What are the common congenital heart defects in females with Turner syndrome diagnosed in childhood or adolescence?

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Common Congenital Heart Defects in Turner Syndrome

Bicuspid aortic valve is the most common congenital heart defect in Turner syndrome, occurring in 15-39% of patients, followed by aortic coarctation in 7-21% of cases. 1, 2, 3

Primary Cardiac Malformations

Bicuspid Aortic Valve (Most Common)

  • Bicuspid aortic valve affects 15-30% of Turner syndrome patients according to major guidelines, though recent echocardiographic studies using optimized imaging techniques report prevalence as high as 39%. 1, 2, 4, 3
  • This represents the single most frequent cardiac anomaly, surpassing the historically reported predominance of coarctation. 5, 3
  • A systolic ejection click on physical examination correlates with bicuspid aortic valve in 82% of cases. 5

Aortic Coarctation (Second Most Common)

  • Aortic coarctation occurs in 7-18% of Turner syndrome patients, with most recent large cohort studies reporting 21% prevalence. 6, 1, 2, 3
  • The typical location is juxtaductal, just distal to the left subclavian artery. 6
  • Coarctation is independently associated with increased aortic dissection risk and requires lifelong surveillance. 1, 4

Ascending Aortic Dilation

  • Approximately 33% of Turner syndrome patients develop ascending aortic dilation, representing a major cause of premature cardiovascular mortality. 1, 2, 4
  • This is an acquired progressive condition rather than a congenital defect per se, but reflects intrinsic aortic wall abnormalities present from birth. 6

Other Structural Defects

  • Partial anomalous pulmonary venous drainage occurs at significantly higher rates than in the general population. 7
  • Ventricular septal defects, atrial septal defects, and other valve abnormalities occur less frequently but should be screened. 6, 8
  • Overall, 50-56% of Turner syndrome patients have some form of structural cardiac anomaly. 2, 3

Critical Diagnostic Approach

Mandatory Initial Evaluation

  • All patients with Turner syndrome require comprehensive cardiovascular evaluation at diagnosis with BOTH transthoracic echocardiography AND cardiac MRI. 1, 4
  • Echocardiography alone misses critical abnormalities, particularly mid-ascending aortic pathology. 1, 4
  • The echocardiogram must include suprasternal and high parasternal windows; failure to obtain these views is associated with missed diagnoses. 3

Karyotype-Specific Patterns

  • The 45,X monosomy karyotype is associated with the most severe malformations. 7
  • Ring chromosome patterns show very high incidence of bicuspid aortic valve. 7
  • X isochromosome patterns may have lower rates of cardiac anomalies. 7

Common Diagnostic Pitfalls

The most critical error is relying solely on transthoracic echocardiography without cardiac MRI, which systematically misses mid-ascending aortic pathology and subtle coarctation. 1, 4

Using absolute aortic measurements without indexing to body surface area (calculating Aortic Size Index) dramatically underestimates dissection risk due to the characteristic short stature in Turner syndrome. 1, 2, 4

Incomplete echocardiographic windows—particularly omitting suprasternal and high parasternal views—results in failure to identify hemodynamically significant lesions. 3

References

Guideline

Cardiovascular Management in Turner Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Clinical Features and Management of Turner Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Cardiovascular Management in Turner Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Heart disease in Turner's syndrome.

Helvetica paediatrica acta, 1988

Research

Turner syndrome and the heart: cardiovascular complications and treatment strategies.

American journal of cardiovascular drugs : drugs, devices, and other interventions, 2002

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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