Increased Cardiovascular Risk in Ankylosing Spondylitis

Yes, patients with ankylosing spondylitis have a significantly increased risk of cardiovascular disease compared to the general population, with cardiovascular and cerebrovascular mortality elevated by approximately 36% overall. 1

Magnitude of the Risk

The cardiovascular disease risk in AS is substantially elevated and clinically significant:

  • Patients with AS demonstrate a 36% increased risk of cardiovascular and cerebrovascular death (adjusted HR 1.36,95% CI 1.13-1.65), with men showing even higher risk at 46% (HR 1.46,95% CI 1.13-1.87). 2

  • The standardized mortality ratios in AS range from 1.6 to 1.9, with circulatory disease being one of the main causes of death. 1

  • AS patients have approximately a two-fold higher prevalence of ischemic heart disease compared to controls. 3

Mechanisms Behind Increased Risk

The elevated cardiovascular risk stems from both traditional risk factors and disease-specific inflammatory processes:

  • Chronic systemic inflammation in AS directly contributes to accelerated atherosclerosis, independent of traditional cardiovascular risk factors. 1, 4

  • AS patients demonstrate increased subclinical atherosclerosis, with significantly greater carotid intima-media thickness (0.62 ± 0.09 mm vs 0.57 ± 0.09 mm in controls, p=0.02) even after adjusting for traditional cardiovascular risk factors. 5

  • Disease activity, number of flares, and duration of flares over time all contribute independently to cardiovascular risk. 1

  • AS patients have a higher prevalence of traditional cardiovascular risk factors than the general population, compounding their inflammatory risk. 3

Clinical Implications for Management

EULAR guidelines explicitly state that rheumatologists must be aware of and actively manage this increased cardiovascular risk:

  • Rheumatologists are responsible for ensuring cardiovascular disease risk management is performed in AS patients. 1

  • Cardiovascular risk assessment should be performed at least once every 5 years and reconsidered following major changes in antirheumatic therapy. 1

  • Disease activity should be controlled optimally to lower cardiovascular risk, as inflammation is a key modifiable risk factor. 1

AS-Specific Cardiac Manifestations

Beyond atherosclerotic disease, AS patients face additional cardiac complications:

  • Increased prevalence of cardiac abnormalities including aortic valve dysfunction and conduction disorders, though their precise contribution to overall cardiovascular risk requires further study. 1, 4

  • These AS-related cardiac manifestations appear to have an etiological link to the underlying inflammatory process. 4

Important Clinical Pitfalls

Common errors in managing cardiovascular risk in AS include:

  • Failing to recognize that cardiovascular risk management is a core responsibility of the rheumatologist, not just the primary care physician. 1

  • Underestimating the cardiovascular toxicity of NSAIDs and corticosteroids, which are commonly used in AS but carry cardiovascular risks that must be weighed against their anti-inflammatory benefits. 1

  • Not performing regular cardiovascular risk assessments every 5 years or after major treatment changes. 1

  • Inadequate control of disease activity, which independently drives cardiovascular risk through chronic inflammation. 1

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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