In an adult presenting to the emergency department with acute chest pain, how should I trend high‑sensitivity cardiac troponin (hs‑cTn) using the 0‑ and 1‑hour European Society of Cardiology (ESC) algorithm to rule in or rule out myocardial infarction?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 23, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Trending High-Sensitivity Cardiac Troponin in Acute Chest Pain

Use the 0- and 1-hour ESC algorithm as your first-line approach: obtain blood at presentation (0 h) and exactly 1 hour later, then apply assay-specific cutoffs for both absolute values and delta changes to rule-out or rule-in myocardial infarction. 1

Algorithm Selection and Timing

The 2021 European Society of Cardiology guidelines designate the 0/1-hour protocol as the best option for hs-cTn trending, with the 0/2-hour protocol as the second-best alternative. 1 The 2022 American College of Cardiology consensus acknowledges multiple validated pathways but notes the 0/1-hour algorithm takes advantage of superior assay sensitivity and precision while ruling out the largest proportion of patients. 1

Blood Draw Timing Requirements

  • Draw the first sample immediately at presentation (time 0) regardless of symptom duration. 2
  • Draw the second sample at exactly 1 hour after the first draw when using the 0/1-hour algorithm—timing precision is critical to algorithm validity. 1
  • If your institution cannot reliably obtain 1-hour samples, use the 0/2-hour algorithm instead, which is equally complex but more practical for some centers. 1

ESC 0/1-Hour Algorithm: Three-Pathway Approach

The algorithm stratifies patients into three groups using assay-specific cutoffs for absolute troponin concentrations and delta changes:

Rule-Out Pathway (Low Risk)

Patients meet rule-out criteria when both conditions are satisfied: 1

  • Very low baseline hs-cTn at 0 hours (assay-specific thresholds: <5 ng/L for Roche hs-cTnT, <4 ng/L for Abbott hs-cTnI, <3 ng/L for Siemens hs-cTnI), OR
  • Low baseline hs-cTn at 0 hours (<12 ng/L for Roche, <5 ng/L for Abbott, <6 ng/L for Siemens) AND minimal 1-hour delta (<3 ng/L for Roche, <2 ng/L for Abbott, <3 ng/L for Siemens)

This pathway achieves a negative predictive value exceeding 99% for myocardial infarction in validation cohorts. 1 Patients in this category can be safely discharged from the emergency department with outpatient follow-up. 1

Critical caveat: The very-low baseline rule-out applies only if chest pain onset occurred >3 hours before presentation—earlier presenters require the full 0- and 1-hour serial assessment. 1, 2

Rule-In Pathway (High Risk)

Patients meet rule-in criteria when either condition is present: 1

  • High baseline hs-cTn at 0 hours (≥52 ng/L for Roche, ≥64 ng/L for Abbott, ≥120 ng/L for Siemens), OR
  • Significant 1-hour delta (≥5 ng/L for Roche, ≥6 ng/L for Abbott, ≥12 ng/L for Siemens) regardless of baseline value

The positive predictive value for myocardial infarction in the rule-in group is approximately 75%. 1 Most rule-in patients without MI have conditions requiring invasive coronary angiography or cardiac MRI (Takotsubo syndrome, myocarditis), so admission to a coronary care unit and early angiography are appropriate. 1

Observation Zone (Intermediate Risk)

Patients who meet neither rule-out nor rule-in criteria fall into the observation zone and require additional testing: 1

  • Repeat hs-cTn at 3 hours from presentation
  • Perform risk stratification with clinical scores (HEART, TIMI)
  • Consider provocative testing (stress imaging, coronary CT angiography) if serial troponins remain in the observation range

Alternative Protocols When 0/1-Hour Is Not Feasible

0/2-Hour Algorithm

Identical three-pathway logic as 0/1-hour but with the second draw at 2 hours. 1 This protocol is better for very early presenters (symptom onset <2 hours) because it allows more time for troponin release, reducing false-negative results on the declining limb of late-presenting infarctions. 1

0/3-Hour Algorithm

Use a single hs-cTn <99th percentile if symptoms began >6 hours ago and the patient is now pain-free; otherwise, require both 0- and 3-hour values <99th percentile. 1 This approach has lower sensitivity and negative predictive value than 0/1-hour or 0/2-hour protocols and rules out fewer patients. 1 The 2021 ESC guidelines note that three recent large studies show the 0/3-hour algorithm balances efficacy and safety less favorably than the 0/1-hour protocol. 1

