What are the British Society for Paediatric Endocrinology and Diabetes (BSPED) guidelines for treating diabetic ketoacidosis in a 30‑kg child?

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BSPED Guidelines for Pediatric DKA Management in a 30-kg Child

Begin immediate fluid resuscitation with 0.9% normal saline at 10-20 mL/kg (300-600 mL for a 30-kg child) over the first hour, followed 1-2 hours later by continuous IV regular insulin at 0.05-0.1 units/kg/hour (1.5-3 units/hour), while never exceeding 50 mL/kg total fluid in the first 4 hours to prevent cerebral edema. 1, 2, 3

Initial Assessment and Diagnosis

Obtain STAT laboratory studies including venous blood gas, complete blood count with differential, urinalysis, plasma glucose, BUN, electrolytes, creatinine, calcium, and phosphorus. 1 Measure β-hydroxybutyrate directly rather than using nitroprusside testing, which only detects acetoacetate and acetone. 1

Calculate corrected serum sodium by adding 1.6 mEq/L for each 100 mg/dL increase in glucose above 100 mg/dL. 1, 2 Estimate effective serum osmolality as 2 × [Na (mEq/L)] + [glucose (mg/dL)] ÷ 18. 1

Fluid Management Protocol

Phase 1: Initial Resuscitation (Hour 0-1)

Administer 0.9% normal saline at 10-20 mL/kg (300-600 mL for a 30-kg child) over the first hour. 1, 2, 3 This is the critical threshold: never exceed 50 mL/kg total fluid volume in the first 4 hours, as this is the most important factor in preventing cerebral edema in children. 1, 2

Phase 2: Ongoing Fluid Therapy (Hours 1-24)

If corrected sodium is normal or elevated, switch to 0.45% NaCl at 4-14 mL/kg/hour (120-420 mL/hour for a 30-kg child). 1, 2 If corrected sodium is low, continue 0.9% NaCl at the same rate. 1, 2

An alternative smooth-rehydration strategy is to provide 1.5 × maintenance fluids (approximately 5 mL/kg/hour for a 30-kg child). 1

Critical osmolality rule: Serum osmolality must change ≤3 mOsm/kg per hour to prevent cerebral edema. 1, 2 Recheck electrolytes, glucose, BUN, creatinine, and osmolality every 2-4 hours. 1, 3

Insulin Therapy

Do NOT give an initial IV insulin bolus in pediatric patients—this is a critical difference from adult DKA protocols. 1, 2, 3

Initiate continuous IV regular insulin infusion 1-2 hours after starting fluid resuscitation at 0.05-0.1 units/kg/hour (1.5-3 units/hour for a 30-kg child). 1, 3 This delay allows for initial volume restoration before insulin drives glucose and potassium intracellularly. 4

If plasma glucose fails to drop ≥50 mg/dL in the first hour, confirm adequate hydration and then double the insulin rate hourly until a steady decline is achieved. 1

Continue insulin until ketoacidosis resolves (pH >7.3, bicarbonate ≥15-18 mEq/L, anion gap normalized), not merely until glucose normalizes. 1, 3 This is essential because ketoacidosis resolution lags behind glucose normalization.

Potassium Management

Never add potassium until urine output is confirmed at ≥0.5 mL/kg/hour (≥15 mL/hour for a 30-kg child). 1 Never start insulin if serum potassium is <3.3 mEq/L—aggressively replace potassium first, as insulin will drive potassium intracellularly and precipitate life-threatening arrhythmias. 2, 3

Once adequate urine output is confirmed, add 20-30 mEq/L potassium to IV fluids, using a mixture of 2/3 potassium chloride and 1/3 potassium phosphate. 1, 2, 3 This combination replaces both chloride and phosphate losses while maintaining serum potassium at 4-5 mEq/L.

Transition to Dextrose-Containing Fluids

When plasma glucose falls to approximately 250 mg/dL, switch to 5% dextrose in 0.45% NaCl (or 0.45-0.75% NaCl based on sodium status) while maintaining potassium supplementation (20-30 mEq/L, 2/3 KCl + 1/3 KPO₄). 1

Continue the insulin infusion at 0.1 units/kg/hour (3 units/hour for a 30-kg child), adjusting dextrose concentration to keep glucose 150-200 mg/dL. 1 This "two-bag system" allows independent adjustment of glucose and electrolyte delivery while maintaining consistent insulin infusion.

Monitoring Protocol

Hourly monitoring: blood glucose and neurological status (mental status, headache, behavior changes). 1, 3

Every 2-4 hours: serum electrolytes (Na, K, Cl), venous pH and bicarbonate, BUN, creatinine, and osmolality. 1, 2, 3

Continuous cardiac monitoring is essential given the risk of arrhythmias from potassium shifts. 1

Severe DKA (pH <7.0, Bicarbonate <10 mEq/L)

Requires ICU admission with central venous and intra-arterial pressure monitoring. 1 More frequent blood chemistry determinations are necessary to guide therapy. 1 Children with severe DKA have a higher risk of cerebral edema and mortality. 1

Cerebral Edema Recognition and Management

Cerebral edema is the most common life-threatening complication of pediatric DKA and occurs more frequently in children than adults. 1, 5 Early warning signs include headache, confusion, behavioral changes, and declining mental status. 1, 3

If cerebral edema is suspected, immediately administer IV mannitol 0.5-1 g/kg over 15 minutes. 3 Hypertonic saline (3%) at 2.5-5 mL/kg over 10-15 minutes is an alternative. 6

Common pitfall: The most critical error is exceeding 50 mL/kg total fluid in the first 4 hours, which dramatically increases cerebral edema risk. 1, 2

Resolution Criteria and Transition

DKA is considered resolved when: glucose <200 mg/dL, serum bicarbonate ≥18 mEq/L, venous pH >7.3, and anion gap ≤12 mEq/L. 2, 3

Administer basal subcutaneous insulin 2-4 hours before stopping IV insulin to prevent rebound hyperglycemia and recurrent ketoacidosis. 3 Continue IV insulin for 1-2 hours after starting subcutaneous insulin. 1

Prevention of Recurrent DKA

Provide 24-hour telephone support for families of children with type 1 diabetes. 1 Educate caregivers on early signs of DKA and the importance of ketone monitoring during intercurrent illness. 1, 7

Emphasize that recurrent DKA is almost always due to insulin omission. 1, 4 Offer psychological support, as recurrent DKA is associated with higher rates of psychiatric illness, depression, and eating disorders. 1

Important distinction: Recent evidence suggests that early isotonic fluid therapy does not confer additional risk and may improve outcomes, leading BSPED to adopt a more permissive approach to fluid administration in their 2020 update. 5, 6 However, the critical threshold of not exceeding 50 mL/kg in the first 4 hours remains paramount. 1, 2

References

Guideline

Pediatric Diabetic Ketoacidosis (DKA) Management Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Fluid Management in Pediatric Diabetic Ketoacidosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Management of Pediatric Diabetic Ketoacidosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

The management of diabetic ketoacidosis in children.

Diabetes therapy : research, treatment and education of diabetes and related disorders, 2010

Research

A Narrative Review on Diabetic Ketoacidosis in Children.

Current pediatric reviews, 2024

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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