What is the prevalence of light-chain monoclonal gammopathies (e.g., light-chain MGUS, light-chain deposition disease, or AL amyloidosis) in patients with Crohn’s disease?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 22, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Prevalence of Light Chain Disease in Crohn's Disease

There is no established association or documented prevalence of light chain monoclonal gammopathies (light-chain MGUS, light-chain deposition disease, or AL amyloidosis) specifically in patients with Crohn's disease. The available evidence does not address this relationship, and these are considered distinct, unrelated disease entities.

Key Context on Light Chain Disorders

Light chain monoclonal gammopathies represent a spectrum of plasma cell disorders that are not known to have any epidemiologic or pathophysiologic connection to inflammatory bowel disease:

General Population Prevalence

  • Light-chain MGUS occurs in approximately 0.8% of the general population aged ≥50 years, contributing to an overall MGUS prevalence of 4.2% when combined with conventional MGUS 1
  • The prevalence is age-dependent, increasing in older populations, with no documented variation based on inflammatory bowel disease status 1

Clinical Manifestations of Light Chain Disease

Light chain disorders primarily affect:

  • Kidneys (most common): light-chain deposition disease, AL amyloidosis, cast nephropathy 2
  • Heart: cardiac AL amyloidosis is the main driver of mortality 2
  • Peripheral nerves, liver, and gastrointestinal tract (less commonly) 2

Gastrointestinal Involvement Considerations

While light chain deposition disease can rarely involve the gastrointestinal tract:

  • LCDD presenting with GI symptoms mimicking inflammatory bowel disease is exceptionally rare and represents case report-level occurrences 3
  • When GI involvement occurs, it is due to direct tissue deposition of monoclonal light chains, not an association with underlying Crohn's disease 3, 4
  • Diagnosis requires tissue biopsy showing extracellular deposits that are Congo red-negative (for LCDD) or Congo red-positive (for AL amyloidosis), with immunohistochemistry demonstrating monoclonal light chain restriction 3, 4

Clinical Pitfall to Avoid

Do not confuse coincidental findings with causative associations. If a patient with known Crohn's disease develops proteinuria, renal insufficiency, or unexplained systemic symptoms, evaluate for monoclonal gammopathy as you would in any patient—based on clinical suspicion from organ dysfunction, not based on their Crohn's disease diagnosis 2.

When to Screen for Light Chain Disease

Screen for monoclonal gammopathy when patients present with:

  • Unexplained proteinuria >0.5 g/24h, especially with renal insufficiency 2
  • Cardiac dysfunction with heart failure and preserved ejection fraction 2
  • Peripheral neuropathy without alternative explanation 2
  • Unexplained GI symptoms with extracellular deposits on biopsy 3

The diagnostic workup includes serum and urine protein electrophoresis with immunofixation, serum free light chain assay with kappa/lambda ratio, and tissue biopsy with immunofluorescence and electron microscopy when organ involvement is suspected 2.

Related Questions

Do patients with inflammatory bowel disease have an increased risk of light‑chain monoclonal gammopathies, and should they be screened for these disorders?
What is the most likely cause of impaired renal function in a 60-year-old male with persistent back pain unresponsive to Non-Steroidal Anti-Inflammatory Drugs (NSAIDs), normal electrolyte levels, elevated creatinine levels, and 20% plasma cells in the bone marrow biopsy?
What are the differential diagnoses for a patient with an M-spike (monoclonal spike) in the beta-2 globulin region and abnormal free lambda light chains, aside from Monoclonal Gammopathy of Undetermined Significance (MGUS)?
What can cause a normal kappa light chain level in the serum and a very high kappa light chain level in the urine in an older adult with possible history of hematological disorders or kidney disease?
When should a patient with elevated kappa (κ) light chains and hematuria be referred to a hematologist?
In a healthy adult, what are the benefits of creatine monohydrate and what is the recommended daily dosage and timing for supplementation?
When does creatine kinase (CK) reach its peak level after the onset of myocardial infarction?
How should the estimated glomerular filtration rate (eGFR) be calculated and interpreted for a Black adult female?
In adult patients with decompensated cirrhosis and hepatorenal syndrome who cannot receive terlipressin or norepinephrine, is octreotide appropriate as adjunct therapy, and what dosing regimen, combination with midodrine and albumin, monitoring parameters, and criteria for escalation should be used?
What is the normal estimated glomerular filtration rate (eGFR) range for a healthy adult, including Black women, and is any race adjustment required when using the current race‑free CKD‑EPI creatinine equation?
What non‑prescription alternative therapies are appropriate for an older adult with chronic obstructive pulmonary disease experiencing a severe exacerbation with tachycardia and tachypnea?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.