What is the diagnosis and mechanism of a 62-year-old woman with a 5-year history of progressive depigmentation patches that do not tan with adjacent skin and show no fluorescence on Wood's (Wood's ultraviolet lamp) examination?

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Diagnosis: Vitiligo

This is vitiligo, an acquired chronic depigmentation disorder characterized by progressive loss of functioning epidermal melanocytes, presenting as white patches that do not fluoresce under Wood's lamp examination. 1

Clinical Presentation Confirming Vitiligo

The clinical features in this case are classic for vitiligo:

  • Progressive depigmentation patches over 5 years indicate the chronic, stepwise nature of melanocyte destruction 1
  • Patches that do not tan with adjacent skin reflect complete loss of melanocytes, which are the melanin-producing cells responsible for tanning 2
  • No fluorescence on Wood's lamp is characteristic of vitiligo, as Wood's light delineates areas of complete pigment loss without enhancement (unlike some fungal infections that fluoresce) 1
  • No surface changes are expected in vitiligo skin, distinguishing it from inflammatory or scaly conditions 1

Pathogenic Mechanism

The pathogenesis involves autoimmune destruction of melanocytes, with multiple contributing factors:

Primary Autoimmune Mechanism

  • Autoimmune-mediated melanocyte destruction is the predominant mechanism, where immune activation leads to progressive loss of functioning epidermal and hair follicle melanocytes 3, 2
  • Approximately 34% of adults with vitiligo have autoimmune thyroid disease, reflecting the strong autoimmune connection 1, 4
  • The condition shows genetic predisposition with polygenic, multifactorial inheritance overlapping with other autoimmune disorders 5, 2

Contributing Pathogenic Factors

  • Increased oxidative stress contributes to melanocyte destruction 2
  • Genetic factors involving melanocyte-specific genes (DDR1, XBP1, NLRP1, PTPN22) play a role 2
  • Neural theory may explain distribution patterns, particularly in segmental variants where lesions follow dermatomes or Blaschko's lines 3

Essential Clinical Actions

Screen for thyroid function immediately, as the British Journal of Dermatology guidelines specifically recommend checking thyroid function in all adults with vitiligo given the 34% prevalence of autoimmune thyroid disease 1, 4

Diagnostic Confirmation

  • The diagnosis is straightforward in classical presentations and can be made confidently in primary care 1
  • Wood's light examination confirms complete depigmentation (no fluorescence) and helps assess disease extent 1

Common Pitfall

Failing to screen for thyroid disease is a critical error, as this represents a treatable comorbidity present in one-third of vitiligo patients 4, 3

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Pathogenesis of Vitiligo

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Autoimmune Connection in Vitiligo and Erythema Annulare Centrifugum

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

[Vitiligo. Diagnosis, differential diagnosis, and current patient management].

Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete, 2009

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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