Treatment of Hip Trochanteric Bursitis and Gluteal Tendon Tendinosis/Tendinitis
Start with conservative therapy including NSAIDs, physical therapy with eccentric hip abductor strengthening, and activity modification; if symptoms persist beyond 6 weeks, proceed to ultrasound-guided corticosteroid injection into the trochanteric bursa. 1
Initial Diagnostic Approach
- Obtain plain radiographs first to exclude other causes of hip pain such as arthritis, fractures, or bone tumors before proceeding with treatment 2
- Recognize that trochanteric bursitis and gluteus medius/minimus tendinosis frequently coexist and can be difficult to differentiate clinically 1, 3
- Consider ultrasound or MRI if diagnosis remains unclear after radiographs, as ultrasound can effectively detect both bursitis and evaluate gluteal tendons 2, 1
First-Line Conservative Treatment (0-6 Weeks)
Pharmacological Management
- Begin with NSAIDs for pain relief and anti-inflammatory effects 1
- For patients with cardiovascular disease or risk factors, consider acetaminophen (up to 4 grams daily) as first-line before NSAIDs 1
- Add proton-pump inhibitors in patients at risk for GI bleeding when NSAIDs are necessary 1
Physical Therapy Protocol
- Implement supervised exercise programs focusing on eccentric strengthening of hip abductor muscles (gluteus medius and minimus), which are more effective than passive interventions 1
- Include stretching exercises for the iliotibial band and lower back 4
- Apply cryotherapy (ice for 10-minute periods through a wet towel) for acute pain relief 1
- Prioritize land-based physical therapy over aquatic therapy 1
Activity Modification
- Reduce repetitive loading activities that aggravate lateral hip pain 1
- Avoid activities that increase pressure on the affected area 1
Second-Line Treatment (After 6 Weeks of Failed Conservative Therapy)
Corticosteroid Injection
- Inject 24 mg betamethasone with 1% lidocaine (or equivalent) into the trochanteric bursa under ultrasound guidance 1, 4
- Ultrasound guidance improves accuracy and ensures proper placement 1
- Use peritendinous rather than intratendinous injections to avoid deleterious effects on tendon substance 1
- This provides both diagnostic information and therapeutic benefit 1
Alternative Non-Invasive Option
- Consider extracorporeal shock wave therapy as a safe, noninvasive, and effective treatment for chronic tendinopathies after failed initial management 1, 5
- Systematic review evidence shows low-energy shock wave therapy superior to other nonoperative modalities 5
Refractory Cases (After 3-6 Months of Comprehensive Conservative Treatment)
Surgical Intervention
- Reserve surgery only after failure of 3-6 months of comprehensive conservative treatment 1
- Surgical options include:
- Surgical efficacy ranges from good to excellent outcomes in refractory cases based on VAS and Harris Hip Scores 5
Critical Clinical Pitfalls to Avoid
- Do not assume isolated bursitis—gluteus medius/minimus tendinosis coexists in most cases and may be the primary pathology 1, 7
- Avoid blind corticosteroid injections—always use ultrasound guidance to ensure proper bursal placement and avoid intratendinous injection 1
- In patients with hip prostheses, be cautious of misinterpreting adverse reactions to metal debris (ARMD) as trochanteric bursitis 1
- Do not rely solely on passive interventions (massage, ultrasound, heat)—these should only supplement active physical therapy 1
- Avoid premature surgical referral—most patients respond to conservative measures, with symptom resolution rates of 49-100% with appropriate nonoperative treatment 5
Treatment Algorithm Summary
- Weeks 0-6: NSAIDs + eccentric strengthening physical therapy + activity modification + cryotherapy
- Week 6+ (if persistent): Add ultrasound-guided corticosteroid injection OR extracorporeal shock wave therapy
- Months 3-6+ (if refractory): Consider surgical intervention (bursectomy, iliotibial band release, or tendon repair)
This stepped approach ensures appropriate escalation while recognizing that trochanteric bursitis is self-limiting in the majority of patients 5, 7.