Initial Treatment for Supraventricular Tachycardia (SVT)
Begin with immediate assessment of hemodynamic stability: if the patient is unstable (hypotension, altered mental status, shock, chest pain, or acute heart failure), proceed directly to synchronized cardioversion at 50-100J without attempting vagal maneuvers or medications; if stable, start with vagal maneuvers followed by adenosine if unsuccessful. 1, 2
Hemodynamically Unstable Patients
Perform immediate synchronized cardioversion without delay. 3, 1, 2
- Do not waste time with vagal maneuvers or pharmacologic therapy in unstable patients—every second counts and cardioversion is the definitive treatment. 2
- Start with 50-100J biphasic energy and increase stepwise if the initial shock fails. 1
- Have defibrillation capability immediately available in case the rhythm degenerates to ventricular fibrillation. 2
- Perform cardioversion after adequate sedation/anesthesia only if the patient is stable enough to tolerate brief delay. 3
Hemodynamically Stable Patients: Step-by-Step Algorithm
Step 1: Vagal Maneuvers (First-Line)
Perform vagal maneuvers immediately as Class I, Level B evidence supports this as first-line treatment. 3, 1
- Modified Valsalva maneuver is the most effective technique with 43% success rate and 2.8-3.8 times more effective than standard Valsalva. 1, 2, 4
- Technique: Patient bears down against closed glottis for 10-30 seconds (equivalent to 30-40 mm Hg intrathoracic pressure) while supine, then immediately lies flat with legs elevated. 3, 1
- Alternative: Carotid sinus massage for 5-10 seconds after confirming absence of bruit by auscultation (apply steady pressure over right or left carotid sinus). 3
- Ice-cold wet towel to face is another option based on diving reflex. 3
- Never apply pressure to the eyeball—this is dangerous and abandoned. 3
Step 2: Adenosine (If Vagal Maneuvers Fail)
Administer adenosine 6 mg rapid IV bolus as Class I, Level B recommendation with 90-95% effectiveness for terminating SVT. 3, 1, 2, 5
- Give as rapid IV push through large peripheral vein followed immediately by 20 mL saline flush. 1
- If unsuccessful, give 12 mg, then another 12 mg if needed. 1
- Critical dosing adjustments: Reduce to 3 mg for patients taking dipyridamole, carbamazepine, or with transplanted heart; larger doses may be needed with theophylline, caffeine, or theobromine. 1
- Have electrical cardioversion equipment immediately available as adenosine may precipitate atrial fibrillation that could conduct rapidly and cause ventricular fibrillation. 3, 1
- Contraindicated in asthma patients due to risk of severe bronchoconstriction. 1
- Expect minor, brief side effects (<1 minute) in approximately 30% of patients. 3
- Adenosine will also unmask atrial flutter or atrial tachycardia by causing transient AV block without terminating the rhythm. 2
Step 3: Alternative Pharmacologic Therapy (If Adenosine Fails or Contraindicated)
Intravenous diltiazem or verapamil are Class IIa, Level B recommendations for hemodynamically stable SVT. 3
- Intravenous beta blockers are also reasonable as Class IIa, Level C recommendation. 3
- These agents have 80-98% success rates for SVT termination. 3
Step 4: Synchronized Cardioversion (If Pharmacologic Therapy Fails)
Perform synchronized cardioversion after adequate sedation/anesthesia when medications are ineffective or contraindicated in stable patients. 3
- This is highly effective and is Class I, Level B recommendation. 3
Critical Safety Warnings and Pitfalls
Wide-Complex Tachycardia Warning
Never give AV nodal blocking agents (verapamil, diltiazem, adenosine, beta blockers) to patients with wide-complex tachycardia (QRS ≥120 ms) of uncertain etiology—treat as ventricular tachycardia until proven otherwise. 1, 2
- Giving verapamil or diltiazem for ventricular tachycardia can lead to hemodynamic collapse. 3
- Obtain 12-lead ECG immediately to determine if QRS is narrow (<120 ms) or wide (≥120 ms). 2
Pre-Excited Atrial Fibrillation Warning
For irregular wide-complex tachycardia (pre-excited AF), never give adenosine, verapamil, diltiazem, or beta blockers—these can accelerate ventricular rate and cause ventricular fibrillation. 3, 2
- Use synchronized cardioversion if unstable, or IV ibutilide/procainamide if stable. 3, 2
- These medications slow conduction over the accessory pathway rather than enhancing it. 3
Heart Failure Warning
Avoid verapamil and diltiazem in patients with systolic heart failure due to risk of hemodynamic collapse. 1
Post-Conversion Management
- Patients often have atrial or ventricular premature complexes immediately after conversion that may reinitiate tachycardia—an antiarrhythmic drug may be required to prevent acute recurrence. 3
- Arrange cardiology follow-up for consideration of catheter ablation, which has 94.3-98.5% single-procedure success rate. 2, 5
Special Consideration: Automatic Tachycardias
Automatic tachycardias (ectopic atrial tachycardia, multifocal atrial tachycardia, junctional tachycardia) are not responsive to cardioversion and require rate control with AV nodal blocking agents instead. 1