Immediate Management of Ventricular Tachycardia
For patients presenting with ventricular tachycardia, immediately assess hemodynamic stability and perform synchronized direct current cardioversion without delay if the patient shows hypotension, altered mental status, chest pain, heart failure, or shock. 1, 2, 3
Initial Assessment
Determine hemodynamic stability first - this single determination drives all subsequent management decisions. 1, 2
Look specifically for:
- Hypotension or signs of shock 1, 2
- Altered mental status or syncope 1, 2
- Ischemic chest pain 1, 2
- Acute heart failure signs 1, 2
Obtain a 12-lead ECG immediately in hemodynamically stable patients to confirm the diagnosis and characterize the VT morphology. 1, 2
Presume any wide-complex tachycardia is VT when the diagnosis is unclear - treating supraventricular tachycardia as VT is safer than the reverse. 3
Management Algorithm Based on Hemodynamic Status
Hemodynamically Unstable VT
Perform immediate synchronized cardioversion starting with maximum output for any patient with hemodynamic instability. 1, 2, 3
- Begin with 200 J unsynchronized for polymorphic VT resembling ventricular fibrillation 3
- Use 100 J synchronized for monomorphic VT with rates >150 bpm 3
- Provide immediate sedation before cardioversion in conscious but hypotensive patients 1, 2, 3
- If no defibrillator is immediately available, attempt a precordial thump while preparing for cardioversion 4, 2
Do not delay cardioversion to attempt pharmacological conversion in unstable patients - this is a critical pitfall that worsens outcomes. 1, 2
Hemodynamically Stable VT
For stable patients, you have two first-line options:
Option 1: Electrical cardioversion remains appropriate even in stable patients and is most efficacious. 1, 5
Option 2: Pharmacological therapy if early termination is desired:
Intravenous procainamide is the preferred first-line agent for stable monomorphic VT, demonstrating the greatest efficacy for rhythm conversion. 3, 5
Intravenous amiodarone is preferred over procainamide in patients with heart failure, suspected myocardial ischemia, or impaired left ventricular function. 4, 1, 3, 6
Beta-blockers are first-line therapy unless contraindicated, particularly in the post-MI setting. 4
Intravenous lidocaine is only moderately effective and should be considered second-line. 4, 1
Special VT Subtypes
For polymorphic VT:
- Direct current cardioversion is first-line if hemodynamically compromised 3
- Intravenous beta-blockers for recurrent polymorphic VT, especially if ischemia suspected 3
- Intravenous amiodarone for recurrent polymorphic VT without QT prolongation 3
- Urgent revascularization should be considered when ischemia cannot be excluded 3
For LV fascicular VT (RBBB morphology with left axis deviation):
- Intravenous verapamil or beta-blockers may be considered 1, 2
- This is the ONLY scenario where calcium channel blockers are appropriate 1, 3
Distinguish accelerated idioventricular rhythm (ventricular rate <120 bpm) from true VT - this is usually a harmless reperfusion rhythm requiring no treatment. 4, 2
Post-Conversion Management
Monitor continuously for VT recurrence after successful conversion - recurrence is common. 1, 2
Evaluate and correct underlying causes: 4, 2
- Ongoing myocardial ischemia (most critical)
- Electrolyte abnormalities, particularly hypokalemia 4, 2
- Hypoxia and acid-base disturbances 4, 2
- Pump failure 4
Consider catheter ablation for: 1
- Ischemic heart disease with recurrent VT 1
- Scar-related heart disease with incessant VT or electrical storm (urgent ablation recommended) 1, 3
- First episode of sustained VT in patients with ischemic heart disease 1
Critical Pitfalls to Avoid
Never delay cardioversion in unstable patients while attempting pharmacological conversion - this is the most dangerous error. 1, 2
Avoid calcium channel blockers (verapamil, diltiazem) in patients with VT and structural heart disease - they may precipitate hemodynamic collapse and worsen outcomes. 1, 3
Do not assume wide-complex tachycardia is supraventricular - when uncertain, always treat as VT. 3
Avoid adenosine for irregular or polymorphic wide-complex tachycardias - it is inappropriate and potentially harmful. 2
Do not use prophylactic antiarrhythmic drugs other than beta-blockers - they may increase mortality. 2
Ensure proper amiodarone administration if selected: 6