Assay-Specific Cutoffs for the 0/1-Hour Algorithm

You must use the validated thresholds for your institution's specific troponin assay—cutoffs are not interchangeable: 1

Assay Very Low (0 h) Low (0 h) No Delta (1 h) High (0 h) Delta (1 h)
Roche hs-cTnT <5 ng/L <12 ng/L <3 ng/L ≥52 ng/L ≥5 ng/L
Abbott hs-cTnI <4 ng/L <5 ng/L <2 ng/L ≥64 ng/L ≥6 ng/L
Siemens hs-cTnI <3 ng/L <6 ng/L <3 ng/L ≥120 ng/L ≥12 ng/L

Common Pitfalls and How to Avoid Them

Timing Errors

Never use emergency department arrival time as "time 0" if the exact blood draw time is documented—the algorithm requires precise 1-hour intervals between samples, not approximate times. 1 A sample drawn at 55 minutes or 75 minutes invalidates the algorithm's validated cutoffs.

Early Presenters

Do not apply the single very-low troponin rule-out to patients presenting <3 hours from symptom onset—troponin may not yet be detectable even in evolving MI. 1, 2 These patients require the full serial 0- and 1-hour assessment.

Late Presenters on the Declining Curve

The 0/1-hour algorithm may miss late-presenting MI when troponin is on the flat portion of a declining curve (e.g., patient presents 18 hours after symptom onset with troponin of 45 ng/L at 0 h and 43 ng/L at 1 h). 1 In patients with symptom onset >12 hours ago, consider extending observation and repeating troponin at 3–6 hours to capture the full kinetic pattern. 2

Non-ACS Troponin Elevation

Troponin elevation is non-specific—heart failure, pulmonary embolism, myocarditis, renal failure, and sepsis all elevate troponin. 1, 2 The rule-in pathway identifies myocardial injury, not necessarily acute coronary syndrome. Integrate ECG findings, echocardiography, and clinical context to differentiate ischemic from non-ischemic causes. 1

Chronic Troponin Elevation

Patients with chronic kidney disease, heart failure, or stable coronary disease often have baseline troponin above the 99th percentile. 2 In these patients, serial trend matters more than absolute value—a change <20% does not meet the diagnostic threshold for acute injury. 1, 2

Implementation in Clinical Practice

  • Hospitals should adopt a standardized chest-pain protocol specifying troponin sampling intervals according to assay type (conventional vs. high-sensitivity). 2
  • Central laboratory turnaround time for troponin results must be ≤1 hour to enable timely decision-making with the 0/1-hour algorithm. 2
  • Real-world implementation studies, including one randomized controlled trial, confirm the very high safety and efficacy of the ESC 0/1-hour algorithm when applied in routine emergency department practice. 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Troponin Monitoring Protocol for Acute Chest Pain

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Related Questions

Is it advisable to perform an electrocardiogram (ECG) at 0 hours and at 1 hour in patients with suspected acute coronary syndrome (ACS) when using high-sensitivity troponin I (hs-TnI)?
How often are cardiac enzymes, such as troponin, checked in the Emergency Room (ER)?
What is the recommended protocol for ordering serial cardiac enzymes, including troponin levels, in patients with suspected acute coronary syndrome?
How do you interpret and use high sensitivity troponin (hs-troponin) in diagnosing acute myocardial infarction (AMI)?
What troponin assay and values are recommended for excluding myocardial infarction (MI) in chest pain lasting >3 hours, and is a second result needed?
A 46‑year‑old woman presents with extreme fatigue, laboratory evidence of iron deficiency (low ferritin, low transferrin saturation) and a perimenopausal hormone profile (luteinizing hormone 14 IU/L, follicle‑stimulating hormone 18 IU/L, estradiol 32 pg/mL, progesterone 0.5 ng/mL, dehydroepiandrosterone sulfate 33 µg/dL); what is the appropriate evaluation and management?
Can Belbuca (buccal buprenorphine) be used safely in a patient with heart failure and atrial fibrillation, and what precautions are needed?
What is the mechanism of mammography in breast cancer screening?
As a primary care provider, what steps should I take when a patient’s MRI reveals a partially empty sella?
In a symptomatic child with rising anti‑Typhi H antibody titers and negative anti‑Typhi O, how should the suspected typhoid infection be managed?
In a hypotensive patient with coronary artery disease and a heart rate of 203 beats per minute, what is the appropriate management?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.