Fetal Heart Rate >160 bpm with Strong Contractions: Immediate Management
When fetal heart rate exceeds 160 bpm during strong contractions, immediately implement intrauterine resuscitation measures while simultaneously assessing for the two most critical causes: chorioamnionitis (especially if membranes are ruptured) and fetal hypoxia from uteroplacental insufficiency. 1, 2
Immediate Resuscitative Actions
Perform these interventions without delay:
- Discontinue oxytocin infusion immediately if being administered, as uterine hyperstimulation can cause both fetal tachycardia and uteroplacental hypoperfusion 1, 3
- Reposition the mother to left lateral decubitus to improve uteroplacental blood flow and relieve potential cord compression 1, 2
- Administer supplemental oxygen at 6-10 L/min via face mask to maximize fetal oxygenation 1, 2
- Give an intravenous fluid bolus to increase maternal intravascular volume and uteroplacental perfusion 1, 2
Critical Diagnostic Assessment
While implementing resuscitation, immediately evaluate:
- Measure maternal temperature and complete vital signs to detect fever or infection—chorioamnionitis is the most frequent cause of fetal tachycardia after membrane rupture 1, 2
- Assess baseline fetal heart rate variability on the monitor—tachycardia with moderate variability is far less concerning than tachycardia with minimal or absent variability 1, 4
- Perform or review recent vaginal examination to document cervical dilation, fetal station, and rule out rapid labor progression 1
- Check for presence of decelerations—recurrent late or variable decelerations accompanying tachycardia suggest evolving fetal compromise 4, 5
Infection-Specific Management
If chorioamnionitis is suspected based on maternal fever, uterine tenderness, or foul-smelling amniotic fluid:
- Initiate broad-spectrum intravenous antibiotics immediately without waiting for culture results 1, 2
- Switch to continuous electronic fetal monitoring if using intermittent auscultation, as infection requires closer surveillance 1, 2
Ongoing Monitoring Strategy
- Maintain continuous electronic fetal monitoring to detect development of late decelerations or loss of variability that would signal worsening fetal status 1, 4
- Consider internal fetal scalp electrode placement if external monitoring provides poor signal quality, as accurate assessment of baseline variability is essential for risk stratification 1, 2
- Apply NICHD categorization: Category I (normal baseline 110-160 bpm with moderate variability) allows continued monitoring; Category II (indeterminate, including isolated tachycardia) requires increased surveillance and interventions; Category III (tachycardia with absent variability AND recurrent late/variable decelerations or bradycardia) mandates expedited delivery 4
Indications for Expedited Delivery
Proceed to operative vaginal delivery or cesarean section if:
- Tachycardia persists with absent baseline variability AND recurrent late or prolonged decelerations (Category III tracing)—this combination predicts current or impending fetal asphyxia 4, 1
- Intrauterine resuscitation and infection treatment fail to improve the tracing over a reasonable observation period 1
- Progressive loss of variability occurs despite interventions, as this markedly raises concern for fetal acidosis 1, 5
Critical Pitfalls to Avoid
- Do not assume all fetal tachycardia represents distress—true fetal supraventricular tachycardia (heart rate typically >200 bpm, often with loss of monitor contact) can mimic distress and lead to unnecessary cesarean delivery 6
- Do not rely on tachycardia alone to make delivery decisions—the presence or absence of baseline variability is the key discriminator between compensated and decompensated fetal status 4, 1, 5
- Recognize that hydralazine administration for maternal hypertension can cause fetal tachycardia as a side effect, requiring close monitoring but not necessarily indicating fetal compromise 4
- Normal fetal heart rate is 110-160 bpm; rates of 150-160 bpm with strong contractions may represent normal upper baseline rather than pathologic tachycardia if variability and accelerations are preserved 4, 7