What is the recommended dosing regimen of TYVALZI (sovateltide, IRL‑1620) for adult patients with acute ischemic stroke?

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Last updated: February 8, 2026View editorial policy

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Sovateltide Dosing for Acute Ischemic Stroke

The recommended dosing regimen of sovateltide (TYVALZI, IRL-1620) for adult patients with acute ischemic stroke is 0.3 µg/kg administered intravenously as a bolus over 1 minute, given in three doses at 3 ± 1 hour intervals on Days 1,3, and 6 (total daily dose of 0.9 µg/kg on each treatment day), initiated within 24 hours of stroke onset. 1, 2

Dosing Protocol

Administration Schedule:

  • Dose: 0.3 µg/kg per administration 1, 2
  • Route: Intravenous bolus over 1 minute 2
  • Frequency: Three doses per treatment day, separated by 3 ± 1 hour intervals 1, 2
  • Treatment Days: Day 1, Day 3, and Day 6 after randomization 1, 2
  • Total Daily Dose: 0.9 µg/kg on each of the three treatment days 2

Timing of Initiation

  • Treatment window: Must be initiated within 24 hours of stroke symptom onset 1, 2
  • Median time to first dose: Approximately 18 hours after stroke onset in clinical trials 1
  • Optimal timing: Earlier administration within the 24-hour window may be preferable, with 14-20 hours being typical in trial populations 2

Patient Selection Criteria

Inclusion parameters based on regulatory approval studies:

  • Age 18-78 years 1
  • Radiologically confirmed acute cerebral ischemic stroke 1, 2
  • National Institutes of Health Stroke Scale (NIHSS) score ≥ 6 at presentation 1
  • Presenting within 24 hours of symptom onset 1, 2

Exclusion criteria:

  • Recurrent stroke 1
  • Patients receiving endovascular therapy 1, 2
  • Intracranial hemorrhage 1, 2

Integration with Standard of Care

Sovateltide is administered as an adjunct to standard stroke care, not as a replacement for established therapies. 3, 2 This means:

  • Intravenous thrombolysis with alteplase should still be administered when indicated and not contraindicated 3
  • Mechanical thrombectomy for large vessel occlusions remains the priority intervention when appropriate 3
  • Standard supportive care including blood pressure management, glucose control, and antiplatelet therapy (aspirin 160-325 mg started 24-48 hours post-stroke if not receiving thrombolysis) should continue 4

Safety Profile

  • Hemodynamic stability: Sovateltide does not affect blood pressure, heart rate, or other hemodynamic parameters at therapeutic doses 2
  • Hemorrhage risk: Intracranial hemorrhage rates were similar between sovateltide (8.75%) and control groups (8.97%) in Phase III trials 1
  • Tolerability: No drug-related adverse events were reported in clinical trials 2
  • Laboratory parameters: No effects on biochemical or hematological parameters 2

Clinical Context and Evidence Quality

The dosing regimen is based on a Phase III randomized, double-blind, placebo-controlled trial that led to regulatory approval in India 1. This represents the highest quality evidence available for sovateltide dosing. The trial demonstrated:

  • 22.67% absolute increase in patients achieving modified Rankin Scale (mRS) 0-2 at 90 days (OR 2.75) 1
  • 17.05% absolute increase in patients achieving NIHSS 0-5 at 90 days (OR 2.67) 1
  • 51.28% vs 72.50% of patients showing ≥2 point improvement on mRS (OR 2.50) 1

Important Caveats

Current guideline limitations: The American Heart Association/American Stroke Association guidelines do not specifically mention sovateltide, as they state that no neuroprotective agents have demonstrated efficacy in improving stroke outcomes (Class III: No Benefit; Level of Evidence A for neuroprotective agents in general) 3. However, this reflects the guideline publication date predating sovateltide's Phase III trial completion.

Geographic availability: Sovateltide has received regulatory approval in India but is not yet approved in the United States 1. A multinational RESPECT-ETB trial is planned for US FDA approval 1.

Mechanism distinction: Unlike traditional neuroprotective agents that have failed in stroke trials, sovateltide works through endothelin-B receptor agonism to increase cerebral blood flow, provide anti-apoptotic effects, and promote neurovascular remodeling 1, 2, 5, 6. This mechanistic difference may explain its clinical efficacy where other neuroprotective strategies have failed.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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