For a 61-year-old diabetic, hypertensive, coronary artery disease patient with dilated cardiomyopathy (ejection fraction ~31%) and recent defaulted extrapulmonary tuberculosis who became hypotensive on day 2 requiring norepinephrine and dopamine, is the shock primarily cardiogenic or septic, what caused the sudden deterioration, should early dobutamine/milrinone or intra‑aortic balloon pump have been used, and are the urinary red‑blood‑cell casts due to cardiorenal syndrome or a tuberculosis‑related glomerulonephritis?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 12, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

{
  "question": "Good morning sir\n**1.Case report**\nA [AGE]-year-old male who is a k/c/o T2DM/SHTN/CAD was brought with complaints of breathlessness, Bilateral swelling of legs, abdominal distension for about [DURATION]. On admission he was conscious, Oriented tachypneic with Bilateral creptitations all over lung fields. He had a history of EPTB for which he took ATT for [DURATION] and stopped on his own.\nHe was Provisionally diagnosed as \nVOLUME OVERLOAD STATE/CAD/DCM /HFREF[EF%]/EPTB DEFAULTER/T2DM /SHTN \n\n[DAY]-He developed hypotension and was started on norepinephrine and was escalated to dual ionotropes with dopamine \n\nHe was on ionotropes till his death on [DAY].\n\nEcg showed LBBB with low voltage complexes\n\nInvestigations revealed elevated total counts with elevated rft, elevated total bilirubin, elevated esr, positive crp. sputum reports were negative, urine hb was positive, urine rbc cast was positive\n\nHe was treated with higher antibiotics and supportive care\n\nEcho -DCM /EF [EF%]\n\n*Queries*\n1.Adherence to CAD drugs and diet -whether he adhered or not \n2.History of EPTB where and how was he diagnosed and why he defaulted\n3.Any evaluation done regarding etiology of DCM \n\n *QUESTIONS*\n1.cause of shock purely Cardiogenic or associated sepsis\n2.cause of sudden deterioration in [DAY] \n3.any association between EPTB and shock in this case\n4.Initial bp was [BP] mmhg -Any role of dobutamine or milrinone in this phase \n5.Use of IABP in this case -if possible then why and what is the timeline and prerequisites in this case\n6.Positive Rbc casts -was it cardiac or non cardiac cause \n7.was it a cardiorenal syndrome or any possibility of glomerulonephritis \n8.If possible of glomerulonephritis, then can EPTB history be attributed to this\nThank you sir\ncrisp discussion\n*Do not change the language of the question*"
}

Related Questions

In a 61-year-old male with type 2 diabetes, hypertension, coronary artery disease, dilated cardiomyopathy (EF 31%), volume overload, and recent incomplete treatment for extrapulmonary tuberculosis who developed hypotension on day 2 requiring norepinephrine and dopamine, what is the primary cause of shock (cardiogenic vs sepsis), what precipitated the sudden deterioration, is there any association between the tuberculosis and shock, should dobutamine or milrinone have been used, is intra‑aortic balloon pump indicated, are the positive RBC casts of cardiac or non‑cardiac origin, and could this represent cardiorenal syndrome or glomerulonephritis possibly related to tuberculosis?
What is the management of Acute Kidney Injury (AKI) secondary to cardiogenic shock?
What is the role of dobutamine in pediatric cardiogenic shock?
In adults with acute cardiogenic shock (often post‑myocardial infarction), what were the efficacy and safety outcomes of milrinone versus dobutamine in the DOREMI trial, and how should the inotrope be selected?
What is the role of milrinone and levophed (levonorepinephrine) in treating cardiogenic shock?
In an adult patient with ST‑segment elevation myocardial infarction who has achieved successful fibrinolysis with alteplase or tenecteplase and has no contraindications to antiplatelet or anticoagulant therapy, why is a routine early percutaneous coronary intervention strategy superior to a watch‑and‑wait approach?
What is the recommended oral dose of co‑amoxiclav (amoxicillin‑clavulanate) for an uncomplicated urinary‑tract infection in a child, including weight‑based dosing and adjustments for infants under 3 months and renal impairment?
Can the Titmus fly test assess both fine and coarse stereopsis?
Please provide a detailed admission order for a first‑trimester pregnant woman presenting with 12 days of watery diarrhea, low‑back pain, and electrolyte disturbances (hypokalemia, hypomagnesemia, borderline low ionized calcium).
Which erectile dysfunction treatments have minimal or no hepatic metabolism and are safe for patients with liver disease?
What empiric antibiotic regimen should be started for an 88‑year‑old woman with cloudy amber urine, small hematuria, large leukocyte esterase, 4–10 red blood cells, numerous white blood cells, moderate bacteria, and few white‑cell clumps while awaiting culture results?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.