Does spironolactone increase low-density lipoprotein (LDL) cholesterol?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 11, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Does Spironolactone Increase LDL Cholesterol?

No, spironolactone does not increase LDL cholesterol; in fact, it either has no effect or may modestly reduce LDL-C levels based on available evidence.

Evidence from Clinical Studies

The most direct evidence comes from a 1983 study examining spironolactone 100 mg daily over 12 months in hypertensive patients, which found that total cholesterol and LDL cholesterol remained unchanged throughout the treatment period 1. This demonstrates that spironolactone does not adversely affect LDL-C levels even with prolonged use at standard doses.

More recent evidence from 2011 in non-obese women with polycystic ovary syndrome showed that spironolactone 100 mg daily for 6 months actually decreased both total cholesterol (from 4.8 to 4.4 mmol/L) and LDL cholesterol (from 2.5 to 2.2 mmol/L), both statistically significant reductions 2. This suggests spironolactone may have beneficial effects on lipid profiles in certain populations.

Comparative Context with Other Diuretics

A 1988 review clarified that spironolactone has a lesser effect on lipids than thiazide diuretics, which are known to increase total cholesterol by 6-7% on average through raised LDL or VLDL cholesterol 3. This positions spironolactone as a more lipid-neutral diuretic option.

Systematic Review Findings

A 2021 systematic review and meta-analysis of 11 randomized placebo-controlled trials (640 participants) in patients with both hypertension and diabetes found no significant difference in lipid metabolism parameters between spironolactone and placebo groups 4. This high-quality evidence confirms the lipid-neutral profile of spironolactone across diverse patient populations.

Clinical Implications

  • Spironolactone can be safely used without concern for LDL elevation in patients requiring aldosterone antagonism for heart failure, resistant hypertension, or cirrhotic ascites 5, 6
  • The drug's cardiovascular benefits (30% mortality reduction in severe heart failure) far outweigh any theoretical lipid concerns 5, 6
  • HDL cholesterol may decrease modestly (from 1.5 to 1.0 mmol/L after 12 months), which is the primary lipid change observed with spironolactone 1

Important Caveats

The primary metabolic concern with spironolactone is a mild, transient increase in glycosylated hemoglobin (HbA1c) by approximately 0.3% in diabetic patients, not lipid abnormalities 4. Monitor glucose control rather than lipid panels when initiating spironolactone in patients with diabetes.

Related Questions

What are the side effects of Aldactone (spironolactone)?
What are the effects of combining spironolactone, magnesium glycinate, and alpha-lipoic acid?
Can spironolactone (50 milligrams) be taken concurrently with a progesterone capsule?
Does spironolactone carry less risk of causing gout flare-up compared to triamterene/hydrochlorothiazide (Maxzide)?
What are the next steps for managing persistent bilateral lower leg edema in a 69-year-old female with impaired renal function (GFR 49), diabetes mellitus (DM) with elevated hemoglobin A1c (HbA1c) of 11.9, and currently taking Lantus (insulin glargine) and Lasix (furosemide) 20 mg daily?
What is the recommended octreotide dosing regimen for an adult with hepatorenal syndrome?
What oral magnesium supplementation regimen should be used to correct mild-to-moderate hypomagnesemia in an adult patient without severe renal insufficiency or gastrointestinal malabsorption?
In a 57-year-old woman with dysfunctional uterine bleeding, weight 55 kg, who has received 500 mg oral tranexamic acid and now has stable blood pressure, should IV tranexamic acid be administered and what dose is appropriate?
How is hyperosmolar hyperglycemic coma diagnosed in an older adult with type 2 diabetes presenting with sudden altered mental status?
What are the causes, diagnostic investigations, and treatment options for piriformis syndrome?
Can you create a SOAP note with three differential diagnoses for a patient who presents with new symmetric distal peripheral neuropathy, reports about 10 standard alcoholic drinks per week, has no personal history of diabetes, autoimmune disease, or prior neuropathy, and has first-degree relatives with diabetes mellitus, chronic alcohol abuse, and rheumatoid arthritis?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.