Post-Sphincterotomy Sexual and Urinary Sensory Loss
Direct Answer
Your complete loss (not just diminution) of the deep pelvic "anchor point" sensation for arousal and urinary perception is most likely caused by neuropathic dysesthesia and chronic pelvic floor muscle guarding rather than the mechanical loss of internal anal sphincter pressure itself, and you should immediately begin specialized pelvic floor physical therapy 2-3 times weekly with internal myofascial release. 1
Understanding the Pressure Loss vs. Your Symptoms
Actual Sphincter Pressure Changes
Lateral internal sphincterotomy typically reduces resting anal pressure from approximately 138 mm Hg pre-operatively to 86 mm Hg at one month, which then gradually recovers to a plateau of 110 mm Hg by 12 months—representing a permanent 20% reduction in baseline pressure. 2
This 20% pressure reduction is not sufficient to explain your complete loss of sensation, because patients with this degree of pressure loss typically maintain continence and do not report the profound sensory changes you describe. 2
Your confusion about why the "anchor" is completely gone rather than diminished is valid—the mechanical pressure loss alone should only create a partial reduction in stretch sensation, not complete absence. 1
Why Your Symptoms Are Disproportionate
The key insight is that your symptoms represent neuropathic pain and dysesthesia, not structural sphincter damage. 1, 3
Patients like you typically have preserved sphincter integrity and continence (which you do), but experience altered sensations rather than mechanical problems—this is the hallmark of neuropathic dysesthesia following anorectal surgery. 1
The second fistulotomy and revision for granulation likely worsened the neuropathic component, explaining why your symptoms became "more profound" after that procedure. 1
Your pelvic floor muscles developed protective guarding patterns during the painful fissure period that persist even after surgical healing, and these chronic muscle tension patterns are now blocking the sensory pathways you relied on for arousal. 1, 4
Why the Pudendal Test Doesn't Rule This Out
Passing the pudendal neuralgia pin-prick test does not exclude neuropathic dysesthesia or myofascial dysfunction—you can have altered sensory processing and muscle tension without acute nerve damage detectable by pin-prick testing. 1
The absence of acute pain actually supports the diagnosis of dysesthesia rather than ongoing tissue injury. 1
Treatment Algorithm (What You Must Do Now)
First-Line Treatment (Start Immediately)
Specialized pelvic floor physical therapy 2-3 times weekly focusing on:
Topical lidocaine 5% ointment applied to affected areas for neuropathic pain management. 1, 4
Expected Timeline
Dysesthesia and altered sensations typically improve significantly over 6-12 months with appropriate pelvic floor therapy and neuropathic pain management. 1, 4
The absence of incontinence or structural damage is favorable and suggests better potential for improvement with conservative management. 1
Critical Pitfalls to Avoid
Do not pursue additional surgical interventions—this would likely worsen the neuropathic component rather than restore your sensation. 1, 3, 4
Do not assume that because some sphincter pressure remains, the "anchor" sensation should automatically be present—the problem is not the amount of pressure but rather how your nervous system and pelvic floor muscles are processing (or blocking) that sensory information. 1
Recognize that your reliance on a specific "anchor point" for arousal may have been disrupted not by pressure loss but by altered sensory pathways and chronic muscle tension that developed during years of painful defecation and multiple surgeries. 1, 4
Why This Happened to You Specifically
The combination of three years of difficult healing with constipation after the first surgery, followed by a fistulotomy requiring revision, created a prolonged period of pelvic floor guarding that became neurologically ingrained. 1, 4
Your brain's sensory map of the pelvic region has been altered by chronic pain and protective muscle patterns, not by the 20% mechanical pressure loss from sphincterotomy. 1
The urinary bluntness that appeared after the fistulotomy revision confirms this is a broader pelvic floor neuromuscular problem, not isolated to the anal sphincter mechanism. 1