Can you provide detailed, high‑yield, board‑style notes for Chapter 191 (Antiviral Drugs) of Fitzpatrick 9th edition, organized with must‑know highlights for dermatology specialty exams?

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Chapter 191: Antiviral Drugs – High-Yield Board Notes

HERPES SIMPLEX VIRUS (HSV) ANTIVIRALS

First-Line Oral Agents

Valacyclovir, famciclovir, and acyclovir are the three FDA-approved first-line oral antivirals for HSV, with valacyclovir and famciclovir preferred due to superior dosing convenience. 1

Herpes Labialis (Cold Sores)

  • Valacyclovir 2g PO twice daily for 1 day – reduces episode duration by 1.0 day 1
  • Famciclovir 1500mg PO single dose – equally effective, convenient single-day dosing 1
  • Critical timing: Initiate at earliest prodrome or within 24 hours of symptom onset – peak viral titers occur in first 24 hours 1

Genital Herpes – First Episode

  • Acyclovir 400mg PO three times daily for 7-10 days 1
  • Valacyclovir 1g PO twice daily for 10 days (alternative) 1
  • Extend treatment if healing incomplete after 10 days 2

Orofacial Herpes – First Episode or Severe

  • Valacyclovir 1g PO twice daily for 7-10 days 2
  • Higher doses for oral/pharyngeal involvement: Acyclovir 400mg PO five times daily 2

Ocular HSV (Keratitis/Conjunctivitis with Corneal Involvement)

  • Acyclovir 200-400mg PO five times daily PLUS topical antiviral 1
  • Valacyclovir 500mg PO 2-3 times daily (alternative) 1
  • Never use topical antivirals alone – substantially less effective than systemic therapy 2

Intravenous Acyclovir

Indications

  • Severe HSV infections
  • Immunocompromised patients with severe disease
  • HSV encephalitis

Dosing

  • Standard: 5mg/kg IV every 8 hours
  • Immunocompromised: Acyclovir 400mg PO 3-5 times daily or 5mg/kg IV every 8 hours 2

Critical Administration Details

  • Infuse over >10 minutes – rapid infusion (<10 minutes) causes transient elevations in serum creatinine/BUN in 5-10% of patients 3
  • Inflammation/phlebitis at injection site occurs in ~9% 3

Adverse Effects

  • Most common: Nausea/vomiting (7%), inflammation at injection site (9%) 3
  • Renal toxicity: Elevated creatinine/BUN (5-10%), renal failure (rare) 3
  • Neurologic: Encephalopathy, seizures, confusion, hallucinations, tremor – particularly in elderly 3
  • Hematologic: Anemia, neutropenia, thrombocytopenia (<1%) 3
  • Severe: Stevens-Johnson syndrome, toxic epidermal necrolysis, tissue necrosis with extravasation 3

Acyclovir-Resistant HSV

If lesions persist despite 7-10 days of appropriate valacyclovir therapy, suspect HSV resistance – all acyclovir-resistant strains are also resistant to valacyclovir. 2

Resistance Rates

  • Immunocompetent hosts: <0.5% 1
  • Immunocompromised patients: 7% 1

Treatment of Resistant HSV

  • IV foscarnet 40mg/kg every 8 hours until clinical resolution – treatment of choice 1, 2

Safety Profile

  • All oral antivirals (acyclovir, valacyclovir, famciclovir) are generally well-tolerated with minimal adverse events 1
  • Common side effects: Headache (<10%), nausea (<4%), diarrhea (mild-moderate) 1

VARICELLA-ZOSTER VIRUS (VZV) ANTIVIRALS

Approved Drug Classes

  • Acyclic guanosine analogues (acyclovir, valacyclovir, famciclovir)
  • Nucleoside analogues
  • 5-substituted 2'-deoxyuridine analogues
  • Antibodies 4

CYTOMEGALOVIRUS (CMV) ANTIVIRALS

First-Line Agents

  • Acyclic guanosine analogues (ganciclovir, valganciclovir)
  • Acyclic nucleoside phosphonate analogues (cidofovir)
  • Pyrophosphate analogues (foscarnet)
  • Oligonucleotides 4

Valganciclovir (Oral Prodrug of Ganciclovir)

Critical Drug Interactions

  • Zidovudine: Causes neutropenia and anemia – monitor CBC with differential and hemoglobin frequently 5
  • Probenecid: Increases ganciclovir levels – monitor for ganciclovir toxicity 5
  • Mycophenolate mofetil (MMF): Increases ganciclovir and MMF metabolite concentrations in renal impairment – monitor for toxicity of both drugs 5
  • Didanosine: Increases didanosine concentrations – monitor for didanosine toxicity 5

