What are the causes of vitamin B12 (cobalamin) deficiency in children?

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Causes of Vitamin B12 Deficiency in Children

Vitamin B12 deficiency in children primarily results from maternal deficiency transmitted through breastfeeding, malabsorption disorders, and inadequate dietary intake, with exclusively breastfed infants of deficient mothers representing the most common and preventable cause.

Maternal-Related Causes

Maternal Dietary Deficiency

  • Vegetarian and vegan maternal diets are the leading cause of infant B12 deficiency, as vitamin B12 is found exclusively in animal foods (meat, fish, poultry, cheese, milk, eggs) 1.
  • Exclusively breastfed infants develop deficiency between 2-12 months of age when maternal stores are inadequate 2, 3.
  • Infants born to deficient mothers have low hepatic B12 storage at birth, making them vulnerable even when mothers are asymptomatic 3.

Maternal Malabsorption Conditions

  • Maternal pernicious anemia (autoimmune destruction of intrinsic factor) causes infant deficiency through inadequate breast milk B12 content 4.
  • Maternal gastrectomy eliminates intrinsic factor production, leading to severe maternal and subsequent infant deficiency 5.
  • Maternal atrophic gastritis affecting the gastric body impairs B12 absorption and can affect breastfed infants 6.

Gastrointestinal Causes in Children

Surgical Causes

  • Distal ileum resection >20 cm causes permanent B12 malabsorption requiring lifelong supplementation, as the terminal ileum is the sole absorption site 1, 6.
  • Total or partial gastrectomy eliminates intrinsic factor production necessary for B12 absorption 7.
  • Bariatric surgery reduces hydrochloric acid and intrinsic factor availability 6.

Malabsorption Disorders

  • Celiac disease (gluten enteropathy) damages the small intestinal mucosa, impairing B12 absorption 1, 7.
  • Small bowel bacterial overgrowth competes for available B12 before absorption 7.
  • Fish tapeworm infestation (Diphyllobothrium latum) consumes dietary B12 7.
  • Inflammatory bowel disease, particularly ileal Crohn's disease, damages the absorption site 6.

Autoimmune Conditions

  • Pernicious anemia in children results from anti-intrinsic factor or anti-parietal cell antibodies preventing B12 absorption 4.
  • Autoimmune gastritis progressively destroys gastric parietal cells 8.

Medication-Induced Deficiency

Multiple medications interfere with B12 absorption or utilization 6, 8:

  • Metformin (especially >4 months use)
  • Proton pump inhibitors and H2 receptor antagonists (>12 months use)
  • Colchicine
  • Anticonvulsants (phenobarbital, pregabalin, primidone)
  • Sulfasalazine
  • Methotrexate

Inadequate Intake

Dietary Insufficiency

  • Strict vegetarian/vegan diets without supplementation provide no B12, as it occurs only in animal products 1, 7.
  • Inadequate dietary intake in children with restricted diets or poor appetite 1.

Increased Requirements

  • Pregnancy and lactation increase B12 requirements, potentially depleting maternal stores 7.
  • Conditions causing increased demand: thyrotoxicosis, hemolytic anemia, hemorrhage, malignancy, hepatic and renal disease 7.

Special Populations at Risk

Infants on Parenteral Nutrition

  • Long-term parenteral nutrition without adequate B12 supplementation (recommended 0.3 μg/kg/day for infants <12 months) 1.

Children with Chronic Kidney Disease

  • Children on dialysis have decreased intake, increased clearance, and dialysis-related losses of water-soluble vitamins including B12 1.

Genetic/Metabolic Causes

  • Congenital disorders of B12 metabolism including transcobalamin deficiency (TCN2 gene) and intracellular cobalamin metabolism defects (MMACHC, MMADHC, MTRR, MTR genes) 6.
  • These present with elevated methylmalonic acid despite normal or elevated serum B12 6.

Critical Clinical Pitfalls

Never administer folic acid before treating B12 deficiency, as folic acid may mask anemia while allowing irreversible neurological damage to progress 6, 7. This is particularly dangerous as doses of folic acid >0.1 mg/day can produce hematologic remission in B12-deficient patients while neurologic manifestations continue unchecked 7.

Vitamin B12 deficiency allowed to progress >3 months produces permanent degenerative spinal cord lesions 7. Symptoms in infants include vomiting, lethargy, failure to thrive, hypotonia, developmental regression, and abnormal movements (tremors, myoclonus) appearing between 2-12 months of age 2.

Always obtain maternal nutritional history in breastfed infants presenting with developmental delay or neurological symptoms, specifically asking about vegetarian/vegan diets, gastrectomy, or autoimmune conditions 2, 3, 4.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Vitamin B12 deficiency: case report and review of literature.

The Pan African medical journal, 2021

Research

[Vitamin B12 deficiency in an infant child of a mother with pernicious anemia].

Andes pediatrica : revista Chilena de pediatria, 2022

Research

Brain atrophy caused by vitamin B12-deficient anemia in an infant.

Journal of pediatric hematology/oncology, 2011

Guideline

Vitamin B12 and Magnesium Deficiency Diagnosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Management of Low Vitamin B12 with Negative IFA and PCA Tests

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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