What is the most likely new diagnosis in a patient with persistent dyspnea after stopping a blood transfusion and administering diuretics, who has fever, borderline hypotension, and marginal oxygen saturation on a 35% venturi mask?

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Transfusion-Related Acute Lung Injury (TRALI)

The most likely diagnosis is Transfusion-Related Acute Lung Injury (TRALI), not fluid overload, and the critical error was administering diuretics—which are ineffective and potentially harmful in TRALI. 1, 2

Why This Is TRALI, Not TACO

The clinical presentation definitively points to TRALI based on three key features:

  • Hypotension (BP 105/62) is the diagnostic hallmark that distinguishes TRALI from transfusion-associated circulatory overload (TACO), which would present with hypertension and cardiovascular changes suggesting fluid overload 1, 2
  • Fever (39.1°C) accompanying respiratory distress is a common symptom of TRALI and indicates immune-mediated lung injury rather than simple volume overload 1, 2
  • Lack of response to diuretics confirms this is non-cardiogenic pulmonary edema; TACO would improve with diuretic therapy, whereas TRALI does not respond because the mechanism is capillary leak, not volume overload 1, 3

Pathophysiology and Timing

TRALI characteristically presents with non-cardiogenic pulmonary edema within 1-2 hours after transfusion, with the hallmark triad of hypoxemia, dyspnea, and hypotension. 1 The mechanism involves donor leukocyte antibodies (HLA class I, class II, or granulocyte-specific antibodies) interacting with recipient neutrophils, causing acute respiratory distress and cardiovascular instability. 1, 2

The patient's symptoms developed during or shortly after transfusion and persisted despite stopping the transfusion and administering diuretics, which is consistent with the typical TRALI timeline of onset within 6 hours of transfusion. 2, 4, 3

Immediate Management Required NOW

Stop all further transfusions immediately and do NOT give more diuretics. 1, 2 The correct management includes:

  • Administer 100% oxygen to address the severe hypoxemia (current O2 sat 94% on 35% venturi mask is inadequate) 1, 2
  • Prepare for intubation and mechanical ventilation given the persistent dyspnea and marginal oxygenation 1
  • Maintain blood pressure with IV fluid resuscitation using normal saline or lactated Ringer's solution, not diuretics 1
  • Call for critical care support immediately as TRALI is a leading cause of transfusion-related mortality despite being underdiagnosed 1

Critical Pitfall Already Made

The administration of diuretics was based on the incorrect assumption that this was fluid overload (TACO). 1 TRALI requires supportive care with oxygen therapy and critical care measures, not volume removal. 1, 3 Diuresis is not indicated in TRALI and can worsen hypotension. 3

Post-Stabilization Actions

  • Report the reaction to the blood bank immediately to remove the implicated donor from the pool, as required by the FDA 1
  • Continue observation for at least 24 hours, as TRALI can evolve over the first 6-12 hours post-transfusion 1
  • Send post-reaction blood samples for complete blood count, direct antiglobulin test, repeat crossmatch, PT, aPTT, and fibrinogen to exclude hemolytic reaction 1

Expected Recovery

When managed with immediate supportive care, most patients with TRALI recover within 96 hours of symptom onset. 1 The prognosis is substantially better than most causes of acute lung injury when recognized and treated appropriately. 5

References

Guideline

Diagnosis and Management of Transfusion-Related Acute Lung Injury (TRALI)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Transfusion-Related Acute Lung Injury (TRALI) Diagnosis and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Transfusion-related acute lung injury: past, present, and future.

American journal of clinical pathology, 2008

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Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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