Management of Tachycardia at 164 bpm
The immediate priority is determining hemodynamic stability and identifying whether this is a narrow or wide complex tachycardia on ECG, as this fundamentally dictates whether you proceed to immediate synchronized cardioversion versus pharmacologic management. 1
Initial Assessment Algorithm
Step 1: Assess Hemodynamic Stability
- Check for signs of instability: acute altered mental status, ischemic chest pain, acute heart failure, hypotension, or shock 1
- If ANY of these are present and attributed to the tachycardia → proceed immediately to synchronized cardioversion 1
- Apply supplementary oxygen and establish IV access while evaluating 1
Step 2: Obtain 12-Lead ECG (if patient is stable)
- Determine QRS width: narrow (<120 ms) versus wide (≥120 ms) 1
- Assess regularity of rhythm 1
- Do not delay cardioversion in unstable patients to obtain ECG 1
Management Based on Rhythm Type
If Hemodynamically UNSTABLE (Regardless of Rhythm)
Immediate synchronized cardioversion is indicated 1, 2
- Initial energy: 50-100 J for SVT 1, 3
- Sedate if conscious and time permits 1
- If unable to synchronize, use unsynchronized high-energy shocks 1
If Hemodynamically STABLE with Narrow Complex (<120 ms)
Critical caveat: At 164 bpm with narrow complex, this could be sinus tachycardia, SVT, atrial flutter, or atrial fibrillation. The approach differs significantly 1.
For Regular Narrow Complex Tachycardia (likely SVT/AVNRT):
If vagal maneuvers fail → Adenosine 6 mg IV rapid push 2, 4, 3
If adenosine fails → IV beta-blockers or calcium channel blockers 1
For Irregular Narrow Complex (likely Atrial Fibrillation/Flutter):
- Rate control with IV beta-blockers, diltiazem, or verapamil 1
- Atrial flutter often more difficult to rate-control than AF 1
- If rate control fails, consider synchronized cardioversion 1
Important distinction: If this is sinus tachycardia (upper limit ~220 minus patient's age), do NOT treat the rate - identify and treat the underlying cause (fever, hypovolemia, anemia, hypoxia) 1. Normalizing heart rate when it's compensatory can be detrimental 1.
If Hemodynamically STABLE with Wide Complex (≥120 ms)
Assume ventricular tachycardia until proven otherwise 1
- If definitely supraventricular with aberrancy, manage as narrow complex SVT 1
- If uncertain or likely VT: IV procainamide or amiodarone 1
- Amiodarone preferred if impaired LV function or heart failure 1
- If pharmacologic therapy fails → synchronized cardioversion at 100 J initial energy 1
Critical Contraindications
Never give adenosine, verapamil, or diltiazem if:
- Pre-excited atrial fibrillation suspected (irregular wide complex) - can precipitate ventricular fibrillation 3
- Wide complex tachycardia of uncertain etiology 1
Avoid calcium channel blockers in:
- Systolic heart failure 3, 5
- Heart block or sinus node dysfunction without pacemaker 1
- Known pre-excitation syndromes 1
Post-Conversion Management
- Monitor for recurrence and premature complexes 3
- Refer all patients to electrophysiology/cardiology for definitive management 1, 2
- Catheter ablation is first-line definitive therapy with 94.3-98.5% success rate 2, 4
- If ablation declined: oral beta-blockers, diltiazem, or verapamil for long-term management (80-98% success) 2, 4