What are the specific clinical, biochemical, and imaging features of Pseudohypoparathyroidism (PHP) type 1b?

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From the Research

Pseudohypoparathyroidism type 1b (PHP1b) is characterized by resistance to parathyroid hormone (PTH) primarily in the kidneys, without the physical features of Albright hereditary osteodystrophy, and is typically associated with hypocalcemia, hyperphosphatemia, and elevated PTH levels, as reported in a recent case study 1.

Clinical Features

The clinical presentation of PHP1b includes symptoms such as paresthesias, muscle cramps, tetany, and seizures due to hypocalcemia. Patients with PHP1b typically have a normal physical appearance without short stature, round face, or brachydactyly, as noted in a study published in the Journal of pediatric endocrinology & metabolism 2.

  • Hypocalcemia
  • Hyperphosphatemia
  • Elevated PTH levels
  • Normal vitamin D status
  • Paresthesias
  • Muscle cramps
  • Tetany
  • Seizures

Biochemical Features

Biochemically, PHP1b shows resistance to PTH with impaired urinary cAMP and phosphate excretion in response to PTH administration.

  • Low serum calcium (typically 6.5-8.0 mg/dL)
  • Elevated phosphate (>5.0 mg/dL)
  • Markedly increased PTH levels (often 2-10 times normal)
  • Impaired urinary cAMP and phosphate excretion in response to PTH administration

Imaging Features

Imaging may reveal basal ganglia calcifications on brain CT, particularly in patients with longstanding hypocalcemia.

  • Basal ganglia calcifications on brain CT
  • Normal bone radiographs without evidence of rickets or osteomalacia

Molecular Features

PHP1b results from epigenetic alterations in the GNAS locus, specifically loss of methylation at the maternal GNAS A/B differentially methylated region, leading to decreased Gsα protein expression in renal proximal tubules, as demonstrated in a study published in the Journal of bone and mineral research 3.

  • Loss of methylation at the maternal GNAS A/B differentially methylated region
  • Decreased Gsα protein expression in renal proximal tubules

Treatment

Treatment focuses on correcting hypocalcemia with calcium supplements (1-3 g elemental calcium daily) and active vitamin D analogs (calcitriol 0.25-2.0 μg daily), with dose adjustments based on serum calcium, phosphate, and PTH levels, as recommended in a recent study 1.

  • Calcium supplements (1-3 g elemental calcium daily)
  • Active vitamin D analogs (calcitriol 0.25-2.0 μg daily)
  • Dose adjustments based on serum calcium, phosphate, and PTH levels

References

Research

Pseudohypoparathyroidism type 1B (PHP1B), a rare disorder encountered in adolescence.

Journal of pediatric endocrinology & metabolism : JPEM, 2020

Research

A Large Inversion Involving GNAS Exon A/B and All Exons Encoding Gsα Is Associated With Autosomal Dominant Pseudohypoparathyroidism Type Ib (PHP1B).

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2017

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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