Which enzymatic deficiency most characteristically causes increased phosphoribosyl pyrophosphate (PRPP) levels?

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PRPP Synthetase Superactivity

PRPP synthetase superactivity (not a deficiency, but rather enzyme overactivity) is the enzymatic abnormality that most characteristically causes increased phosphoribosyl pyrophosphate (PRPP) levels. 1

Key Distinction: Overactivity vs. Deficiency

The question asks about enzymatic deficiency causing increased PRPP, but this represents a common conceptual pitfall. Increased PRPP levels result from PRPP synthetase superactivity (gain-of-function), not deficiency. 1 This is an important diagnostic distinction when evaluating patients with hyperuricemia and neurological symptoms.

Diagnostic Context and Testing Algorithm

When evaluating elevated uric acid levels, particularly in the context of autism spectrum disorders or unexplained hyperuricemia:

  • First, measure serum and urine uric acid levels 1
  • If elevated, proceed with testing for:
    • Hypoxanthine-guanine phosphoribosyltransferase (HGPRT) enzyme activity 1
    • PRPP synthetase superactivity testing 1
  • If uric acid is low, consider a purine/pyrimidine panel (measuring uracil excretion, xanthine, hypoxanthine) 1

HGPRT Deficiency: The Alternative Cause of Hyperuricemia

While PRPP synthetase superactivity increases PRPP directly, HGPRT deficiency also leads to increased PRPP accumulation indirectly because HGPRT normally consumes PRPP in the purine salvage pathway. 2, 3

HGPRT Deficiency Clinical Spectrum:

  • Complete deficiency causes Lesch-Nyhan syndrome with severe action dystonia, choreoathetosis, cognitive deficits, self-injurious behavior, and uric acid overproduction 2
  • Partial deficiency (Kelley-Seegmiller syndrome) presents with variable neurological symptoms ranging from mild to severe, always with hyperuricemia 3
  • All HGPRT-deficient patients demonstrate uric acid overproduction associated with lithiasis and gout 2
  • X-linked recessive inheritance affects males primarily, with females as carriers 2

Diagnostic Approach for HGPRT Deficiency:

  • Clinical findings: Hyperuricemia, hyperuricosuria, psychomotor delay 2
  • Enzymatic testing: HGPRT activity determination in hemolysate, intact erythrocytes, or fibroblasts 2
  • Molecular testing: Over 300 disease-associated mutations in the HPRT1 gene have been identified on Xq26 2, 4

Critical Clinical Pitfall

Do not confuse enzyme deficiency with enzyme superactivity. The question's phrasing suggests a deficiency causing increased PRPP, but increased PRPP results from PRPP synthetase overactivity. However, HGPRT deficiency can also lead to PRPP accumulation through impaired consumption in the salvage pathway. 1, 2

Management Considerations

  • For uric acid overproduction: Allopurinol treatment with carefully adjusted doses to avoid xanthine lithiasis 2
  • For neurological manifestations: Benzodiazepines and GABA inhibitors like baclofen for spasticity and dystonia 2
  • Physical rehabilitation: Management of dysarthria, dysphagia, hand control devices, walking aids, and posture management 2

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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