Why does the BCG (Bacille Calmette-Guérin) vaccine cause scarring in infants and young children from regions with high tuberculosis prevalence?

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Last updated: February 4, 2026View editorial policy

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Why BCG Vaccine Leaves a Scar

BCG vaccination causes scarring because the normal immune response to the live attenuated mycobacterium involves formation of a bluish-red pustule within 2-3 weeks, which ulcerates at approximately 6 weeks (creating a lesion ~5mm in diameter), then forms a scab that heals with permanent scar formation within 3 months. 1

Mechanism of Scar Formation

The scarring process represents the expected local inflammatory response to the percutaneously administered live vaccine:

  • Initial pustule formation occurs within 2-3 weeks as the immune system responds to the mycobacterial antigens at the injection site 1
  • Ulceration phase develops around 6 weeks post-vaccination, creating a draining lesion that should be kept clean and bandaged 1
  • Healing and scar formation typically completes within 3 months, leaving a permanent mark at the puncture site in the deltoid area 1

The scar represents successful local tissue inflammation and healing following the controlled mycobacterial infection at the vaccination site. 2

Variability in Scar Formation

Not all vaccinated individuals develop visible scars, with only 51-83% of vaccinated children showing permanent scarring. 3

Key determinants of whether a scar forms include:

  • BCG strain used - different strains have varying rates of scar formation 1
  • Administration technique - intradermal injection produces more consistent scarring than subcutaneous injection 4
  • Size of injection wheal at time of administration 5
  • Age at vaccination - vaccination within the first 48 hours of life has higher rates of scar failure (approximately 10%) 6
  • Co-administered vaccines and micronutrients may influence local immune response 5

Clinical Significance of Scarring

The presence of a BCG scar does not predict whether the vaccine provided protection against tuberculosis disease. 3

However, research suggests broader health implications:

  • Children with BCG scars have better overall survival compared to vaccinated children without scars (mortality rate ratio 0.61,95% CI: 0.51-0.74), with the strongest effect in the first two years of life 5
  • Scar presence correlates with tuberculin skin test reactivity - 58% of children with scars are TST-positive versus only 15% without scars 7
  • Infants with BCG lesions or scars have fewer symptoms of sepsis and more localized (rather than disseminated) infections, suggesting non-tuberculosis-specific immune benefits 8

Abnormal Scar Formation

Hypertrophic scars occur in 28-33% of vaccinated persons, and keloid scars develop in approximately 2-4%. 1

These represent exaggerated healing responses rather than vaccine failure. Accelerated or more severe reactions may indicate:

  • Previous M. tuberculosis infection causing an enhanced response upon vaccination 1
  • Immunocompromised status, particularly HIV infection, which increases risk of prolonged reactions and complications 1, 4

Key Clinical Pitfalls

  • Absence of a scar does not mean vaccination failure - up to 49% of vaccinated children may lack visible scars yet still mount appropriate cellular immune responses (88-95% positive leukocyte migration inhibition to PPD) 6
  • Reactions persisting beyond 3 months warrant evaluation for complications including BCG lymphadenitis or osteitis, not dismissal as normal healing 4
  • BCG vaccination does not always leave an identifiable scar, making it difficult to confirm vaccination history based on physical examination alone 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

BCG Vaccination and Tuberculosis Infection Risk

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

BCG Vaccination Complications and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

BCG scar and tuberculin reactivity in children and adults.

Scandinavian journal of infectious diseases, 2008

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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