Does oral medroxyprogesterone acetate (Provera) affect bone mineral density?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 14, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Does Oral Provera (Medroxyprogesterone Acetate) Affect Bone Density?

Oral medroxyprogesterone acetate (Provera) is distinct from injectable depot medroxyprogesterone acetate (DMPA/Depo-Provera), and the bone density concerns documented in guidelines and research specifically apply to the injectable formulation, not the oral pill form.

Critical Distinction Between Formulations

The evidence provided exclusively addresses injectable DMPA (Depo-Provera), not oral medroxyprogesterone acetate tablets. This is a crucial clinical distinction:

  • Injectable DMPA causes significant bone mineral density loss during use, particularly in the first two years, though this loss is largely reversible after discontinuation 1, 2, 3
  • Oral medroxyprogesterone acetate (Provera pills) is used at much lower doses for different indications (menstrual regulation, hormone replacement therapy component, endometrial protection) and does not carry the same bone density warnings 4

Why Injectable DMPA Affects Bone Differently

The injectable formulation creates sustained supraphysiologic progestin levels that suppress ovarian function more profoundly than oral formulations:

  • DMPA causes prolonged hypoestrogenism by disrupting the hypothalamic-pituitary-gonadal axis, leading to decreased estradiol levels 4
  • Estradiol is osteoprotective and inhibits osteoclast activity; when levels are subphysiologic, bone resorption predominates over formation 4
  • This mechanism is specific to the high-dose, long-acting injectable preparation

Evidence Specific to Injectable DMPA (Not Oral Provera)

Bone Density Changes with Injectable DMPA:

  • Long-term DMPA users (≥10 years) showed 68.1% prevalence of low bone mass and 29.8% prevalence of osteoporosis compared to 36.6% and 2.4% respectively in IUD users 5
  • Adolescent DMPA users experienced annualized bone loss of -1.81% at the hip and -0.97% at the spine compared to gains in nonusers 6
  • BMD loss is most pronounced in the first 2 years of DMPA use, then stabilizes to approximately 1% per year 7

Reversibility After Discontinuation:

  • Substantial BMD recovery occurs after DMPA discontinuation, with bone density returning toward baseline values 1, 2
  • Adolescent discontinuers experienced significant BMD gains: hip +1.34%, spine +2.86%, whole body +3.56% annually post-discontinuation 6

Current Guidelines for Injectable DMPA (Not Applicable to Oral Provera)

The American College of Obstetricians and Gynecologists provides specific guidance for injectable DMPA only:

  • Does not recommend limiting DMPA use to 2 years despite the FDA black-box warning 1, 2, 3
  • Does not recommend routine bone density monitoring in DMPA users 2, 3
  • Emphasizes that the risk of unintended pregnancy from restricting contraceptive options outweighs potential bone concerns 1

Bone Health Counseling for Injectable DMPA Users:

  • Daily calcium intake of 1,300 mg 1, 2, 3
  • Daily vitamin D intake of 600 IU 1, 2, 3
  • Regular weight-bearing exercise 1, 2, 3
  • Smoking cessation and alcohol avoidance 1, 3

Clinical Bottom Line

Oral medroxyprogesterone acetate (Provera pills) does not carry the bone density concerns associated with injectable DMPA. The extensive literature on bone loss relates specifically to the depot injectable formulation, which creates a unique pharmacologic profile not replicated by oral administration. When prescribing oral Provera for menstrual disorders, endometrial protection in hormone therapy, or other indications, bone density loss is not a primary concern requiring specific monitoring or supplementation beyond standard age-appropriate recommendations 4.

Common Pitfall to Avoid:

  • Do not conflate injectable DMPA (Depo-Provera) with oral medroxyprogesterone acetate (Provera pills)—they have entirely different risk profiles regarding bone health 4, 1

References

Guideline

Bone Health Implications of Depo‑Provera (DMPA) Use

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Bone Mineral Density Loss with Depo Provera

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Depo-Provera Use and Bone Health Considerations

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Related Questions

What is the risk of osteoporosis in a woman using depot medroxyprogesterone acetate (Depo‑Provera), particularly with continuous use longer than two years?
Is the Depo (Depot Medroxyprogesterone Acetate, DMPA) shot a suitable contraceptive option for teenagers?
What are the risks of using weight loss shots in a patient with a normal Body Mass Index (BMI) of 23, an absent menstrual cycle due to an Intrauterine Device (IUD), and potential risk of osteoporosis?
What is the maximum duration for treatment with Depo-Provera (medroxyprogesterone acetate) injections?
When should Depo-Provera (medroxyprogesterone acetate) be discontinued in a woman with normal Bone Mineral Density (BMD) after more than 10 years of use?
When should cephalexin 500 mg three times daily be stopped for a patient who began it on postoperative day [DATE] after an uncomplicated surgery with normal renal function?
How should clindamycin be dosed, adjusted for hepatic or renal impairment, and monitored in an adult with uncomplicated cellulitis who requires it due to a β‑lactam allergy or suspected MRSA, and what is the appropriate treatment duration?
In a 6‑month‑old infant with intermittent inward deviation of the right eye during feeding and a normal bilateral red reflex, what is the most appropriate initial management?
In a 49‑year‑old woman with type 2 diabetes mellitus, hemoglobin A1c 10.9 %, weight 123 kg (BMI ≈ 41 kg/m²), currently on basal insulin glargine (Lantus) 50 U daily, weekly semaglutide (Ozempic) 0.5 mg, and metformin 1000 mg daily, what should her basal insulin dose, insulin‑to‑carbohydrate ratio, and correction factor be?
In a 34‑year‑old woman with recurrent sudden “push” sensations causing falls and vertigo, and a normal brain MRI, which of the following is most commonly associated: diplopia, headache, hearing loss, recent upper‑respiratory infection, or Horner’s syndrome?
Can a man over 50 with benign prostatic hyperplasia use beta‑sitosterol as primary therapy, and how effective is it compared to alpha‑adrenergic blockers or 5‑alpha‑reductase inhibitors?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.