Ganciclovir

  • Milestone drug for systemic and organ-specific CMV disease 6
  • Nucleoside analogue targeting viral DNA polymerase 6

Foscarnet

  • Pyrophosphate analogue, second-line agent 6
  • Used for ganciclovir-resistant CMV 6

Cidofovir

  • Nucleotide analogue, second-line agent 6
  • Targets viral DNA polymerase 6

Letermovir (Newest Agent)

  • Targets viral terminase complex (different mechanism than DNA polymerase inhibitors) 6
  • Approved for CMV prophylaxis in hematopoietic stem cell transplant recipients 6
  • Avoids cross-resistance with DNA polymerase inhibitors 6

Resistance Considerations

  • All first-line agents (ganciclovir, foscarnet, cidofovir) target viral DNA polymerase – cross-resistance possible 6
  • Letermovir offers alternative mechanism for resistant cases 6

HEPATITIS B VIRUS (HBV) ANTIVIRALS

Approved Drug Classes

  • Lamivudine
  • Interferons
  • Nucleoside analogues
  • Acyclic nucleoside phosphonate analogues 4

Treatment Initiation Criteria

HBeAg-Positive Chronic Hepatitis B

  • Serum HBV DNA >20,000 IU/mL AND ALT >2× normal 7
  • Consider treatment if ALT remains elevated >6 months 7
  • Initial options: Interferon-α or lamivudine 7

HBeAg-Negative Chronic Hepatitis B

  • Serum HBV DNA >20,000 IU/mL AND ALT >2× normal 7
  • For lower HBV DNA (2,000-20,000 IU/mL) with borderline/minimally elevated ALT: Consider liver biopsy – treat if moderate/severe inflammation or significant fibrosis 7
  • Preferred agents: Pegylated interferon-α, adefovir, or entecavir (due to need for long-term treatment) 7

Lamivudine Resistance Management

If adefovir is added:

  • Continue lamivudine indefinitely – decreases hepatitis flares during transition and reduces adefovir resistance risk 7

If switching to entecavir:

  • Stop lamivudine – continued lamivudine-resistant mutations increase entecavir resistance risk 7

Adefovir Resistance Management

In patients with no prior nucleoside analogue exposure:

  • Add lamivudine or entecavir 7

In patients with prior lamivudine resistance:

  • Add lamivudine – but durability unknown, re-emergence of lamivudine-resistant mutations reported 7

Entecavir Resistance Management

  • Add or switch to adefovir or tenofovir 7

Telbivudine Resistance Management

  • Add adefovir or tenofovir 7
  • Switch to Truvada (emtricitabine 200mg + tenofovir 300mg combination) 7
  • Switch to entecavir (but pre-existing telbivudine-resistant mutations predispose to entecavir resistance) 7

Monitoring During Treatment

  • Serum HBV DNA (PCR assay) every 3-6 months 7
  • Check medication compliance with virologic breakthrough 7
  • Confirm antiviral resistance with genotypic testing 7

Prevention of Resistance

  • Avoid unnecessary treatment 7
  • Initiate with potent antiviral with low resistance rate or combination therapy 7
  • Switch to alternative therapy in primary non-responders 7

Retreatment After Interferon Failure

  • Patients who failed prior interferon-α (standard or pegylated) may be retreated with nucleoside analogues if they meet treatment criteria 7

Primary Non-Response

  • <2 log decrease in serum HBV DNA after ≥6 months of nucleoside analogue therapy – switch to alternative treatment 7

HEPATITIS C VIRUS (HCV) ANTIVIRALS

Approved Drug Classes

  • Ribavirin
  • Interferons
  • NS3/4A protease inhibitors
  • NS5A inhibitors
  • NS5B polymerase inhibitors 4

Combination Therapy

  • Ribavirin + interferon-α superior to interferon-α alone for chronic hepatitis C 8

INFLUENZA ANTIVIRALS

Approved Drug Classes

  • Ribavirin
  • Matrix 2 (M2) protein inhibitors (amantadine)
  • RNA polymerase inhibitors
  • Neuraminidase inhibitors (zanamivir, oseltamivir) 4

Neuraminidase Inhibitors vs. Amantadine

  • Neuraminidase inhibitors (zanamivir, oseltamivir) active against both influenza A and B 8
  • Amantadine only active against influenza A 8
  • Neuraminidase inhibitors are well-tolerated 8

