In an adult on chronic dialysis with hemoglobin 11 g/dL, low serum iron, low total iron‑binding capacity, and high ferritin, what is the appropriate management of anemia?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 15, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Management of Anemia in Dialysis Patient with Functional Iron Deficiency

This patient has functional iron deficiency (low TSAT with high ferritin) and should receive intravenous iron supplementation despite the elevated ferritin of 890 ng/mL, as the low TSAT of 23.5% indicates insufficient iron availability for erythropoiesis. 1, 2

Understanding the Iron Parameters

Your patient presents with a classic pattern of functional iron deficiency:

  • Hemoglobin 110 g/L (at target per guidelines) 1
  • TSAT 23.5% (calculated from iron 9.7 and TIBC 41.2) - below optimal target
  • Ferritin 890 ng/mL - elevated but does not contraindicate iron therapy when TSAT is low

The low TSAT (<25%) with elevated ferritin indicates that stored iron is not being mobilized effectively for red blood cell production, a condition termed functional iron deficiency. 1, 3

Treatment Recommendation

Intravenous Iron Administration

Administer IV iron supplementation to increase TSAT above 25% and optimize erythropoiesis, even with ferritin >800 ng/mL. 1, 2

  • The 2008 Canadian Society of Nephrology guidelines specifically address this scenario, recommending consideration of IV iron when ferritin is 800-1200 ng/mL if TSAT <25% and hemoglobin is suboptimal or ESA doses are high 1
  • Give 100-125 mg IV iron at each hemodialysis session for 8-10 consecutive doses to address the functional deficiency 2, 4
  • The American Journal of Kidney Diseases supports higher TSAT targets of 30-50% to optimize ESA response and reduce ESA requirements by up to 40% 2

Safety Monitoring

Withhold IV iron only if TSAT exceeds 50% or ferritin exceeds 800 ng/mL AND TSAT is adequate. 2, 4

  • In this case, while ferritin is 890 ng/mL (slightly above 800), the low TSAT of 23.5% justifies continued iron therapy 1
  • Monitor TSAT and ferritin every 3 months during maintenance therapy 2, 4
  • Ferritin levels of 300-800 ng/mL are common in dialysis patients without evidence of adverse iron-mediated effects 2

ESA Management Considerations

Evaluate whether the patient is receiving erythropoiesis-stimulating agents (ESAs), as this influences iron requirements. 1, 5, 6

  • If on ESAs: The functional iron deficiency is likely ESA-induced, as EPO stimulates erythropoiesis beyond the rate at which stored iron can be mobilized 1
  • Administer supplemental iron when serum ferritin is less than 100 mcg/L or when serum transferrin saturation is less than 20% per FDA labeling for both epoetin alfa and darbepoetin alfa 5, 6
  • If not on ESAs and hemoglobin remains at 110 g/L: Consider initiating ESA therapy only if hemoglobin drops below 100 g/L, as current hemoglobin is at guideline target 1, 5, 6

Maintenance Strategy After Initial Course

Once TSAT reaches >25%, transition to maintenance dosing of 25-125 mg IV iron weekly to prevent recurrent functional iron deficiency. 2, 4

  • Hemodialysis patients require ongoing IV iron due to substantial blood losses from dialysis procedures, residual blood in tubing, frequent blood sampling, and poor oral absorption 2
  • Need-based, continuous, low-dose iron (10-60 mg given 1-3 times weekly based on monthly TSAT and ferritin monitoring) achieves better hemoglobin response than intermittent pulse dosing 7

Common Pitfalls to Avoid

Do not withhold iron solely based on elevated ferritin when TSAT is low. 1

  • The combination of high ferritin with low TSAT specifically indicates functional iron deficiency requiring treatment 1, 3
  • Ferritin is an acute phase reactant and may be elevated due to inflammation rather than true iron overload 3, 8
  • Serum ferritin remains the most reliable diagnostic parameter for iron stores in stable dialysis patients, but must be interpreted alongside TSAT 8

Oral iron is inadequate for hemodialysis patients and should not be used as primary therapy. 1, 2

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Iron Supplementation Guidelines for ESRD Patients

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Iron Replacement Therapy for Chronic Kidney Disease Patients

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Diagnosis of iron deficiency in chronic renal failure.

American journal of kidney diseases : the official journal of the National Kidney Foundation, 1999

Related Questions

Should a patient with normal iron and ferritin levels, low iron saturation, high Total Iron Binding Capacity (TIBC), and normal hemoglobin levels receive iron supplements?
What is the best management approach for a patient with diabetes and Chronic Kidney Disease (CKD), presenting with iron deficiency anemia?
What interventions are recommended for a 70-year-old female with iron deficiency anemia (IDA), mild chronic kidney disease (CKD), low ferritin, and impaired renal function, as indicated by an estimated glomerular filtration rate (eGFR) of 57?
Should iron supplementation be adjusted for a dialysis patient with a ferritin level of 1057, iron saturation of 20.19%, iron level of 39, and TIBC of 194?
In a patient with normal hemoglobin and hematocrit, low serum iron, low transferrin saturation, and a ferritin level of 62 µg/L (within normal range), should iron supplementation be started?
Which bedside sepsis risk score (quick Sequential Organ Failure Assessment (qSOFA) or Sequential Organ Failure Assessment (SOFA)) should I use to decide if a patient with suspected infection needs urgent escalation?
Why does Cushing's disease (chronic excess endogenous cortisol) cause hypocalcemia?
What is the recommended starting dose of insulin glargine (basal insulin) for an adult with type 1 diabetes?
Why does Cushing's disease cause atrial fibrillation that is refractory to cardioversion?
Can the adult National Early Warning Score 2 be used in pediatric patients?
What laboratory abnormalities are typical in Cushing’s disease caused by an ACTH (adrenocorticotropic hormone)‑secreting pituitary adenoma?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.