What is Mycosis fungoides, a type of cutaneous T-cell lymphoma?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 5, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Mycosis Fungoides: Definition and Clinical Overview

Mycosis fungoides is the most common type of cutaneous T-cell lymphoma (CTCL), characterized by malignant skin-homing CD4+ T cells that infiltrate the skin, typically presenting with progressive patches, plaques, and potentially tumors, with an indolent clinical behavior in early stages. 1

Classification and Epidemiology

  • Mycosis fungoides represents approximately 60-70% of all primary cutaneous lymphomas and is classified as an indolent cutaneous T-cell lymphoma in the WHO-EORTC classification system 1, 2

  • The annual incidence is 0.7 per 100,000 population in the U.K., with a significantly higher male predominance (1.6:1.0 ratio) and peak age between 50-74 years 1

  • Approximately 20% of patients are diagnosed between ages 25-49 years, and mycosis fungoides may rarely present in childhood 1

Clinical Presentation and Disease Stages

The disease progresses through three distinct clinical stages:

  • Patch stage: Flat, erythematous, scaly lesions that commonly mimic eczema or psoriasis, most frequently appearing on the buttocks, trunk, and breast 2, 3

  • Plaque stage: Raised, infiltrated lesions that develop from patches or appear de novo 4, 2

  • Tumor stage: Nodular lesions that may ulcerate, indicating more advanced disease 4, 5

  • Erythroderma (generalized redness affecting >80% body surface area) can occur and is associated with worse prognosis 4, 5

Immunophenotype and Diagnosis

The classic immunophenotype is CD3+, CD4+, CD45RO+ (memory T-cell marker), and CD8-negative, though rare CD8+ variants exist 6

  • Essential diagnostic markers on paraffin-embedded tissue include: CD2, CD3, CD4, CD8, CD20 (to exclude B-cell lymphoma), and CD30 (to exclude CD30+ lymphoproliferative disorders) 1, 6

  • Histology shows characteristic epidermotropic infiltrates of small-to-medium lymphocytes with Pautrier microabscesses 6, 2

  • Multiple ellipse skin biopsies targeting different lesional areas are required, as diagnosis remains challenging especially in early patch stage 6, 3

  • T-cell receptor (TCR) gene rearrangement analysis is strongly recommended to detect clonality, ideally on fresh tissue 6

  • HTLV-1 serology must be checked in all patients to exclude adult T-cell leukemia/lymphoma, which can mimic CTCL, particularly in endemic areas 1

Prognosis by Stage

Prognosis is strongly determined by extent and type of skin involvement:

  • Stage IA: 96-100% 5-year survival with median survival exceeding 32 years; most patients die from causes other than mycosis fungoides 6, 5

  • Stage IB/IIA: Median survival greater than 11 years, with 24% likelihood of disease progression 5

  • Stage IIB (tumor stage): Median survival of 3 years, with 30-40% developing extracutaneous dissemination 5

  • Stage III (erythroderma): Median survival of 4.5 years 5

  • Stage IVB (visceral involvement): 0-15% 5-year survival with median survival of 13 months 6, 5

  • Increasing age is a poor prognostic factor, while younger patients typically present with early-stage disease and have excellent outcomes 1

Relationship to Sézary Syndrome

Mycosis fungoides and Sézary syndrome are closely related both clinically and pathogenetically but are classified separately, with Sézary syndrome representing an aggressive leukemic variant characterized by erythroderma, peripheral blood involvement with malignant Sézary cells, and lymphadenopathy 1, 7

Critical Diagnostic Pitfalls

  • The nonspecific skin presentation in early stages frequently mimics common inflammatory dermatoses such as eczema and psoriasis, making diagnosis challenging and often requiring multiple biopsies over time 2, 3

  • Folliculotropic variant has worse prognosis (36% disease-specific survival at 5 years for stage IB) and requires recognition on histology 6

  • Large cell transformation (often CD30+) can develop in plaque-type or erythrodermic mycosis fungoides and is associated with less favorable outcome 5

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Mycosis Fungoides: a review.

Clinical and experimental dermatology, 2025

Research

Mycosis fungoides: an updated review of clinicopathologic variants.

The American Journal of dermatopathology, 2014

Research

Diagnosis and management of mycosis fungoides.

Oncology (Williston Park, N.Y.), 2010

Research

Mycosis fungoides.

Critical reviews in oncology/hematology, 2008

Guideline

Staging and Management of Mycosis Fungoides

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Cutaneous T cell lymphoma-mycosis fungoides and Sezary syndrome: an update.

Giornale italiano di dermatologia e venereologia : organo ufficiale, Societa italiana di dermatologia e sifilografia, 2009

Related Questions

What are the treatment options for mycosis fungoides?
What are the next steps for managing a patient with confirmed mycosis fungoides (MF) regarding clinical examination, history, investigations, and referrals to other specialists?
What is Mycosis Fungoides Tumor Stage?
What are the next steps and referrals for a patient with confirmed mycosis fungoides (cutaneous T-cell lymphoma) with nodal affection and potential systemic involvement, after requesting a Positron Emission Tomography (PET) or Computed Tomography (CT) scan with contrast and referring to oncology/hematology and general surgery?
Can a computed tomography (CT) scan of the pelvis, chest, and abdomen help detect lymphadenopathy in a patient with early patch stage Mycosis fungoides?
How does stage 3 Chronic Kidney Disease (CKD) affect Hormone Replacement Therapy (HRT) options?
What is the recommended treatment for a patient with a confirmed diagnosis of vitamin D deficiency, considering factors such as severity of deficiency, calcium intake, and potential underlying medical conditions like kidney or liver disease?
What is the recommended dose of epinephrine (adrenaline) nebulisation and intravenous (IV) dexamethasone for a neonate with stridor?
Is N-acetylcysteine (NAC) 600mg three times a day (TDS) justified in a patient with acute bronchitis, acute bronchospasm, and acute suppurative otitis media (ASOM)?
What is the first line treatment for a patient with a confirmed diagnosis of Clostridioides difficile (C. diff) infection?
Does long-term use of esomeprazole (proton pump inhibitor) affect bone health in older adults or individuals with a history of osteoporosis?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.