RESPIRATORY SYNCYTIAL VIRUS (RSV) ANTIVIRALS

Approved Agents

  • Ribavirin
  • Antibodies 4

HUMAN PAPILLOMAVIRUS (HPV) – EXTERNAL ANOGENITAL WARTS

Imiquimod

Mechanism

  • Immune response modifier 4

FDA-Approved Indications

  • External anogenital warts (HPV)
  • Actinic keratosis
  • Superficial basal cell carcinoma 9

Actinic Keratosis Regimen

  • Apply 2 times per week for 16 weeks to 25 cm² contiguous treatment area 9
  • Apply to entire treatment area before sleep, leave on ~8 hours 9
  • Complete clearance rate: 44-46% (vs. 3-4% vehicle) 9
  • Partial clearance (≥75% lesions cleared): 58-60% (vs. 10-14% vehicle) 9
  • 48% of patients experience increase in AK lesions during treatment (subclinical lesions become apparent) – similar response to those without increase 9

Superficial Basal Cell Carcinoma Regimen

  • Apply 5 times per week for 6 weeks to target tumor + 1 cm margin 9
  • Target tumor: 0.5-4.0 cm², maximum diameter 2.0 cm, biopsy-confirmed sBCC 9
  • Apply before sleep, leave on ~8 hours 9
  • Composite clearance (clinical + histological): 75% (vs. 2% vehicle) at 12 weeks post-treatment 9
  • 6% of clinically clear patients had residual tumor on histological examination 9

Other HPV Treatments

  • Sinecatechins
  • Podofilox 4

HIV POST-EXPOSURE PROPHYLAXIS (PEP)

First-Line Regimen (2025 Guidelines)

Bictegravir/emtricitabine/tenofovir alafenamide (BIC/FTC/TAF) 50/200/25mg as single tablet once daily for 28 days is the preferred PEP regimen. 10

Critical Timing

  • Initiate ideally within 24 hours, absolutely within 72 hours maximum – efficacy decreases significantly with delayed initiation 10
  • Complete full 28-day course regardless of regimen 10

Advantages of TAF Over TDF

  • Tenofovir alafenamide (TAF) preferred over tenofovir disoproxil fumarate (TDF) – superior renal and bone safety profiles 10
  • Strongly preferred in patients with impaired renal function 10

Alternative Multi-Tablet Regimen

  • Dolutegravir 50mg once daily PLUS emtricitabine/tenofovir alafenamide 200/25mg once daily for 28 days 10

Monitoring Requirements

  • Baseline: Rapid or laboratory HIV antigen/antibody combination test 10
  • 4-6 weeks: HIV Ag/Ab test + HIV NAT 10
  • 12 weeks: Laboratory HIV Ag/Ab combination immunoassay + HIV NAT 10

Critical Pitfalls to Avoid

  • Never prescribe only two NRTIs (e.g., tenofovir/emtricitabine alone) – inadequate protection, requires third drug (integrase inhibitor) 10
  • Never delay initiation beyond 72 hours – effectiveness drops precipitously 10

Transition to PrEP After PEP

  • For MSM with/at risk for kidney dysfunction, osteopenia, or osteoporosis: Daily tenofovir alafenamide/emtricitabine recommended 10

HIV POST-EXPOSURE PROPHYLAXIS (2001 Guidelines – Historical Context)

Basic Regimen

  • Zidovudine (ZDV/AZT) 600mg/day in 2-3 divided doses + Lamivudine (3TC) 150mg twice daily (available as Combivir) 7
  • Advantages: ZDV associated with decreased HIV transmission risk in CDC case-control study; serious toxicity rare for PEP; safe in pregnancy; single tablet twice daily 7
  • Disadvantages: Common side effects (low adherence); potential resistance; unknown delayed toxicity 7

Alternate Basic Regimens

Lamivudine + Stavudine

  • 3TC 150mg twice daily + d4T 40mg twice daily (30mg if <60kg) 7
  • Well-tolerated, rare serious toxicity, twice-daily dosing 7

Didanosine + Stavudine

  • ddI 400mg daily on empty stomach + d4T 40mg twice daily (adjust for <60kg) 7
  • Effective against ZDV/3TC-resistant strains 7
  • Major disadvantages: Difficult to administer, unpalatable; serious toxicity (neuropathy, pancreatitis, hepatitis); ddI interferes with quinolone antibiotics and indinavir absorption 7
  • Monitor closely for pancreatitis, lactic acidosis, hepatitis 7

