What are the different types of shock?

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Types of Shock

Four Major Categories of Shock

Shock is classified into four major categories based on the primary organ system involved: hypovolemic (blood/fluid compartment), distributive (vascular system), cardiogenic (cardiac dysfunction), and obstructive (circulatory blockage). 1, 2


1. Hypovolemic Shock

Pathophysiology

  • Results from absolute intravascular volume loss leading to inadequate tissue perfusion 2, 3
  • Decreased cardiac output occurs secondary to insufficient preload 4

Hemodynamic Profile

  • Decreased cardiac index (<2.2 L/min/m²) 1
  • Decreased central venous pressure (CVP) 1
  • Decreased pulmonary capillary wedge pressure (PCWP) 1
  • Increased systemic vascular resistance (SVR) as compensatory mechanism 1
  • Tachycardia and decreased pulse pressure from reduced stroke volume 1
  • Decreased mixed venous oxygen saturation (SvO2 <70%) 1

Management

  • Immediate aggressive fluid resuscitation with balanced crystalloids is the definitive therapy—vasopressors are NOT primary treatment 5
  • Vasopressors may only be used transiently for life-threatening hypotension while simultaneously achieving hemorrhage control and volume restoration 5
  • Definitive control of bleeding source is essential 5

2. Distributive Shock

Pathophysiology

  • State of relative hypovolemia from pathological redistribution of intravascular volume due to widespread vasodilation 2
  • Most commonly septic shock, but includes neurogenic and anaphylactic shock 3, 6

Hemodynamic Profile

  • Normal or increased cardiac index in early stages 1
  • Decreased systemic vascular resistance (SVR) from pathological vasodilation 1, 7
  • Normal or decreased PCWP 1
  • Normal or decreased CVP 1
  • Clinical presentation includes hypotension, warm extremities, and elevated lactate 1

Neurogenic Shock Specifics

  • Presents with bradycardia (not tachycardia) due to loss of sympathetic tone 7
  • Decreased SVR without compensatory tachycardia distinguishes it from other shock types 7
  • Does NOT show elevated CVP or PCWP seen in cardiogenic shock 7

Management

  • Norepinephrine is first-line vasopressor after adequate fluid resuscitation with balanced crystalloids 5
  • Target mean arterial pressure (MAP) ≥65 mmHg 5, 7
  • Add vasopressin (up to 0.03 units/min) if hypotension persists despite norepinephrine 5
  • For neurogenic shock specifically: vasopressors are first-line after spinal immobilization, with norepinephrine preferred 7
  • Fluid resuscitation accompanies vasopressor therapy, though primary problem is vasodilation rather than volume depletion 7

3. Cardiogenic Shock

Pathophysiology

  • Results from primary cardiac dysfunction with inadequate cardiac output despite adequate preload 1, 2
  • Most commonly caused by acute myocardial infarction (7-10% of AMI cases) 1
  • Associated with 30-day mortality of 40-45% despite contemporary treatment 1

Hemodynamic Profile

  • Decreased cardiac index (<2.2 L/min/m²) 1
  • Increased PCWP (>15 mmHg, often >20 mmHg) from left ventricular failure 1
  • Increased CVP (>15 mmHg) from elevated right-sided filling pressures 1
  • Increased SVR as compensatory mechanism 1
  • Tachycardia (attempting to maintain cardiac output), decreased pulse pressure, decreased SvO2 1
  • Clinical signs: pulmonary edema, jugular venous distension, cool extremities, altered mental status, oliguria 1

SCAI Classification Stages

  • Stage A (at risk): Normal hemodynamics, normotension, clear lungs, normal perfusion 8, 1
  • Stage B (beginning shock): Clinical evidence of relative hypotension or tachycardia without hypoperfusion 8, 1
  • Stage C (classic shock): Hypoperfusion requiring intervention (inotropes, pressors, or mechanical support) beyond volume resuscitation 8, 1
  • Stage D (deteriorating/doom): Similar to Stage C but worsening despite initial interventions 8, 1
  • Stage E (extremis): Cardiac arrest with ongoing CPR and/or ECMO, requiring multiple interventions 8, 1

