Is alpha-1 antitrypsin (A1AT) replacement therapy initiated earlier in Europe compared to the United States, where it is typically not administered until emphysema is present?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: April 16, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the Guidelines

In Europe, alpha-1 antitrypsin (AAT) replacement therapy is generally provided earlier than in the United States, often before emphysema develops, as supported by the American Thoracic Society/European Respiratory Society statement 1. The primary goal of AAT replacement therapy is to slow the progression of lung damage by restoring protective levels of this important protease inhibitor.

  • The standard treatment involves weekly intravenous infusions of purified human AAT at a dose of 60 mg/kg, as shown to be biologically effective in increasing lung levels of AAT in AAT-deficient individuals 1.
  • European guidelines typically recommend initiating AAT replacement therapy for patients with severe AAT deficiency (serum levels <11 μmol/L or approximately 50-80 mg/dL) who have declining lung function, even without established emphysema.
  • In contrast, US guidelines usually require evidence of emphysema or other pulmonary disease before starting therapy, highlighting the difference in approach between the two regions. The rationale behind the European approach is that early intervention may slow the progression of lung damage by restoring protective levels of this important protease inhibitor, potentially preserving lung function before irreversible structural damage occurs, as suggested by observational studies such as the German–Danish study and the NHLBI Registry 1.
  • These studies suggest that progression of emphysema may be slowed in patients with moderate emphysema (FEV1 31–65% predicted), and mortality may be decreased in patients with a lower FEV1.
  • AAT deficiency causes lung damage through unopposed neutrophil elastase activity that destroys lung tissue, and earlier replacement aims to prevent this destructive process rather than treating established disease.

From the Research

Alpha 1 Antitrypsin Replacement Therapy in Europe and the United States

  • The availability and timing of alpha 1 antitrypsin replacement therapy in Europe compared to the United States are not explicitly stated in the provided studies 2, 3, 4, 5, 6.
  • However, it is mentioned that intravenous infusion of alpha-1 antitrypsin was approved by the United States Food and Drug Administration (FDA) in 1987 to treat emphysema associated with alpha-1 antitrypsin deficiency (AATD) 5.
  • The studies suggest that alpha-1 antitrypsin replacement therapy is typically considered for individuals with severe forms of AATD, and its effectiveness in treating less severe forms is still being researched 2, 4, 5.
  • There is no direct comparison between Europe and the United States regarding the timing of alpha 1 antitrypsin replacement therapy provision in the provided studies.

Timing of Alpha 1 Antitrypsin Replacement Therapy

  • One study suggests that therapy should ideally be started earlier than normally envisaged and before the onset of clinical emphysema 4.
  • Another study mentions that alpha-1 antitrypsin replacement therapy has proven clinical efficacy in slowing the decline of lung function associated with AATD progression, but it is only recommended for individuals with the most severe forms of AATD 5.
  • The optimal timing of alpha 1 antitrypsin replacement therapy is still a topic of research, and more studies are needed to determine the best approach 2, 3, 4, 5, 6.

Research and Emerging Treatment Strategies

  • There is currently extensive research into AATD and its treatment, with new therapeutic technologies being investigated, such as gene repair and other interference strategies, as well as the use of chemical chaperones 5.
  • New sources of alpha-1 antitrypsin are also being investigated to ensure there are enough supplies to meet future demand, and new methods of assessing response to treatment are being evaluated 5.
  • The COVID-19 pandemic has prompted physicians to develop additional strategies for delivering alpha-1 antitrypsin therapy, including self-administration, which can improve independence and well-being for patients with AATD 6.

Related Questions

Is alpha-1 antitrypsin (A1AT) replacement therapy initiated earlier in Australia compared to the United States, where it is typically not administered until emphysema is present?
What are the treatment recommendations for patients with low alpha 1 antitrypsin (A1AT) levels?
What is the best management approach for alpha 1 anti-trypsin disease patients who smoke?
Is weekly infusion of Alpha-1 proteinase inhibitor (Alpha-1 antitrypsin) at 60 mg/kg IV considered medically necessary and standard of care for a patient with alpha-1 antitrypsin deficiency and severe Chronic Obstructive Pulmonary Disease (COPD) with emphysema?
Is Prolastin (Alpha-1 Antitrypsin) home infusion medically indicated for a patient with Chronic Obstructive Pulmonary Disease (COPD) and Alpha-1 Antitrypsin Deficiency, with severe lung function impairment and documented emphysema?
What is the life expectancy for a patient with recurrent pancreatic cancer after a Whipple (pancreaticoduodenectomy) procedure?
What is the next step in managing a 76-year-old male with persistent hypertension despite being on amlodipine (10mg), valsartan (Valtaran) (320mg), furosemide (Lasix) (40mg), and metoprolol (25mg twice daily)?
What is the frequency of application for Enstilar (calcipotriene and betamethasone dipropionate)?
What are milk of calcium cysts?
Is alpha-1 antitrypsin (A1AT) replacement therapy initiated earlier in Australia compared to the United States, where it is typically not administered until emphysema is present?
How old must a tracheostomy site be?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.