Expanded Regimen (Basic + One of Following)

Indinavir

  • 800mg every 8 hours on empty stomach 7
  • Serious toxicity: Nephrolithiasis (requires 8 glasses fluid/day); hyperbilirubinemia (avoid late pregnancy) 7

Nelfinavir

  • 750mg three times daily OR 1250mg twice daily with meals/snack 7
  • Potent HIV inhibitor, twice-daily dosing improves adherence 7
  • Contraindicated with: Astemizole, terfenadine, ergot derivatives, rifampin, cisapride, St. John's Wort, lovastatin, simvastatin, pimozide, midazolam, triazolam 7
  • Accelerates clearance of oral contraceptives (use alternative contraception) 7

Efavirenz

  • 600mg daily at bedtime 7
  • Advantages: Does not require phosphorylation (may be active earlier); once-daily dosing 7
  • Major disadvantages: Rash (can progress to Stevens-Johnson syndrome); CNS side effects (dizziness, somnolence, insomnia, abnormal dreaming, severe psychiatric symptoms); teratogenic – contraindicated in pregnancy 7
  • Contraindicated with: Astemizole, cisapride, midazolam, triazolam, ergot derivatives, St. John's Wort 7

Abacavir

  • 300mg twice daily (available as Trizivir with ZDV/3TC) 7
  • Potent HIV inhibitor, well-tolerated 7
  • Severe hypersensitivity reactions (usually within first 6 weeks) 7

Lopinavir/Ritonavir (Kaletra)

  • 400/100mg twice daily 7
  • Potent HIV inhibitor, well-tolerated 7
  • Contraindicated with: Flecainide, propafenone, astemizole, terfenadine, ergot derivatives, rifampin, cisapride, St. John's Wort, lovastatin, simvastatin, pimozide, midazolam, triazolam 7
  • Accelerates clearance of oral contraceptives 7

Nevirapine

  • 200mg daily for 2 weeks, then 200mg twice daily 7
  • Major disadvantages: Severe hepatotoxicity (including liver failure requiring transplantation in PEP patient); rash progressing to Stevens-Johnson syndrome 7

Follow-Up Testing (2001 Guidelines)

HBV Exposures

  • Anti-HBs testing 1-2 months after last vaccine dose 7
  • Cannot assess vaccine response if HBIG received in previous 3-4 months 7

HCV Exposures

  • Baseline and follow-up anti-HCV and ALT at 4-6 months 7
  • HCV RNA at 4-6 weeks if earlier diagnosis desired 7
  • Confirm repeatedly reactive anti-HCV EIAs with supplemental tests 7
  • No PEP recommended for HCV 7

HIV Exposures

  • HIV antibody testing for ≥6 months: Baseline, 6 weeks, 3 months, 6 months 7
  • Test if acute retroviral syndrome symptoms occur 7
  • Evaluate within 72 hours after exposure, monitor for drug toxicity ≥2 weeks 7
  • Advise precautions to prevent secondary transmission during follow-up 7

GENERAL ANTIVIRAL PRINCIPLES

Mechanism of Action

  • Antivirals inhibit viral replication by interacting with specific elements of the viral replication cycle 11

Therapeutic Window Challenges

  • Acute infections (influenza, COVID-19) offer small therapeutic window due to pathophysiological dynamics 11
  • Optimal timing: Immediately after virus exposure – frequently limits practical application 11

Persistent vs. Acute Viral Infections

  • Persistent viruses (HIV, HBV, herpes viruses): Cannot be cured with current agents 11
  • Acute infections: Complete viral elimination possible 11

Pre-Exposure and Post-Exposure Prophylaxis

  • Established for: HIV, influenza A/B 11
  • Gaining relevance for: SARS-CoV-2 11

Resistance Concerns

  • Herpesvirus resistance to antivirals is of concern 8
  • Validation of alternative treatments for documented resistance required 8

Historical Milestone

  • Idoxuridine (1963): First antiviral drug approved 4
  • 90 antiviral drugs in 13 functional groups approved over 50 years 4

References

Guideline

Oral Antiviral Treatment for Herpes Simplex

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Management of Herpes Lesions

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Approved Antiviral Drugs over the Past 50 Years.

Clinical microbiology reviews, 2016

Research

Antiviral Drugs Against Herpesviruses.

Advances in experimental medicine and biology, 2021

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Antiviral therapy: current options and challenges.

Schweizerische medizinische Wochenschrift, 2000

Guideline

Tenofovir Alafenamide for Post-Exposure Prophylaxis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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