What are the lab findings for primary hyperaldosteronism (Conn's syndrome)?

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From the Guidelines

Primary Hyperaldosteronism (Conn's Syndrome) Lab Findings

Primary hyperaldosteronism, also known as Conn's syndrome, is characterized by elevated plasma aldosterone levels and suppressed plasma renin activity. The typical lab findings include:

  • Elevated plasma aldosterone levels, typically exceeding 15 ng/dL 1
  • Suppressed plasma renin activity, often below 1.0 ng/mL/h 1
  • Hypokalemia, with serum potassium levels less than 3.5 mmol/L, although this is not always present 1
  • Metabolic alkalosis, with elevated serum bicarbonate levels above 28 mmol/L 1

Diagnostic Confirmation

The diagnosis of primary hyperaldosteronism can be confirmed by:

  • Administering a saline infusion test, where 2 liters of 0.9% saline solution are infused over 4 hours, and plasma aldosterone levels remain elevated above 10 ng/dL at the end of the test 1
  • Oral sodium loading test (with 24-h urine aldosterone) or IV saline infusion test with plasma aldosterone at 4 h of infusion 1
  • Adrenal vein sampling to determine whether the increased aldosterone production is unilateral or bilateral in origin 1

Key Points

  • The aldosterone-to-renin ratio is a useful screening test for primary hyperaldosteronism, with a ratio greater than 30 suggesting the diagnosis 1
  • Patients with primary hyperaldosteronism may exhibit resistant hypertension, hypokalemia, and metabolic alkalosis 1
  • The condition can be caused by unilateral adrenal adenoma or bilateral adrenal hyperplasia 1

From the Research

Lab Findings for Primary Hyperaldosteronism (Conn's Syndrome)

The lab findings for primary hyperaldosteronism (Conn's syndrome) include:

  • High plasma and urinary aldosterone levels 2
  • Suppressed plasma renin activity (PRA) 2, 3
  • Elevated aldosterone/renin ratio 2, 3, 4
  • Normal to high normal PRA levels in some cases, despite high aldosterone levels 2
  • High serum creatinine levels and hyperkalemia after spironolactone treatment 2
  • Failure to suppress plasma aldosterone concentration (PAC) after saline infusion 5, 4

Diagnostic Criteria

The diagnostic criteria for primary hyperaldosteronism include:

  • Elevated aldosterone/renin ratio 3, 4
  • Failure to suppress PAC after saline infusion 5, 4
  • PAC above 10 ng/dL (>277 pmol/L) after saline infusion 6
  • PAC above 14 ng/dL (397 pmol/L) at 120 minutes after saline infusion 5

Prevalence and Presentation

Primary hyperaldosteronism can occur in approximately 5-15% of hypertensive patients, with many cases presenting with normal serum potassium concentrations 3, 6

  • The prevalence of primary hyperaldosteronism may vary among different populations, with a study in Singapore finding a prevalence of at least 5% in the adult Asian hypertensive population 6
  • Hypokalemia is not a reliable predictor of primary hyperaldosteronism, as many patients may have normal serum potassium levels 2, 3, 6

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Primary hyperaldosteronism without suppressed renin due to secondary hypertensive kidney damage.

The Journal of clinical endocrinology and metabolism, 2000

Research

Normokalemic hyperaldosteronism in patients with resistant hypertension.

The Israel Medical Association journal : IMAJ, 2002

Research

Diagnosis of primary hyperaldosteronism.

Medicina clinica, 2022

Research

Prevalence of primary aldosteronism among Asian hypertensive patients in Singapore.

The Journal of clinical endocrinology and metabolism, 2000

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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