What is photosensitivity?

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What is Photosensitivity?

Photosensitivity is an abnormal cutaneous reaction to ultraviolet radiation (UVR) or visible light that occurs at lower doses than would normally cause damage in non-photosensitive individuals. 1, 2

Clinical Definition and Mechanisms

Photosensitivity encompasses two distinct pathophysiologic mechanisms:

  • Phototoxicity: A non-immunologic reaction where UV radiation activates a photosensitizing substance in the skin, causing direct cellular damage through free radical formation and DNA damage. This is dose-dependent and can occur on first exposure. 3

  • Photoallergy: An immunologic reaction requiring prior sensitization, where UV radiation converts a chemical into an antigen that triggers a delayed-type hypersensitivity response. This is much less common than phototoxicity. 3

Clinical Manifestations

The presentation varies widely depending on the underlying cause:

  • Acute phototoxic reactions: Burning, tingling sensation, and erythema resembling exaggerated sunburn in sun-exposed areas, typically appearing within hours of exposure 4, 5

  • Photoallergic reactions: Eczema-like rash, pruritus, and vesiculation that may extend beyond sun-exposed areas, appearing 24-72 hours after exposure 1, 5

  • Other patterns: Urticaria (solar urticaria), pigmentation changes (blue-grey discoloration with amiodarone), onycholysis, and blistering 4, 1, 5

Categories of Photosensitive Disorders

Primary (Idiopathic) Photodermatoses

These conditions are entirely caused by solar exposure: 1, 2

  • Polymorphic light eruption (PLE): Recurrent pruritic papulo-vesicular eruption appearing within hours of sun exposure, most common primary photodermatosis 1
  • Chronic actinic dermatitis (CAD): Persistent eczematous reaction in chronically sun-exposed skin 6, 1
  • Solar urticaria: Rapid onset of urticaria after sun exposure 6, 1
  • Actinic prurigo: Excoriated papules and nodules on face and limbs, most prominent distally 1
  • Hydroa vacciniforme: Discrete erythematous macules evolving into blisters within days of exposure 1

Drug- and Chemical-Induced Photosensitivity

Several hundred substances can invoke photosensitive reactions: 3, 5

  • Common culprits: Tetracyclines, fluoroquinolones, NSAIDs, thiazide diuretics, cardiovascular medications (especially amiodarone), and antidepressants 7, 4
  • Amiodarone-specific: Causes photosensitivity in over 50% of treated patients, with potential for blue-grey pigmentation in 1-2% with extended sun exposure 4

Photo-Aggravated Dermatoses

Pre-existing skin conditions worsened by UV exposure: 2, 8

  • Lupus erythematosus, psoriasis, atopic dermatitis, rosacea, pemphigus, and erythema multiforme 2, 8

Genodermatoses with Photosensitivity

Inherited disorders with deficient photoprotection: 2, 8

  • Xeroderma pigmentosum, albinism, vitiligo, and DNA repair deficient disorders 2, 8

Metabolic Photodermatoses

Accumulation of endogenous chromophores: 8

  • Porphyrias (congenital enzymatic disorders) and pellagra (acquired) 8

Diagnostic Approach

A detailed history focusing on timing and pattern of reactions, specific triggers, distribution of affected areas, associated symptoms, and medication review is essential. 6

Key diagnostic elements include:

  • Phototesting: Minimal erythema dose (MED) determination for UVB and minimal phototoxic dose (MPD) for UVA, performed by dermatology specialists 9, 6
  • Laboratory evaluation: Complete blood count, comprehensive metabolic panel, antinuclear antibodies if lupus suspected, porphyrin levels if porphyria suspected 6
  • Physical examination: Document distribution and morphology of lesions, signs of chronic photodamage, and ocular involvement 6

Clinical Context in Phototherapy

Understanding photosensitivity is critical when administering UV-based treatments:

  • Skin type assessment: Subjective Fitzpatrick classification is not always a reliable predictor of objective erythema sensitivity obtained from MED testing 9
  • Individual variability: MED varies by a factor of four in white individuals, while MPD for PUVA varies by at least 10-fold between individuals 9
  • Delayed reactions: PUVA erythema peaks at 48-96 hours (or later), requiring treatment intervals of at least 2-3 days 9, 7

Common pitfall: Do not rely solely on skin phototyping to determine starting doses for phototherapy—objective MED or MPD testing is more reliable for preventing severe burns. 9

References

Research

Photosensitivity disorders: cause, effect and management.

American journal of clinical dermatology, 2002

Research

Diseases associated with photosensitivity.

Journal of photochemistry and photobiology. B, Biology, 2001

Research

Scientific problems of photosensitivity.

Medicina (Kaunas, Lithuania), 2006

Guideline

Amiodarone and Skin Cancer Risk

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Drug-induced photosensitivity.

Prescrire international, 2009

Guideline

Workup for Increased Sun Sensitivity

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Treatment of Photosensitivity

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

[Skin diseases with photosensitivity].

La Revue du praticien, 1992

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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