Refractory Cardiogenic Shock Criteria

  • Cardiac power output (CPO) <0.6 W is the most critical threshold 1
  • Persistent tissue hypoperfusion despite adequate doses of two vasoactive medications and treatment of underlying etiology 1
  • Cardiac index <2.2 L/min/m² despite vasopressor and inotropic support 1
  • Systolic blood pressure <80 mmHg despite maximal treatment 1

Management

  • For AMI-related cardiogenic shock: immediate coronary angiography within 2 hours with intent to revascularize 1
  • Norepinephrine is first-line vasopressor when MAP needs pharmacologic support, particularly with tachycardia 1, 5
  • Dobutamine is first-line inotrope when signs of low cardiac output persist (up to 20 μg/kg/min) 1, 5
  • Consider dopamine only if bradycardia is present 5
  • Use phenylephrine or vasopressin in afterload-dependent states (aortic stenosis, mitral stenosis) 5
  • Consider temporary mechanical circulatory support when end-organ function cannot be maintained pharmacologically 1
  • Intra-aortic balloon pump (IABP) is NOT routinely recommended except for mechanical complications 1
  • Target cardiac index >2.0 L/min/m² with PCWP <20 mmHg 1

4. Obstructive Shock

Pathophysiology

  • Results from mechanical obstruction to circulation causing hypoperfusion due to elevated resistance 2
  • Common causes include massive pulmonary embolism, tension pneumothorax, cardiac tamponade 3, 6

Hemodynamic Profile

  • Elevated CVP from impaired venous return or right heart obstruction 1
  • Decreased cardiac output from mechanical obstruction 6
  • Variable SVR depending on specific etiology 6

Management

  • Immediate life-saving intervention to relieve obstruction is definitive treatment 2
  • Fluid challenge may optimize preload before definitive intervention 1
  • Specific interventions depend on cause: needle decompression for tension pneumothorax, pericardiocentesis for tamponade, thrombolysis/embolectomy for massive PE 3, 6

Critical Differentiation Points

Distinguishing Cardiogenic from Hypovolemic Shock

Both present with decreased cardiac output, tachycardia, decreased pulse pressure, and decreased SvO2, making differentiation challenging 1

The critical distinguishing feature is ventricular filling pressure:

  • Cardiogenic shock: PCWP >15 mmHg, CVP elevated 1
  • Hypovolemic shock: PCWP decreased, CVP decreased 1

Clinical examination findings:

  • Cardiogenic: pulmonary edema, jugular venous distension, signs of organ hypoperfusion 1
  • Hypovolemic: flat neck veins, dry mucous membranes, history of volume loss 1

When diagnosis remains unclear: Invasive hemodynamic monitoring with pulmonary artery catheter provides definitive measurements 1

Point-of-Care Ultrasound Findings

  • Cardiogenic shock: Decreased LV contractility, dilated ventricles, B-lines indicating pulmonary edema 1
  • Hypovolemic shock: Hyperdynamic LV with small chamber size, collapsed IVC 1
  • Obstructive shock: Specific findings based on etiology (RV strain in PE, pericardial effusion in tamponade) 1

General Principles Across All Shock Types

Monitoring

  • Arterial catheter placement should occur as soon as practical in all patients requiring vasopressors 5
  • Monitor lactate clearance as marker of treatment response 1
  • Pulmonary artery catheterization provides definitive hemodynamic measurements in refractory or unclear cases 1

Timing

  • Early vasopressor use reduces organ failure incidence 5
  • Vasopressors may be initiated during fluid resuscitation and weaned as tolerated 5
  • Adequate fluid resuscitation must precede or accompany vasopressor therapy in hypovolemic, cardiogenic, and obstructive shock 5

Common Pitfall

Do NOT confuse late-stage septic shock with cardiogenic shock—septic shock can develop myocardial depression, but the primary hemodynamic pattern remains distributive with decreased SVR 1

References

Guideline

Hemodynamic Differentiation of Shock Types

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

The Nomenclature, Definition and Distinction of Types of Shock.

Deutsches Arzteblatt international, 2018

Research

Optimizing fluid therapy in shock.

Current opinion in critical care, 2019

Guideline

Vasopressor Management by Shock Type

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Neurogenic Shock Diagnosis and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

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Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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