What are the causes of elevated platelet counts (thrombocytosis)?

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Causes of Elevated Platelets (Thrombocytosis)

Primary (Clonal) Thrombocytosis

Primary thrombocytosis arises from myeloproliferative neoplasms and requires sustained platelet counts ≥450 × 10⁹/L with bone marrow evidence of megakaryocyte proliferation. 1, 2

Essential Thrombocythemia (ET)

  • ET is diagnosed when platelet count remains ≥450 × 10⁹/L on repeat testing, with bone marrow showing proliferation of enlarged, mature megakaryocytes without left-shift of other lineages. 3, 2
  • JAK2 V617F mutation is present in 50-60% of ET patients, making it the primary molecular diagnostic marker. 2
  • When JAK2 V617F is negative, proceed with CALR and MPL mutation testing to identify clonal disease. 2
  • Diagnosis mandates exclusion of polycythemia vera, primary myelofibrosis, chronic myeloid leukemia, and myelodysplastic syndromes. 2

Polycythemia Vera (PV)

  • PV harbors JAK2 V617F mutation in >90% of cases, with diagnostic hemoglobin thresholds ≥18.5 g/dL in men or ≥16.5 g/dL in women. 2
  • Iron deficiency can mask PV by normalizing hemoglobin levels, making iron studies essential before excluding this diagnosis. 2
  • Serum erythropoietin levels are typically subnormal in PV. 2

Primary Myelofibrosis (PMF)

  • PMF shows JAK2 V617F mutation in approximately 50% of patients, with bone marrow demonstrating atypical megakaryocyte proliferation and reticulin or collagen fibrosis. 2
  • PMF frequently presents with leukoerythroblastosis, elevated LDH, anemia, and splenomegaly. 2

Chronic Myeloid Leukemia (CML)

  • CML can cause thrombocytosis and requires BCR-ABL1 testing for exclusion. 1, 2

Secondary (Reactive) Thrombocytosis

Secondary thrombocytosis accounts for 83% of all cases and arises from identifiable underlying conditions that stimulate platelet production. 4

Tissue Damage and Trauma

  • Surgery, burns, and trauma represent the most common cause of secondary thrombocytosis (32.2% of cases). 4
  • Post-surgical states and trauma are recognized triggers of reactive thrombocytosis. 2

Infections

  • Acute bacterial or viral infections cause secondary thrombocytosis through inflammatory cytokine production. 1
  • Specific infections include HIV, hepatitis C, Helicobacter pylori, parvovirus, and cytomegalovirus. 2
  • In pediatric empyema, platelet counts >500 × 10⁹/L occur in 93% of cases, peaking at 2 weeks and normalizing by 3 weeks without thromboembolic complications. 2, 5

Chronic Inflammatory Disorders

  • Inflammatory bowel disease and rheumatoid arthritis cause sustained platelet elevation (11.7% of secondary cases). 4, 2
  • Adult-onset Still's disease frequently presents with reactive thrombocytosis that parallels disease activity. 2

Malignancy

  • Solid tumors and lymphoproliferative disorders cause thrombocytosis through inflammatory cytokine production. 1
  • Malignancy-associated thrombocytosis confers increased thrombotic risk, warranting consideration of prophylaxis. 2

Iron Deficiency Anemia

  • Iron deficiency causes secondary thrombocytosis in 11.1% of cases; iron replacement normalizes platelet counts when this is the sole driver. 4, 2
  • Iron studies (ferritin, serum iron, TIBC) are mandatory to detect iron-deficiency-related thrombocytosis. 2

Post-Splenectomy or Hyposplenism

  • Splenectomy or functional asplenia produces sustained platelet elevation. 2

Autoimmune Conditions

  • Antiphospholipid antibody syndrome and systemic lupus erythematosus may induce thrombocytosis. 2
  • Positive ANA testing can help uncover underlying autoimmune etiologies. 2

Drug-Induced

  • Medications including corticosteroids, epinephrine, and erythropoiesis-stimulating agents are recognized causes of secondary thrombocytosis. 1, 2

Diagnostic Approach

Confirm true thrombocytosis by documenting sustained platelet count ≥450 × 10⁹/L on repeat measurement before initiating extensive workup. 2

Initial Laboratory Evaluation

  • Review complete blood count for other cell line abnormalities (elevated hemoglobin suggests PV, leukoerythroblastosis suggests PMF). 1, 2
  • Obtain peripheral blood smear to assess for dysplasia, immature cells, or morphologic abnormalities. 1
  • Order iron studies (ferritin, serum iron, TIBC) to exclude iron deficiency as the cause. 2
  • Measure inflammatory markers (CRP/ESR) to identify underlying inflammatory conditions. 2

Molecular Testing for Primary Thrombocytosis

  • First-line molecular testing is JAK2 V617F assay; if negative, proceed with CALR and MPL mutation analysis. 2
  • Approximately 86% of primary thrombocytosis patients have at least one molecular marker indicative of myeloproliferative neoplasms. 4

Bone Marrow Examination

  • Bone marrow biopsy with aspirate is required when primary disorder is suspected to assess megakaryocyte morphology, cellularity, and fibrosis. 2
  • Bone marrow examination is recommended for individuals older than 60 years or those with systemic symptoms to exclude myelodysplastic syndromes or acute leukemias. 2
  • Bone marrow examination is not indicated in children with classic features of secondary thrombocytosis. 3, 5

Additional Testing

  • BCR-ABL1 testing is essential to exclude chronic myeloid leukemia. 2
  • Hemoglobin and hematocrit measurements help rule out polycythemia vera. 2

Thrombotic Risk Stratification

The median platelet count and incidence of thrombosis are significantly higher in primary thrombocytosis than secondary thrombocytosis. 4

Primary Thrombocytosis

  • Primary thrombocytosis carries substantial thrombotic risk requiring risk stratification to guide cytoreductive therapy and antiplatelet agents. 2

Secondary Thrombocytosis

  • Secondary thrombocytosis rarely leads to thrombosis, even at very high platelet counts. 2
  • In pediatric empyema with platelets >500 × 10⁹/L, platelet function remains normal and no thromboembolic events occur. 2, 5
  • Antiplatelet therapy is not indicated for secondary thrombocytosis unless other cardiovascular indications exist. 1, 2, 5

Management

Secondary Thrombocytosis

  • Identify and treat the underlying condition; avoid unnecessary antiplatelet or cytoreductive agents. 2
  • Re-measure platelet count after resolution of the primary disorder to confirm normalization. 2
  • If thrombocytosis persists beyond the expected timeframe, consider bone marrow evaluation to exclude occult myeloproliferative neoplasm. 2
  • No specific treatment is necessary for secondary thrombocytosis in children. 5

Primary Thrombocytosis

  • Cytoreductive therapy (hydroxyurea or anagrelide) and low-dose aspirin (81-100 mg/day) may be indicated for high-risk patients based on thrombotic risk stratification. 1, 2

Common Pitfalls

  • Avoid diagnosing essential thrombocythemia before excluding iron deficiency with trial of iron replacement, as occult polycythemia vera may be masked. 1
  • In malignancy-related disseminated intravascular coagulation, a decreasing platelet trend from an initially elevated level may be the only sign of DIC, even if absolute count remains normal. 1
  • Do not initiate antiplatelet therapy for secondary thrombocytosis, as it provides no benefit and increases bleeding risk. 5

References

Guideline

Thrombocytosis Causes and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guidelines for Evaluation and Management of Thrombocytosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Management of Thrombocytosis in Children

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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