What are the causes of acute pancreatitis in pregnancy?

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Last updated: February 19, 2026View editorial policy

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Causes of Pancreatitis in Pregnancy

Gallstone disease is the leading cause of acute pancreatitis in pregnancy, accounting for 56-100% of cases, followed by hypertriglyceridemia (which is amplified by pregnancy's physiologic hyperlipidemia), and alcohol abuse. 1, 2, 3

Primary Etiologies

Gallstone-Related Pancreatitis (Most Common)

  • Gallstones cause 56-100% of acute pancreatitis cases during pregnancy, making biliary disease the predominant etiology 3, 4, 5
  • The estimated incidence of gallstone-related disease complicating pregnancy is 0.5-0.8% 2
  • Pregnancy creates a lithogenic environment through gallbladder stasis and increased cholesterol secretion due to elevated estrogen levels 6, 7
  • Risk factors include preexisting gallstones, obesity (high prepregnancy BMI), and elevated serum leptin levels 2
  • Acute pancreatitis most commonly occurs during the third trimester or early postpartum period when biliary stasis is maximal 3, 5

Hypertriglyceridemia-Induced Pancreatitis (Second Most Common)

  • Pregnancy induces a "physiological hyperlipidemia" with 2-fold increases in triglyceride levels during the third trimester due to enhanced lipolytic activity 2
  • Women with baseline triglyceride levels ≥500 mg/dL may develop severe hypertriglyceridemia during the third trimester, creating substantial pancreatitis risk 2
  • Both insulin resistance and hyperestrogenemia amplify hypertriglyceridemia during pregnancy 2
  • Maternal mortality from gestational hypertriglyceridemic pancreatitis approaches 20%, making early recognition critical 2
  • Serum triglyceride levels >11.3 mmol/L (>1000 mg/dL) indicate hypertriglyceridemia as the causative etiology 8

Alcohol-Related Pancreatitis (Less Common in Pregnancy)

  • Alcohol abuse accounts for 16-20% of cases in some series, though it is unusual compared to nonpregnant women 5, 7
  • This represents a significantly lower proportion than in the general population where alcohol causes 20-25% of cases 8

Other Causes

  • Post-ERCP pancreatitis: Pregnancy is an independent risk factor with rates of 12% versus 5% in nonpregnant women 1
  • Hereditary pancreatitis (4% of cases) 5
  • Hypercalcemia: Should be investigated in all cases, especially when gallstones and alcohol are excluded 8
  • Drug-induced causes: Statins are contraindicated in pregnancy, but other medications may contribute 1
  • Idiopathic: Should represent no more than 20-25% of cases after thorough investigation 8

Diagnostic Approach

Initial Laboratory Evaluation

  • Measure serum amylase or lipase together with liver function tests 8
  • Early elevation of aminotransferases or bilirubin strongly suggests biliary (gallstone) etiology 8
  • Obtain fasting lipid panel and serum calcium to evaluate metabolic causes 8, 4
  • Serum triglyceride levels >11.3 mmol/L confirm hypertriglyceridemia as the cause 8

Imaging Strategy

  • Ultrasound of the gallbladder is the first-line imaging modality and should be performed immediately 1, 8
  • Repeat biliary ultrasound if initial study is negative, as it remains sensitive for detecting missed gallstones 8
  • MRCP (without contrast) has 97.98% sensitivity and 84.4% specificity for choledocholithiasis when ultrasound is nondiagnostic 8
  • Endoscopic ultrasound can detect microlithiasis in recurrent cases when other imaging is negative 8
  • CT scanning should be reserved for cases where malignancy needs exclusion or when assessing pancreatic necrosis in severe disease 8

Clinical Pearls and Pitfalls

High-Risk Screening

  • Women with poorly controlled diabetes should have triglyceride levels checked once every trimester 2
  • Do not delay intervention until pancreatitis develops in women with triglycerides ≥500 mg/dL 2

Common Diagnostic Errors

  • The diagnosis of idiopathic pancreatitis should not be accepted without at least two good quality ultrasound examinations 8
  • Do not use low-fat, high-carbohydrate diets without carbohydrate restriction, as excessive carbohydrates are converted to triglycerides 2
  • The goal is to identify a definitive etiology in 75-80% of cases 8

Timing Considerations

  • Most cases (60%) occur in the third trimester when both gallbladder stasis and physiologic hypertriglyceridemia peak 5
  • Postpartum presentation is also common, particularly for gallstone-related disease 3, 6

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Pancreatitis in Pregnancy: Causes and Risk Factors

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Acute pancreatitis during pregnancy: a review.

Journal of perinatology : official journal of the California Perinatal Association, 2014

Research

Acute pancreatitis in pregnancy: an overview.

European journal of obstetrics, gynecology, and reproductive biology, 2011

Research

Pancreatitis in pregnancy: etiology, diagnosis, treatment, and outcomes.

Hepatobiliary & pancreatic diseases international : HBPD INT, 2016

Research

Gallstone Pancreatitis Post-Cesarean Section: A Case Report.

South Dakota medicine : the journal of the South Dakota State Medical Association, 2022

Research

Gallstone disease and pancreatitis in pregnancy.

Gastroenterology clinics of North America, 1992

Guideline

Acute Pancreatitis Risk Factors and Diagnostic Approach

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

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Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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