Can Tylenol (acetaminophen) cause tinnitus?

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Last updated: December 29, 2025View editorial policy

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Can Tylenol Cause Tinnitus?

Yes, frequent acetaminophen (Tylenol) use is associated with an increased risk of developing persistent tinnitus, with the highest risk occurring in individuals using acetaminophen 6-7 days per week.

Evidence for Acetaminophen-Induced Tinnitus

The most robust evidence comes from a large longitudinal cohort study of 69,455 women followed over 22 years, which demonstrated that frequent acetaminophen use was independently associated with higher risk of incident persistent tinnitus 1. Specifically:

  • Women using acetaminophen 6-7 days per week had an 18% increased risk of developing persistent tinnitus compared to those using it less than 1 day per week (hazard ratio 1.18,95% CI 1.07-1.29) 1
  • The risk increased in a dose-dependent manner, with greater frequency of use correlating with higher tinnitus risk (p-trend=0.002) 1
  • This association remained significant after adjusting for multiple confounding variables 1

Clinical Context and Mechanism

Acetaminophen is recognized as one of over 130 drugs and chemicals with potential ototoxic effects 2. The mechanism likely involves biochemical and electrophysiological changes in the inner ear and eighth cranial nerve impulse transmission 2. While the exact pathophysiology remains unclear, ototoxicity from anti-inflammatory agents (including acetaminophen) is well-documented in the medical literature 2, 3.

Risk Stratification by Usage Pattern

The risk profile varies significantly based on frequency of use:

  • Low risk: Occasional use (<1 day/week) - no elevated risk demonstrated 1
  • Moderate risk: Intermittent use (4-5 days/week) - modestly elevated risk 1
  • Highest risk: Daily or near-daily use (6-7 days/week) - 18% increased risk 1

Important Clinical Considerations

Acetaminophen-induced tinnitus may be irreversible in some cases. While the longitudinal study examined incident tinnitus development rather than reversibility, case reports of other analgesic-induced ototoxicity (such as morphine) demonstrate that hearing-related adverse effects do not always resolve after drug discontinuation 4. One documented case required permanent hearing aid use despite stopping the offending medication 4.

Comparison with Other Analgesics

Acetaminophen is not unique in this risk - other commonly used analgesics also increase tinnitus risk 1:

  • NSAIDs (non-aspirin): 17% increased risk with 4-5 days/week use, 7% increased risk with 6-7 days/week use 1
  • Moderate-dose aspirin: 16% increased risk in women under 60 years using it 6-7 days/week 1
  • Low-dose aspirin: No elevated risk even with frequent use 1

Clinical Recommendations

When prescribing or recommending acetaminophen for chronic pain management:

  • Counsel patients about tinnitus risk, particularly those requiring daily or near-daily use 1
  • Monitor for early symptoms of tinnitus (ringing, buzzing, roaring sounds lasting >3 months) 5
  • Consider alternative analgesic strategies for patients requiring chronic pain management, weighing the specific risks of each agent 1
  • Refer promptly to audiology if tinnitus develops, as early intervention may improve outcomes 5

Critical Caveat

The association between acetaminophen and tinnitus does not definitively prove causation - the study was observational and could not account for all potential confounders, including the underlying conditions requiring analgesic use 1. However, the dose-response relationship, biological plausibility through known ototoxic mechanisms, and consistency with other analgesic findings support a likely causal relationship 1, 2.

References

Research

Morphine-induced hearing loss.

American journal of health-system pharmacy : AJHP : official journal of the American Society of Health-System Pharmacists, 2016

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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